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Dopaminergic and adrenergic toxicities on SK-N-MC human neuroblastoma cells are mediated through G protein signaling and oxidative stress

Apoptosis (London), 2007-01, Vol.12 (1), p.167-179

2007

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Interleukin-6 autocrine signaling mediates melatonin MT1/2 receptor-induced STAT3 Tyr705 phosphorylation

Journal of pineal research, 2012-05, Vol.52 (4), p.477-489

2012

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RGS19 enhances cell proliferation through its C-terminal PDZ motif

Cellular signalling, 2010-11, Vol.22 (11), p.1700-1707

2010

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CKBM stimulates MAPKs but inhibits LPS-induced IFN-γ in lymphocytes

Phytotherapy research, 2006-09, Vol.20 (9), p.725-731

2006

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Phosphatidylinositol‐3 kinase is distinctively required for µ‐, but not κ‐opioid receptor‐induced activation of c‐Jun N‐terminal kinase

Journal of neurochemistry, 2004-04, Vol.89 (2), p.391-402

2004

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Integration of G protein signals by extracellular signal‐regulated protein kinases in SK‐N‐MC neuroepithelioma cells

Journal of neurochemistry, 2005-09, Vol.94 (5), p.1457-1470

2005

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Rac and Cdc42‐dependent regulation of c‐Jun N‐terminal kinases by the δ‐opioid receptor

Journal of neurochemistry, 2003-02, Vol.84 (3), p.503-513

2003

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Gq-Mediated Activation of c-Jun N-Terminal Kinase by the Gastrin-Releasing Peptide-Preferring Bombesin Receptor Is Inhibited upon Costimulation of the Gs-Coupled Dopamine D1 Receptor in COS-7 Cells

Molecular pharmacology, 2005-11, Vol.68 (5), p.1354-1364

2005

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Kappa-opioid receptor signals through Src and focal adhesion kinase to stimulate c-Jun N-terminal kinases in transfected COS-7 cells and human monocytic THP-1 cells

The Journal of pharmacology and experimental therapeutics, 2004-07, Vol.310 (1), p.301-310

2004

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Epidermal growth factor differentially augments Gi‐mediated stimulation of c‐Jun N‐terminal kinase activity

British journal of pharmacology, 2004-06, Vol.142 (4), p.635-646

2004

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The first and third intracellular loops together with the carboxy terminal tail of the δ‐opioid receptor contribute toward functional interaction with Gα16

Journal of neurochemistry, 2003-11, Vol.87 (3), p.697-708

2003

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Differential Regulation of CXCL8 Production by Different G Protein Subunits with Synergistic Stimulation by Gi- and Gq-Regulated Pathways

Journal of molecular biology, 2016-09, Vol.428 (19), p.3869-3884

2016

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Activation of Gαq subunits up-regulates the expression of the tumor suppressor Fhit

Cellular signalling, 2013-12, Vol.25 (12), p.2440-2452

2013

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Epidermal growth factor differentially augments G i ‐mediated stimulation of c‐Jun N‐terminal kinase activity

British journal of pharmacology, 2009-01, Vol.142 (4), p.635-646

2009

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Melatonin mt1 and MT2 receptors stimulate c-Jun N-terminal kinase via pertussis toxin-sensitive and -insensitive G proteins

Cellular signalling, 2002-03, Vol.14 (3), p.249-257

2002

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An intact helical domain is required for Gα14 to stimulate phospholipase Cβ

BMC structural biology, 2015-09, Vol.15 (1), p.18-18, Article 18

2015

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Activation state-dependent interaction between Gαq subunits and the Fhit tumor suppressor

Cell communication and signaling, 2013-08, Vol.11 (1), p.59-59, Article 59

2013

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Interleukin‐6 autocrine signaling mediates melatonin MT 1/2 receptor‐induced STAT3 Tyr 705 phosphorylation

Journal of pineal research, 2012-05, Vol.52 (4), p.477-489

2012

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G q -Mediated Activation of c-Jun N-Terminal Kinase by the Gastrin-Releasing Peptide-Preferring Bombesin Receptor Is Inhibited upon Costimulation of the G s -Coupled Dopamine D 1 Receptor in COS-7 Cells

Molecular pharmacology, 2005-11, Vol.68 (5), p.1354-1364

2005

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Activation state-dependent interaction between G[alpha]q subunits and the Fhit tumor suppressor

Cell communication and signaling, 2013-08, Vol.11

2013

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Activation state-dependent interaction between G[alpha] sub(q) subunits and the Fhit tumor suppressor

Cell communication and signaling, 2013-01, Vol.11 (1), p.59-59

2013

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An intact helical domain is required for G[alpha]14 to stimulate phospholipase C[beta]

BMC structural biology, 2015-09, Vol.15

2015

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Gbetagamma signaling and Ca2+ mobilization co-operate synergistically in a Sos and Rac-dependent manner in the activation of JNK by Gq-coupled receptors

Cellular signalling, 2004-07, Vol.16 (7), p.823-836

2004

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Epidermal growth factor differentially augments G(i)-mediated stimulation of c-Jun N-terminal kinase activity

British journal of pharmacology, 2004-06, Vol.142 (4), p.635-646

2004

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Carvacrol and eugenol differentially stimulate intracellular Ca2+ mobilization and mitogen-activated protein kinases in Jurkat-T-cells and monocytic THP-1 cells

Planta medica, 2005-07, Vol.71 (7), p.634-639

2005

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Dopaminergic and adrenergic toxicities on SK-N-MC human neuroblastoma cells are mediated through G protein signaling and oxidative stress

Interleukin-6 autocrine signaling mediates melatonin MT1/2 receptor-induced STAT3 Tyr705 phosphorylation

RGS19 enhances cell proliferation through its C-terminal PDZ motif

CKBM stimulates MAPKs but inhibits LPS-induced IFN-γ in lymphocytes

Phosphatidylinositol‐3 kinase is distinctively required for µ‐, but not κ‐opioid receptor‐induced activation of c‐Jun N‐terminal kinase

Integration of G protein signals by extracellular signal‐regulated protein kinases in SK‐N‐MC neuroepithelioma cells

Rac and Cdc42‐dependent regulation of c‐Jun N‐terminal kinases by the δ‐opioid receptor

Gq-Mediated Activation of c-Jun N-Terminal Kinase by the Gastrin-Releasing Peptide-Preferring Bombesin Receptor Is Inhibited upon Costimulation of the Gs-Coupled Dopamine D1 Receptor in COS-7 Cells

Kappa-opioid receptor signals through Src and focal adhesion kinase to stimulate c-Jun N-terminal kinases in transfected COS-7 cells and human monocytic THP-1 cells

Epidermal growth factor differentially augments Gi‐mediated stimulation of c‐Jun N‐terminal kinase activity

The first and third intracellular loops together with the carboxy terminal tail of the δ‐opioid receptor contribute toward functional interaction with Gα16

Differential Regulation of CXCL8 Production by Different G Protein Subunits with Synergistic Stimulation by Gi- and Gq-Regulated Pathways

Activation of Gαq subunits up-regulates the expression of the tumor suppressor Fhit

Epidermal growth factor differentially augments G i ‐mediated stimulation of c‐Jun N‐terminal kinase activity

Melatonin mt1 and MT2 receptors stimulate c-Jun N-terminal kinase via pertussis toxin-sensitive and -insensitive G proteins

An intact helical domain is required for Gα14 to stimulate phospholipase Cβ

Activation state-dependent interaction between Gαq subunits and the Fhit tumor suppressor

Interleukin‐6 autocrine signaling mediates melatonin MT 1/2 receptor‐induced STAT3 Tyr 705 phosphorylation

G q -Mediated Activation of c-Jun N-Terminal Kinase by the Gastrin-Releasing Peptide-Preferring Bombesin Receptor Is Inhibited upon Costimulation of the G s -Coupled Dopamine D 1 Receptor in COS-7 Cells

Activation state-dependent interaction between G[alpha]q subunits and the Fhit tumor suppressor

Activation state-dependent interaction between G[alpha] sub(q) subunits and the Fhit tumor suppressor

An intact helical domain is required for G[alpha]14 to stimulate phospholipase C[beta]

Gbetagamma signaling and Ca2+ mobilization co-operate synergistically in a Sos and Rac-dependent manner in the activation of JNK by Gq-coupled receptors

Epidermal growth factor differentially augments G(i)-mediated stimulation of c-Jun N-terminal kinase activity

Carvacrol and eugenol differentially stimulate intracellular Ca2+ mobilization and mitogen-activated protein kinases in Jurkat-T-cells and monocytic THP-1 cells

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