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Therapeutic Activity of Anti-AXL Antibody against Triple-Negative Breast Cancer Patient-Derived Xenografts and Metastasis

Clinical cancer research, 2017-06, Vol.23 (11), p.2806-2816

2017

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PTPN13/PTPL1: an important regulator of tumor aggressiveness

Anti-cancer agents in medicinal chemistry, 2011-01, Vol.11 (1), p.78-88

2011

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Hyperactivated NF-κB and AP-1 Transcription Factors Promote Highly Accessible Chromatin and Constitutive Transcription across the Interleukin-6 Gene Promoter in Metastatic Breast Cancer Cells

Molecular and Cellular Biology, 2009-10, Vol.29 (20), p.5488-5504

2009

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FRA-1 expression level regulates proliferation and invasiveness of breast cancer cells

Oncogene, 2005-02, Vol.24 (8), p.1434-1444

2005

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Epithelial-to-Mesenchymal Transition Induced by TGF-β1 Is Mediated by Blimp-1―Dependent Repression of BMP-5

Cancer research (Chicago, Ill.), 2012-12, Vol.72 (23), p.6268-6278

2012

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Ubiquitin-Independent Proteasomal Degradation of Fra-1 Is Antagonized by Erk1/2 Pathway-Mediated Phosphorylation of a Unique C-Terminal Destabilizer

Molecular and Cellular Biology, 2007-06, Vol.27 (11), p.3936-3950

2007

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Mechanisms underlying differential expression of interleukin-8 in breast cancer cells

Oncogene, 2004-08, Vol.23 (36), p.6105-6114

2004

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RelB NF-κB Represses Estrogen Receptor α Expression via Induction of the Zinc Finger Protein Blimp1

Molecular and Cellular Biology, 2009-07, Vol.29 (14), p.3832-3844

2009

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Estrogen induction and overexpression of fibulin-1C mRNA in ovarian cancer cells

Oncogene, 2002-02, Vol.21 (7), p.1097-1107

2002

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The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells

Oncogene, 2012-11, Vol.31 (47), p.4889-4897

2012

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Oestrogen signalling inhibits invasive phenotype by repressing RelB and its target BCL2

Nature cell biology, 2007-04, Vol.9 (4), p.470-478

2007

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PKCθ-induced phosphorylations control the ability of Fra-1 to stimulate gene expression and cancer cell migration

Cancer letters, 2017-01, Vol.385, p.97-107

2017

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Potentiation of ICI182,780 (Fulvestrant)-induced Estrogen Receptor-α Degradation by the Estrogen Receptor-related Receptor-α Inverse Agonist XCT790

The Journal of biological chemistry, 2007-09, Vol.282 (39), p.28328-28334

2007

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The putative tumor suppressor gene PTPN13/PTPL1 induces apoptosis through insulin receptor substrate-1 dephosphorylation

Cancer research (Chicago, Ill.), 2007-07, Vol.67 (14), p.6806-6813

2007

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PTPL1/PTPN13 Regulates Breast Cancer Cell Aggressiveness through Direct Inactivation of Src Kinase

Cancer research (Chicago, Ill.), 2010-06, Vol.70 (12), p.5116-5126

2010

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PKC[theta]-induced phosphorylations control the ability of Fra-1 to stimulate gene expression and cancer cell migration

Cancer letters, 2017-01, Vol.385, p.97-107

2017

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Characterization of the Physical Interaction between Estrogen Receptor α and JUN Proteins

The Journal of biological chemistry, 2001-09, Vol.276 (39), p.36361-36369

2001

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Expression of the putative tumor suppressor gene PTPN13/PTPL1 is an independent prognostic marker for overall survival in breast cancer

International journal of cancer, 2009-02, Vol.124 (3), p.638-643

2009

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The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells: PKCθ pathway stabilises Fra-1

Oncogene, 2012-11, Vol.31 (47), p.4889-97

2012

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The role of sex steroid receptors on lipogenesis in breast and prostate carcinogenesis: a viewpoint

Hormones & cancer, 2010-04, Vol.1 (2), p.63-70

2010

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Increased expression of fatty acid synthase and progesterone receptor in early steps of human mammary carcinogenesis

International journal of cancer, 2007-01, Vol.120 (2), p.224-229

2007

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RU486, a progestin and glucocorticoid antagonist, inhibits the growth of breast cancer cells via the progesterone receptor

The journal of clinical endocrinology and metabolism, 1985-04, Vol.60 (4), p.692-697

1985

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PTPL1/PTPN13 regulates breast cancer cell aggressiveness through direct inactivation of Src kinase: PTPL1 inhibits Src activation and tumor aggressiveness

Cancer research (Chicago, Ill.), 2010-06, Vol.70 (12), p.5116-26

2010

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Unliganded and Liganded Estrogen Receptors Protect against Cancer Invasion via Different Mechanisms

Molecular endocrinology (Baltimore, Md.), 2000-07, Vol.14 (7), p.999-1009

2000

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Receptor-Interacting Protein 140 Binds c-Jun and Inhibits Estradiol-Induced Activator Protein-1 Activity by Reversing Glucocorticoid Receptor-Interacting Protein 1 Effect

Molecular endocrinology (Baltimore, Md.), 2003-02, Vol.17 (2), p.287-299

2003

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Therapeutic Activity of Anti-AXL Antibody against Triple-Negative Breast Cancer Patient-Derived Xenografts and Metastasis

PTPN13/PTPL1: an important regulator of tumor aggressiveness

Hyperactivated NF-κB and AP-1 Transcription Factors Promote Highly Accessible Chromatin and Constitutive Transcription across the Interleukin-6 Gene Promoter in Metastatic Breast Cancer Cells

FRA-1 expression level regulates proliferation and invasiveness of breast cancer cells

Epithelial-to-Mesenchymal Transition Induced by TGF-β1 Is Mediated by Blimp-1―Dependent Repression of BMP-5

Ubiquitin-Independent Proteasomal Degradation of Fra-1 Is Antagonized by Erk1/2 Pathway-Mediated Phosphorylation of a Unique C-Terminal Destabilizer

Mechanisms underlying differential expression of interleukin-8 in breast cancer cells

RelB NF-κB Represses Estrogen Receptor α Expression via Induction of the Zinc Finger Protein Blimp1

Estrogen induction and overexpression of fibulin-1C mRNA in ovarian cancer cells

The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells

Oestrogen signalling inhibits invasive phenotype by repressing RelB and its target BCL2

PKCθ-induced phosphorylations control the ability of Fra-1 to stimulate gene expression and cancer cell migration

Potentiation of ICI182,780 (Fulvestrant)-induced Estrogen Receptor-α Degradation by the Estrogen Receptor-related Receptor-α Inverse Agonist XCT790

The putative tumor suppressor gene PTPN13/PTPL1 induces apoptosis through insulin receptor substrate-1 dephosphorylation

PTPL1/PTPN13 Regulates Breast Cancer Cell Aggressiveness through Direct Inactivation of Src Kinase

PKC[theta]-induced phosphorylations control the ability of Fra-1 to stimulate gene expression and cancer cell migration

Characterization of the Physical Interaction between Estrogen Receptor α and JUN Proteins

Expression of the putative tumor suppressor gene PTPN13/PTPL1 is an independent prognostic marker for overall survival in breast cancer

The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells: PKCθ pathway stabilises Fra-1

The role of sex steroid receptors on lipogenesis in breast and prostate carcinogenesis: a viewpoint

Increased expression of fatty acid synthase and progesterone receptor in early steps of human mammary carcinogenesis

RU486, a progestin and glucocorticoid antagonist, inhibits the growth of breast cancer cells via the progesterone receptor

PTPL1/PTPN13 regulates breast cancer cell aggressiveness through direct inactivation of Src kinase: PTPL1 inhibits Src activation and tumor aggressiveness

Unliganded and Liganded Estrogen Receptors Protect against Cancer Invasion via Different Mechanisms

Receptor-Interacting Protein 140 Binds c-Jun and Inhibits Estradiol-Induced Activator Protein-1 Activity by Reversing Glucocorticoid Receptor-Interacting Protein 1 Effect

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