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Redox Paradox: A Novel Approach to Therapeutics-Resistant Cancer

Antioxidants & redox signaling, 2018-11, Vol.29 (13), p.1237-1272

2018

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Chemotherapy-induced cognitive impairment: focus on the intersection of oxidative stress and TNFα

Cellular and molecular life sciences : CMLS, 2021-10, Vol.78 (19-20), p.6533-6540

2021

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Sensitization of Resistant Prostate Cancers to Radiation Through Mitochondrial Translation Targeting Coupled With Overloading Mitochondrial ROS

Free radical biology & medicine, 2022-11, Vol.192, p.99-99

Chaiswing, Luksana

2022

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Mitochondrial ROS Heterogeneity: Adaptive Vulnerability That Can be Targeted to Sensitize Resistant Prostate Cancer to Radiation

Free radical biology & medicine, 2022-11, Vol.192, p.13-13

Chaiswing, Luksana

2022

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RelB Expression Determines the Differential Effects of Ascorbic Acid in Normal and Cancer Cells

Cancer research (Chicago, Ill.), 2017-03, Vol.77 (6), p.1345-1356

2017

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Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury

Clinical cancer research, 2018-04, Vol.24 (7), p.1644-1653

2018

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Frenolicin B Targets Peroxiredoxin 1 and Glutaredoxin 3 to Trigger ROS/4E-BP1-Mediated Antitumor Effects

Cell chemical biology, 2019-03, Vol.26 (3), p.366-377.e12

2019

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Profiles of Radioresistance Mechanisms in Prostate Cancer

Critical reviews in oncogenesis, 2018, Vol.23 (1-2), p.39-67

2018

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Extracellular Vesicles and Cancer Therapy: Insights into the Role of Oxidative Stress

Antioxidants, 2022-06, Vol.11 (6), p.1194

2022

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The RelB-BLNK Axis Determines Cellular Response to a Novel Redox-Active Agent Betamethasone during Radiation Therapy in Prostate Cancer

International journal of molecular sciences, 2022-06, Vol.23 (12), p.6409

2022

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Extracellular vesicle-mediated macrophage activation: An insight into the mechanism of thioredoxin-mediated immune activation

Redox biology, 2019-09, Vol.26, p.101237-101237, Article 101237

2019

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P53 translocation to mitochondria precedes its nuclear translocation and targets mitochondrial oxidative defense protein-manganese superoxide dismutase

Cancer research (Chicago, Ill.), 2005-05, Vol.65 (9), p.3745-3750

2005

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Regulation of prostate cancer cell invasion by modulation of extra- and intracellular redox balance

Free radical biology & medicine, 2012-01, Vol.52 (2), p.452-461

2012

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Hydrogen Peroxide Promotes the Production of Radiation-Derived EVs Containing Mitochondrial Proteins

Antioxidants, 2022-10, Vol.11 (11), p.2119

2022

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Distinct redox profiles of selected human prostate carcinoma cell lines: implications for rational design of redox therapy

Cancers, 2011-09, Vol.3 (3), p.3557-3584

2011

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266 - Alteration of Tumor Microenvironmental Redox State Inhibits Prostate Cancer Metastasis

Free radical biology & medicine, 2016-11, Vol.100, p.S119-S119

2016

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Alteration of Tumor Microenvironmental Redox State Inhibits Prostate Cancer Metastasis

Free radical biology & medicine, 2016-11, Vol.100, p.S119

2016

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Nuclear interaction between ADR-induced p65 and p53 mediates cardiac injury in iNOS (-/-) mice

PloS one, 2014-02, Vol.9 (2), p.e89251

2014

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Oxidative Damage Precedes Nitrative Damage in Adriamycin-Induced Cardiac Mitochondrial Injury

Toxicologic pathology, 2004-09, Vol.32 (5), p.536-547

2004

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Manganese superoxide dismutase and inducible nitric oxide synthase modify early oxidative events in acute Adriamycin-induced mitochondrial toxicity

Molecular cancer therapeutics, 2005-07, Vol.4 (7), p.1056-1064

2005

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A NOVEL DRUG THERAPY INDUCES MITOCHONDRIAL BIOGENESIS AND ATTENUATES COLITIS

Gastroenterology (New York, N.Y. 1943), 2023-04, Vol.164 (4), p.S70-S70

2023

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Manganese superoxide dismutase deficiency enhances cell turnover via tumor promoter-induced alterations in AP-1 and p53-mediated pathways in a skin cancer model

Oncogene, 2002-05, Vol.21 (24), p.3836-3846

2002

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A NOVEL DRUG THERAPY INDUCES MITOCHONDRIAL BIOGENESIS AND ATTENUATES COLITIS

Inflammatory bowel diseases, 2023-01, Vol.29 (Supplement_1), p.S55-S55

2023

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Plausible biochemical mechanisms of chemotherapy-induced cognitive impairment (“chemobrain”), a condition that significantly impairs the quality of life of many cancer survivors

Biochimica et biophysica acta. Molecular basis of disease, 2019-06, Vol.1865 (6), p.1088-1097

2019

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Extracellular/microenvironmental redox state

Antioxidants & redox signaling, 2010-08, Vol.13 (4), p.449-465

2010

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Redox Paradox: A Novel Approach to Therapeutics-Resistant Cancer

Chemotherapy-induced cognitive impairment: focus on the intersection of oxidative stress and TNFα

Sensitization of Resistant Prostate Cancers to Radiation Through Mitochondrial Translation Targeting Coupled With Overloading Mitochondrial ROS

Mitochondrial ROS Heterogeneity: Adaptive Vulnerability That Can be Targeted to Sensitize Resistant Prostate Cancer to Radiation

RelB Expression Determines the Differential Effects of Ascorbic Acid in Normal and Cancer Cells

Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury

Frenolicin B Targets Peroxiredoxin 1 and Glutaredoxin 3 to Trigger ROS/4E-BP1-Mediated Antitumor Effects

Profiles of Radioresistance Mechanisms in Prostate Cancer

Extracellular Vesicles and Cancer Therapy: Insights into the Role of Oxidative Stress

The RelB-BLNK Axis Determines Cellular Response to a Novel Redox-Active Agent Betamethasone during Radiation Therapy in Prostate Cancer

Extracellular vesicle-mediated macrophage activation: An insight into the mechanism of thioredoxin-mediated immune activation

P53 translocation to mitochondria precedes its nuclear translocation and targets mitochondrial oxidative defense protein-manganese superoxide dismutase

Regulation of prostate cancer cell invasion by modulation of extra- and intracellular redox balance

Hydrogen Peroxide Promotes the Production of Radiation-Derived EVs Containing Mitochondrial Proteins

Distinct redox profiles of selected human prostate carcinoma cell lines: implications for rational design of redox therapy

266 - Alteration of Tumor Microenvironmental Redox State Inhibits Prostate Cancer Metastasis

Alteration of Tumor Microenvironmental Redox State Inhibits Prostate Cancer Metastasis

Nuclear interaction between ADR-induced p65 and p53 mediates cardiac injury in iNOS (-/-) mice

Oxidative Damage Precedes Nitrative Damage in Adriamycin-Induced Cardiac Mitochondrial Injury

Manganese superoxide dismutase and inducible nitric oxide synthase modify early oxidative events in acute Adriamycin-induced mitochondrial toxicity

A NOVEL DRUG THERAPY INDUCES MITOCHONDRIAL BIOGENESIS AND ATTENUATES COLITIS

Manganese superoxide dismutase deficiency enhances cell turnover via tumor promoter-induced alterations in AP-1 and p53-mediated pathways in a skin cancer model

A NOVEL DRUG THERAPY INDUCES MITOCHONDRIAL BIOGENESIS AND ATTENUATES COLITIS

Plausible biochemical mechanisms of chemotherapy-induced cognitive impairment (“chemobrain”), a condition that significantly impairs the quality of life of many cancer survivors

Extracellular/microenvironmental redox state

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