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EZH2 promotes degradation of stalled replication forks by recruiting MUS81 through histone H3 trimethylation

Nature cell biology, 2017-11, Vol.19 (11), p.1371

2017

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Repair Pathway Choices and Consequences at the Double-Strand Break

Trends in cell biology, 2016-01, Vol.26 (1), p.52-64

2016

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A prospective study of bone marrow hematopoietic and mesenchymal stem cells in type 1 Gaucher disease patients

PloS one, 2013-07, Vol.8 (7), p.e69293-e69293

2013

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Homologous Recombination Deficiency: Exploiting the Fundamental Vulnerability of Ovarian Cancer

Cancer discovery, 2015-11, Vol.5 (11), p.1137-1154

2015

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Gene polymorphisms and cytokine plasma levels as predictive factors of complications after cardiopulmonary bypass

The Journal of thoracic and cardiovascular surgery, 2012-08, Vol.144 (2), p.467-473.e2

2012

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FANCD2 Maintains Fork Stability in BRCA1/2-Deficient Tumors and Promotes Alternative End-Joining DNA Repair

Cell reports (Cambridge), 2016-06, Vol.15 (11), p.2488-2499

2016

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A first-in-class Polymerase Theta Inhibitor selectively targets Homologous-Recombination-Deficient Tumors

Nature cancer, 2021-06, Vol.2 (6), p.598

2021

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Transcriptional Repressor ZBTB1 Promotes Chromatin Remodeling and Translesion DNA Synthesis

Molecular cell, 2014-04, Vol.54 (1), p.107-118

2014

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The Fanconi anaemia pathway: new players and new functions

Nature reviews. Molecular cell biology, 2016-06, Vol.17 (6), p.337

2016

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Spontaneous abrogation of the G2 DNA damage checkpoint has clinical benefits but promotes leukemogenesis in Fanconi anemia patients

The Journal of clinical investigation, 2011-01, Vol.121 (1), p.184-194

2011

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Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair

Nature (London), 2015-02, Vol.518 (7538), p.258-262

2015

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DNA damage induces nuclear envelope rupture through ATR-mediated phosphorylation of lamin A/C

Molecular cell, 2023-10, Vol.83 (20), p.3659-3668.e10

2023

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DNA damage induces nuclear envelope rupture through ATR-mediated phosphorylation of lamin A/C

Molecular cell, 2023-10, Vol.83 (20), p.3659-3668.e10

2023

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Polθ is phosphorylated by PLK1 to repair double-strand breaks in mitosis

Nature (London), 2023-09, Vol.621 (7978), p.415-422

2023

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CtIP-Mediated Fork Protection Synergizes with BRCA1 to Suppress Genomic Instability upon DNA Replication Stress

Molecular cell, 2018-11, Vol.72 (3), p.568-582.e6

2018

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Biallelic inactivation of REV7 is associated with Fanconi anemia

The Journal of clinical investigation, 2016-09, Vol.126 (9), p.3580-3584

2016

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A unique subset of epithelial ovarian cancers with platinum sensitivity and PARP inhibitor resistance

Cancer research (Chicago, Ill.), 2015-02, Vol.75 (4), p.628-634

2015

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Publisher Correction: Polθ is phosphorylated by PLK1 to repair double-strand breaks in mitosis

Nature (London), 2024-02, Vol.626 (7999), p.E13-E13

2024

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Measurement of nucleotide excision repair activity and correlation with cisplatin sensitivity

Journal of clinical oncology, 2015-05, Vol.33 (15_suppl), p.1077-1077

2015

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Abstract POSTER-BIOL-1331: Nucleotide excision repair mutations confer sensitivity to platinum but not PARP-inhibitors in epithelial ovarian cancer

Clinical cancer research, 2015-08, Vol.21 (16_Supplement), p.POSTER-BIOL-1331-POSTER-BIOL-1331

2015

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Biallelic inactivation of REV7 is associated with Fanconi anemia

The Journal of clinical investigation, 2017-03, Vol.127 (3), p.1117-1117

2017

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Homologous-recombination-deficient tumours are dependent on Polh-mediated repair

Nature (London), 2015-02, Vol.518 (7538), p.258

2015

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Homologous-recombination-deficient tumours are dependent on Pol[theta]-mediated repair

Nature (London), 2015-02, Vol.518 (7538), p.258

2015

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EZH2 promotes degradation of stalled replication forks by recruiting MUS81 through histone H3 trimethylation

Repair Pathway Choices and Consequences at the Double-Strand Break

A prospective study of bone marrow hematopoietic and mesenchymal stem cells in type 1 Gaucher disease patients

Homologous Recombination Deficiency: Exploiting the Fundamental Vulnerability of Ovarian Cancer

Gene polymorphisms and cytokine plasma levels as predictive factors of complications after cardiopulmonary bypass

FANCD2 Maintains Fork Stability in BRCA1/2-Deficient Tumors and Promotes Alternative End-Joining DNA Repair

A first-in-class Polymerase Theta Inhibitor selectively targets Homologous-Recombination-Deficient Tumors

Transcriptional Repressor ZBTB1 Promotes Chromatin Remodeling and Translesion DNA Synthesis

The Fanconi anaemia pathway: new players and new functions

Spontaneous abrogation of the G2 DNA damage checkpoint has clinical benefits but promotes leukemogenesis in Fanconi anemia patients

Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair

DNA damage induces nuclear envelope rupture through ATR-mediated phosphorylation of lamin A/C

DNA damage induces nuclear envelope rupture through ATR-mediated phosphorylation of lamin A/C

Polθ is phosphorylated by PLK1 to repair double-strand breaks in mitosis

CtIP-Mediated Fork Protection Synergizes with BRCA1 to Suppress Genomic Instability upon DNA Replication Stress

Biallelic inactivation of REV7 is associated with Fanconi anemia

A unique subset of epithelial ovarian cancers with platinum sensitivity and PARP inhibitor resistance

Publisher Correction: Polθ is phosphorylated by PLK1 to repair double-strand breaks in mitosis

Bone Marrow Failure in Fanconi Anemia Is Triggered by an Exacerbated p53/p21 DNA Damage Response that Impairs Hematopoietic Stem and Progenitor Cells

Myelodysplasia and leukemia of Fanconi anemia are associated with a specific pattern of genomic abnormalities that includes cryptic RUNX1/AML1 lesions

Measurement of nucleotide excision repair activity and correlation with cisplatin sensitivity

Abstract POSTER-BIOL-1331: Nucleotide excision repair mutations confer sensitivity to platinum but not PARP-inhibitors in epithelial ovarian cancer

Biallelic inactivation of REV7 is associated with Fanconi anemia

Homologous-recombination-deficient tumours are dependent on Polh-mediated repair

Homologous-recombination-deficient tumours are dependent on Pol[theta]-mediated repair

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