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Counter-regulatory function of protein tyrosine phosphatase 1B in platelet-derived growth factor- or fibroblast growth factor-induced motility and proliferation of cultured smooth muscle cells and in neointima formation

Arteriosclerosis, thrombosis, and vascular biology, 2006-03, Vol.26 (3), p.501-507

2006

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Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas

American Journal of Physiology: Cell Physiology, 2006-04, Vol.290 (4), p.C1263

2006

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Gab1, SHP2, and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress

Circulation research, 2005-12, Vol.97 (12), p.1236-1244

2005

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RhoA and Rac1 are both required for efficient wound closure of airway epithelial cells

American journal of physiology. Lung cellular and molecular physiology, 2004-12, Vol.287 (6), p.L1134-L1144

2004

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Chronic insulin treatment amplifies PDGF-induced motility in differentiated aortic smooth muscle cells by suppressing the expression and function of PTP1B

American journal of physiology. Heart and circulatory physiology, 2008-07, Vol.295 (1), p.H163-H173

2008

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Nitric Oxide-Induced Motility in Aortic Smooth Muscle Cells: Role of Protein Tyrosine Phosphatase SHP-2 and GTP-Binding Protein Rho

Circulation research, 2002-09, Vol.91 (5), p.390-397

2002

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Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP

American journal of physiology. Heart and circulatory physiology, 2004-06, Vol.286 (6), p.H2103

2004

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Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas

American Journal of Physiology: Cell Physiology, 2006-04, Vol.59 (4), p.C1263

2006

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Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

American journal of physiology. Heart and circulatory physiology, 2005-04, Vol.288 (4), p.H1859-H1866

2005

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Regulation of cell motility by tyrosine phosphorylated villin

Molecular biology of the cell, 2004-11, Vol.15 (11), p.4807-4817

2004

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Essential role of protein kinase G and decreased cytoplasmic Ca 2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

American journal of physiology. Heart and circulatory physiology, 2005-04, Vol.288 (4), p.H1859-H1866

2005

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Essential role of protein kinase G and decreased cytoplasmic Ca super(2+) levels in NO-induced inhibition of rat aortic smooth muscle cell motility

American journal of physiology. Heart and circulatory physiology, 2005-04, Vol.288 (4), p.H1859-H1866

2005

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Chronic insulin treatment amplifies PDGF-induced motility in differentiated aortic smooth muscle cells by suppressing the expression and function of PTP1B

American journal of physiology. Heart and circulatory physiology, 2008, Vol.64 (1)

2008

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Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

American journal of physiology. Heart and circulatory physiology, 2005-04, Vol.57 (4), p.H1859-H1866

2005

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Treatment With Insulin Uncovers the Motogenic Capacity of Nitric Oxide in Aortic Smooth Muscle Cells: Dependence on Gab1 and Gab1-SHP2 Association

Circulation research, 2003-11, Vol.93 (10), p.e113-e123

2003

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Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130 cas

American Journal of Physiology: Cell Physiology, 2006-04, Vol.290 (4), p.C1263-C1270

2006

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Distribution of D1S80 Alleles and Their Tsp509I Subtypes in Romanian Population

Journal of forensic sciences, 2004-07, Vol.49 (4), p.1-3

2004

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RhoA and Rac1 are both required for efficient wound closure of airway epithelial cells

American journal of physiology. Lung cellular and molecular physiology, 2004-12, Vol.31 (6), p.L1134

2004

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956. Suppression of Tumor Metastasis by Adenoviral-Mediated MRP-1/CD9 Expression

Molecular therapy, 2004-05, Vol.9 (S1), p.S365-S366

2004

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Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP

American journal of physiology. Heart and circulatory physiology, 2004-06, Vol.55 (6), p.2103-2112

2004

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Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H 2 O 2 levels: essential role of cGMP

American journal of physiology. Heart and circulatory physiology, 2004-06, Vol.286 (6), p.H2103-H2112

2004

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Regulation of Cell Motility by Tyrosine Phosphorylated VillinD

Molecular biology of the cell, 2004-11, Vol.15 (11), p.4807-4817

2004

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Counter-regulatory function of protein tyrosine phosphatase 1B in platelet-derived growth factor- or fibroblast growth factor-induced motility and proliferation of cultured smooth muscle cells and in neointima formation

Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas

Gab1, SHP2, and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress

RhoA and Rac1 are both required for efficient wound closure of airway epithelial cells

Chronic insulin treatment amplifies PDGF-induced motility in differentiated aortic smooth muscle cells by suppressing the expression and function of PTP1B

Nitric Oxide-Induced Motility in Aortic Smooth Muscle Cells: Role of Protein Tyrosine Phosphatase SHP-2 and GTP-Binding Protein Rho

Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP

Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas

Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

Regulation of cell motility by tyrosine phosphorylated villin

Essential role of protein kinase G and decreased cytoplasmic Ca 2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

Essential role of protein kinase G and decreased cytoplasmic Ca super(2+) levels in NO-induced inhibition of rat aortic smooth muscle cell motility

Chronic insulin treatment amplifies PDGF-induced motility in differentiated aortic smooth muscle cells by suppressing the expression and function of PTP1B

Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility

Treatment With Insulin Uncovers the Motogenic Capacity of Nitric Oxide in Aortic Smooth Muscle Cells: Dependence on Gab1 and Gab1-SHP2 Association

Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130 cas

Distribution of D1S80 Alleles and Their Tsp509I Subtypes in Romanian Population

RhoA and Rac1 are both required for efficient wound closure of airway epithelial cells

956. Suppression of Tumor Metastasis by Adenoviral-Mediated MRP-1/CD9 Expression

Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP

Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H 2 O 2 levels: essential role of cGMP

Regulation of Cell Motility by Tyrosine Phosphorylated VillinD

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