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Nitric oxide in shock

Kidney international, 2007-09, Vol.72 (5), p.557-565

Cauwels, A.

2007

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Extracellular ATP drives systemic inflammation, tissue damage and mortality

Cell death & disease, 2014-03, Vol.5 (3), p.e1102-e1102

2014

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TLR-2 and TLR-9 are sensors of apoptosis in a mouse model of doxorubicin-induced acute inflammation

Cell death and differentiation, 2011-08, Vol.18 (8), p.1316-1325

2011

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Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models

Cell death & disease, 2012-11, Vol.3 (11), p.e437-e437

2012

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Simultaneous targeting of interleukin-1 and interleukin-18 is required for protection against inflammatory and septic shock

Critical care (London, England), 2014-12, Vol.18 (S2), p.P20-P20, Article P20

2014

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RIP Kinase-Dependent Necrosis Drives Lethal Systemic Inflammatory Response Syndrome

Immunity (Cambridge, Mass.), 2011-12, Vol.35 (6), p.908-918

2011

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Dual role of endogenous nitric oxide in tumor necrosis factor shock: induced NO tempers oxidative stress

Cellular and molecular life sciences : CMLS, 2005-07, Vol.62 (14), p.1632-1640

2005

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Nitrite regulation of shock

Cardiovascular research, 2011-02, Vol.89 (3), p.553-559

2011

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Anaphylactic shock depends on PI3K and eNOS-derived NO

The Journal of clinical investigation, 2006-08, Vol.116 (8), p.2244-2251

2006

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Coexistence of CD14-dependent and independent pathways for stimulation of human monocytes by gram-positive bacteria

Infection and Immunity, 1997-08, Vol.65 (8), p.3255-3260

1997

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Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2

Nature immunology, 2003-04, Vol.4 (4), p.387-393

2003

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Is There NO Treatment For Severe Sepsis?

Libyan journal of medicine, 2008-01, Vol.3 (1), p.34-38

2008

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Is There NO Treatment for Severe Sepsis?

Libyan journal of medicine, 2008-03, Vol.3 (1), p.34-38

2008

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New insights in the contribution and origin of reactive oxygen species in inflammation-induced mitochondrial damage, cell death, cardiovascular failure and shock

Free radical biology & medicine, 2012-09, Vol.53, p.S113-S114

2012

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Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2

Inflammation research, 2003-07, Vol.52 (S2), p.S178-S182

2003

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Hypoxic NO-donor nitrite protects sGC-dependently against morbidity and mortality associated with sterile inflammatory shock in mice

Critical care (London, England), 2011-01, Vol.15 (S1), p.P257-P257, Article P257

2011

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Nitric oxide synthase 2-derived NO in endotoxemia is not sufficient to cause hemodynamic effects

Critical care (London, England), 2008, Vol.12 (Suppl 2), p.P392-P392

2008

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Identification of a Streptococcus pneumoniae Gene Locus Encoding Proteins of an ABC Phosphate Transporter and a Two-Component Regulatory System

Journal of Bacteriology, 1999-02, Vol.181 (4), p.1126-1133

1999

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The Origin and Function of Soluble CD14 in Experimental Bacterial Meningitis

The Journal of immunology (1950), 1999-04, Vol.162 (8), p.4762-4772

1999

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Protection against TNF-Induced Lethal Shock by Soluble Guanylate Cyclase Inhibition Requires Functional Inducible Nitric Oxide Synthase

Immunity (Cambridge, Mass.), 2000-08, Vol.13 (2), p.223-231

2000

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0521. A functional citrulline-arginine-no pathway and nos3 complex is essential to maintain microcirculatory function during endotoxemia

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P30

2014

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Is there NO treatment for severe sepsis?

Libyan journal of medicine, 2008-01, Vol.3 (1), p.AOP:071018

2008

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Response of interleukin‐6‐deficient mice to tumor necrosis factor‐induced metabolic changes and lethality

European journal of immunology, 1994-09, Vol.24 (9), p.2237-2242

1994

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Tumor necrosis factor, its receptors and the connection with interleukin 1 and interleukin 6

Immunobiology (1979), 1993-04, Vol.187 (3-5), p.317-329

1993

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Murine IL-12 is involved in Calmette-Guérin bacillus-induced sensitization and is by itself sufficient to sensitize mice to the lethal effects of human TNF1

The Journal of immunology (1950), 1996-06, Vol.156 (12), p.4686-4690

1996

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Nitric oxide in shock

Extracellular ATP drives systemic inflammation, tissue damage and mortality

TLR-2 and TLR-9 are sensors of apoptosis in a mouse model of doxorubicin-induced acute inflammation

Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models

Simultaneous targeting of interleukin-1 and interleukin-18 is required for protection against inflammatory and septic shock

RIP Kinase-Dependent Necrosis Drives Lethal Systemic Inflammatory Response Syndrome

Dual role of endogenous nitric oxide in tumor necrosis factor shock: induced NO tempers oxidative stress

Nitrite regulation of shock

Anaphylactic shock depends on PI3K and eNOS-derived NO

Coexistence of CD14-dependent and independent pathways for stimulation of human monocytes by gram-positive bacteria

Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2

Is There NO Treatment For Severe Sepsis?

Is There NO Treatment for Severe Sepsis?

New insights in the contribution and origin of reactive oxygen species in inflammation-induced mitochondrial damage, cell death, cardiovascular failure and shock

Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2

Hypoxic NO-donor nitrite protects sGC-dependently against morbidity and mortality associated with sterile inflammatory shock in mice

Nitric oxide synthase 2-derived NO in endotoxemia is not sufficient to cause hemodynamic effects

Identification of a Streptococcus pneumoniae Gene Locus Encoding Proteins of an ABC Phosphate Transporter and a Two-Component Regulatory System

The Origin and Function of Soluble CD14 in Experimental Bacterial Meningitis

Protection against TNF-Induced Lethal Shock by Soluble Guanylate Cyclase Inhibition Requires Functional Inducible Nitric Oxide Synthase

0521. A functional citrulline-arginine-no pathway and nos3 complex is essential to maintain microcirculatory function during endotoxemia

Is there NO treatment for severe sepsis?

Response of interleukin‐6‐deficient mice to tumor necrosis factor‐induced metabolic changes and lethality

Tumor necrosis factor, its receptors and the connection with interleukin 1 and interleukin 6

Murine IL-12 is involved in Calmette-Guérin bacillus-induced sensitization and is by itself sufficient to sensitize mice to the lethal effects of human TNF1

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