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Disruption of an SF2/ASF-dependent exonic splicing enhancer in SMN2 causes spinal muscular atrophy in the absence of SMN1

Nature genetics, 2002-04, Vol.30 (4), p.377-384

2002

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Antitumorigenic potential of STAT3 alternative splicing modulation

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (43), p.17779-17784

2011

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Splicing factor hnRNPH drives an oncogenic splicing switch in gliomas

The EMBO journal, 2011-10, Vol.30 (19), p.4084-4097

2011

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5' UTR control of native ERG and of Tmprss2:ERG variants activity in prostate cancer

PloS one, 2013-03, Vol.8 (3), p.e49721-e49721

2013

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Determinants of Exon 7 Splicing in the Spinal Muscular Atrophy Genes, SMN1 and SMN2

American journal of human genetics, 2006-01, Vol.78 (1), p.63-77

2006

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Induction of Antagonistic Soluble Decoy Receptor Tyrosine Kinases by Intronic PolyA Activation

Molecular cell, 2011-09, Vol.43 (6), p.927-939

2011

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Correction of disease-associated exon skipping by synthetic exon-specific activators

Nature structural & molecular biology, 2003-02, Vol.10 (2), p.120-125

2003

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p53Ψ is a transcriptionally inactive p53 isoform able to reprogram cells toward a metastatic-like state

Proceedings of the National Academy of Sciences - PNAS, 2014-08, Vol.111 (32), p.E3287-E3296

2014

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U1 snRNP-Dependent Suppression of Polyadenylation: Physiological Role and Therapeutic Opportunities in Cancer

International Journal of Cell Biology, 2013, Vol.2013 (2013), p.600-609

2013

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Exploring Pharmacological Functions of Alternatively Spliced Variants of the Mu Opioid Receptor Gene, Oprm1 , via Gene-Targeted Animal Models

International journal of molecular sciences, 2022-03, Vol.23 (6), p.3010

2022

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Therapeutic hemoglobin levels after gene transfer in β-thalassemia mice and in hematopoietic cells of β-thalassemia and sickle cells disease patients

PloS one, 2012-03, Vol.7 (3), p.e32345-e32345

2012

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Alternative Pre-mRNA Splicing of the Mu Opioid Receptor Gene, OPRM1 : Insight into Complex Mu Opioid Actions

Biomolecules (Basel, Switzerland), 2021-10, Vol.11 (10), p.1525

2021

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A mechanism for exon skipping caused by nonsense or missense mutations in BRCA1 and other genes

Nature genetics, 2001, Vol.27 (1), p.55-58

2001

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Peptide-conjugated antisense oligonucleotides for targeted inhibition of a transcriptional regulator in vivo

Nature biotechnology, 2008-01, Vol.26 (1), p.91-100

2008

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Seemingly Neutral Polymorphic Variants May Confer Immunity to Splicing-Inactivating Mutations: A Synonymous SNP in Exon 5 of MCAD Protects from Deleterious Mutations in a Flanking Exonic Splicing Enhancer

American journal of human genetics, 2007-03, Vol.80 (3), p.416-432

2007

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Abstract 4780: An antisense-based approach to induce anti-tumorigenic variants of MET receptor

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.4780-4780

2019

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Abstract 2642: Antisense oligonucleotide-based induction of dominant-negative variants of STAT3 and EGFR in breast and lung cancer cells

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.2642-2642

2019

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Antisense modulation of RNA processing as a therapeutic approach in cancer therapy

Drug discovery today. Therapeutic strategies, 2013, Vol.10 (3), p.e139-e148

2013

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Exonic Splicing Enhancer Motif Recognized by Human SC35 under Splicing Conditions

Molecular and Cellular Biology, 2000-02, Vol.20 (3), p.1063-1071

2000

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Abstract LB-318: Reshaping the cancer proteome: therapeutic targeting of signaling and resistance in cancer by antisense manipulation of RNA processing

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.LB-318-LB-318

2013

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Abstract LB-239: Induction of therapeutic soluble decoy EGFR variants by antisense manipulation of RNA processing

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.LB-239-LB-239

2016

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Abstract 1836: Induction of secreted soluble decoy EGFR variants by splicing interference overcomes drug resistance in human non-small cell lung cancer

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.1836-1836

2018

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A novel hnRNP protein (HAP/SAF-B) enters a subset of hnRNP complexes and relocates in nuclear granules in response to heat shock

Journal of cell science, 1999-05, Vol.112 ( Pt 10) (10), p.1465-1476

1999

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ESEfinder: A web resource to identify exonic splicing enhancers

Nucleic acids research, 2003-07, Vol.31 (13), p.3568-3571

2003

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Listening to silence and understanding nonsense: exonic mutations that affect splicing

Nature reviews. Genetics, 2002-04, Vol.3 (4), p.285-298

2002

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Disruption of an SF2/ASF-dependent exonic splicing enhancer in SMN2 causes spinal muscular atrophy in the absence of SMN1

Antitumorigenic potential of STAT3 alternative splicing modulation

Splicing factor hnRNPH drives an oncogenic splicing switch in gliomas

5' UTR control of native ERG and of Tmprss2:ERG variants activity in prostate cancer

Determinants of Exon 7 Splicing in the Spinal Muscular Atrophy Genes, SMN1 and SMN2

Induction of Antagonistic Soluble Decoy Receptor Tyrosine Kinases by Intronic PolyA Activation

Correction of disease-associated exon skipping by synthetic exon-specific activators

p53Ψ is a transcriptionally inactive p53 isoform able to reprogram cells toward a metastatic-like state

U1 snRNP-Dependent Suppression of Polyadenylation: Physiological Role and Therapeutic Opportunities in Cancer

Exploring Pharmacological Functions of Alternatively Spliced Variants of the Mu Opioid Receptor Gene, Oprm1 , via Gene-Targeted Animal Models

Therapeutic hemoglobin levels after gene transfer in β-thalassemia mice and in hematopoietic cells of β-thalassemia and sickle cells disease patients

Alternative Pre-mRNA Splicing of the Mu Opioid Receptor Gene, OPRM1 : Insight into Complex Mu Opioid Actions

A mechanism for exon skipping caused by nonsense or missense mutations in BRCA1 and other genes

Peptide-conjugated antisense oligonucleotides for targeted inhibition of a transcriptional regulator in vivo

Seemingly Neutral Polymorphic Variants May Confer Immunity to Splicing-Inactivating Mutations: A Synonymous SNP in Exon 5 of MCAD Protects from Deleterious Mutations in a Flanking Exonic Splicing Enhancer

Abstract 4780: An antisense-based approach to induce anti-tumorigenic variants of MET receptor

Abstract 2642: Antisense oligonucleotide-based induction of dominant-negative variants of STAT3 and EGFR in breast and lung cancer cells

Antisense modulation of RNA processing as a therapeutic approach in cancer therapy

Exonic Splicing Enhancer Motif Recognized by Human SC35 under Splicing Conditions

Abstract LB-318: Reshaping the cancer proteome: therapeutic targeting of signaling and resistance in cancer by antisense manipulation of RNA processing

Abstract LB-239: Induction of therapeutic soluble decoy EGFR variants by antisense manipulation of RNA processing

Abstract 1836: Induction of secreted soluble decoy EGFR variants by splicing interference overcomes drug resistance in human non-small cell lung cancer

A novel hnRNP protein (HAP/SAF-B) enters a subset of hnRNP complexes and relocates in nuclear granules in response to heat shock

ESEfinder: A web resource to identify exonic splicing enhancers

Listening to silence and understanding nonsense: exonic mutations that affect splicing

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