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Modeling tumors of the peripheral nervous system associated with Neurofibromatosis type 1: Reprogramming plexiform neurofibroma cells

Stem cell research, 2020-12, Vol.49, p.102068-102068, Article 102068

2020

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Using antisense oligonucleotides for the physiological modulation of the alternative splicing of NF1 exon 23a during PC12 neuronal differentiation

Scientific reports, 2021-02, Vol.11 (1), p.3661-3661, Article 3661

2021

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Characterization of pluripotent stem cells

Nature protocols, 2013-02, Vol.8 (2), p.223-253

2013

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Reprogramming Captures the Genetic and Tumorigenic Properties of Neurofibromatosis Type 1 Plexiform Neurofibromas

Stem cell reports, 2019-02, Vol.12 (2), p.411-426

2019

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Expanding a precision medicine platform for malignant peripheral nerve sheath tumors: New patient‐derived orthotopic xenografts, cell lines and tumor entities

Molecular oncology, 2024-04, Vol.18 (4), p.895-917

2024

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Modeling iPSC-derived human neurofibroma-like tumors in mice uncovers the heterogeneity of Schwann cells within plexiform neurofibromas

Cell reports (Cambridge), 2022-02, Vol.38 (7), p.110385-110385, Article 110385

2022

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Generation of human iPSC-derived neurofibromaspheres for in vitro and in vivo uses

STAR protocols, 2023-06, Vol.4 (2), p.102198-102198, Article 102198

2023

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Unbalancing cAMP and Ras/MAPK pathways as a therapeutic strategy for cutaneous neurofibromas

JCI insight, 2024-01, Vol.9 (3)

2024

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Connexin-26 is a key factor mediating gemcitabine bystander effect

Molecular cancer therapeutics, 2011-03, Vol.10 (3), p.505-517

2011

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Reprogramming Captures the Genetic and Tumorigenic Properties of Neurofibromatosis Type 1 Plexiform Neurofibromas

Stem cell reports, 2019-03, Vol.12 (3), p.639-641

2019

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Chromosomal translocations inactivating CDKN2A support a single path for malignant peripheral nerve sheath tumor initiation

Human genetics, 2021-08, Vol.140 (8), p.1241-1252

2021

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Analysis of intratumor heterogeneity in Neurofibromatosis type 1 plexiform neurofibromas and neurofibromas with atypical features: Correlating histological and genomic findings

Human mutation, 2018-08, Vol.39 (8), p.1112-1125

2018

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Homeobox D10 induces phenotypic reversion of breast tumor cells in a three-dimensional culture model

Cancer research (Chicago, Ill.), 2005-08, Vol.65 (16), p.7177-7185

2005

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HoxA5 Stabilizes Adherens Junctions Via Increased Akt1

Cell adhesion & migration, 2007-10, Vol.1 (4), p.185-195

2007

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Retrovirus‐mediated transfer of the herpes simplex virus thymidine kinase and connexin26 genes in pancreatic cells results in variable efficiency on the bystander killing: Implications for gene therapy

International journal of cancer, 2001-10, Vol.94 (1), p.81-88

2001

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Intratumoral activation of cyclophosphamide by retroviral transfer of the cytochrome P450 2B1 in a pancreatic tumor model. Combination with the HSVtk/GCV system

The journal of gene medicine, 2002-03, Vol.4 (2), p.141-149

2002

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Information and communication technologies for approaching smokers: a descriptive study in primary healthcare

BMC public health, 2015-02, Vol.15 (1), p.2-14 pages, Article 2

2015

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Prognostic Performance of the Cytogenetic Risk Classification Defined by the German-Austrian Myelodysplastic Syndromes (MDS) Study Group and Its Contribution to the International Prognostic Scoring System (IPSS) and the World Health Organization-Based Prognostic Scoring System (WPSS)

Blood, 2010-11, Vol.116 (21), p.4005-4005

2010

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Transfusion intensity, not the cumulative red blood cell transfusion burden, determines the prognosis of patients with myelodysplastic syndrome on chronic transfusion support

American journal of hematology, 2011-03, Vol.86 (3), p.245-250

2011

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Refining the Breakpoints of Three New Translocations Identified in Myelodysplastic Syndromes

Acta haematologica, 2016-01, Vol.135 (2), p.94-100

2016

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Modeling tumors of the peripheral nervous system associated with Neurofibromatosis type 1: Reprogramming plexiform neurofibroma cells

Using antisense oligonucleotides for the physiological modulation of the alternative splicing of NF1 exon 23a during PC12 neuronal differentiation

Characterization of pluripotent stem cells

Reprogramming Captures the Genetic and Tumorigenic Properties of Neurofibromatosis Type 1 Plexiform Neurofibromas

Expanding a precision medicine platform for malignant peripheral nerve sheath tumors: New patient‐derived orthotopic xenografts, cell lines and tumor entities

Modeling iPSC-derived human neurofibroma-like tumors in mice uncovers the heterogeneity of Schwann cells within plexiform neurofibromas

Generation of human iPSC-derived neurofibromaspheres for in vitro and in vivo uses

Deep genomic analysis of malignant peripheral nerve sheath tumor cell lines challenges current malignant peripheral nerve sheath tumor diagnosis

Unbalancing cAMP and Ras/MAPK pathways as a therapeutic strategy for cutaneous neurofibromas

Connexin-26 is a key factor mediating gemcitabine bystander effect

Reprogramming Captures the Genetic and Tumorigenic Properties of Neurofibromatosis Type 1 Plexiform Neurofibromas

Chromosomal translocations inactivating CDKN2A support a single path for malignant peripheral nerve sheath tumor initiation

Analysis of intratumor heterogeneity in Neurofibromatosis type 1 plexiform neurofibromas and neurofibromas with atypical features: Correlating histological and genomic findings

Homeobox D10 induces phenotypic reversion of breast tumor cells in a three-dimensional culture model

HoxA5 Stabilizes Adherens Junctions Via Increased Akt1

Retrovirus‐mediated transfer of the herpes simplex virus thymidine kinase and connexin26 genes in pancreatic cells results in variable efficiency on the bystander killing: Implications for gene therapy

Intratumoral activation of cyclophosphamide by retroviral transfer of the cytochrome P450 2B1 in a pancreatic tumor model. Combination with the HSVtk/GCV system

Information and communication technologies for approaching smokers: a descriptive study in primary healthcare

Prognostic Performance of the Cytogenetic Risk Classification Defined by the German-Austrian Myelodysplastic Syndromes (MDS) Study Group and Its Contribution to the International Prognostic Scoring System (IPSS) and the World Health Organization-Based Prognostic Scoring System (WPSS)

Prognostic impact of chromosomal translocations in myelodysplastic syndromes and chronic myelomonocytic leukemia patients. A study by the spanish group of myelodysplastic syndromes

Transfusion intensity, not the cumulative red blood cell transfusion burden, determines the prognosis of patients with myelodysplastic syndrome on chronic transfusion support

Refining the Breakpoints of Three New Translocations Identified in Myelodysplastic Syndromes

Reciprocal translocations in myelodysplastic syndromes and chronic myelomonocytic leukemias: Review of 5,654 patients with an evaluable karyotype

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