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ICP0 antagonizes Stat 1-dependent repression of herpes simplex virus: implications for the regulation of viral latency

Virology journal, 2006-06, Vol.3 (1), p.44-44

2006

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Complement and CD4 + T cells drive context-specific corneal sensory neuropathy

eLife, 2019-08, Vol.8

2019

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Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency

The Journal of immunology (1950), 2016-08, Vol.197 (4), p.1262-1275

2016

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Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells

Glia, 2014-09, Vol.62 (9), p.1418-1434

2014

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Type I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental Malaria

PLoS pathogens, 2016-10, Vol.12 (10), p.e1005945-e1005945

2016

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Lack of neonatal Fc receptor does not diminish the efficacy of the HSV-1 0ΔNLS vaccine against ocular HSV-1 challenge

Vaccine, 2021-04, Vol.39 (18), p.2526-2536

2021

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Herpes simplex type I (HSV-1) infection of the nervous system: Is an immune response a good thing?

Journal of neuroimmunology, 2010-03, Vol.220 (1), p.1-9

2010

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Microglia and a functional type I IFN pathway are required to counter HSV-1-driven brain lateral ventricle enlargement and encephalitis

The Journal of immunology (1950), 2013-03, Vol.190 (6), p.2807-2817

2013

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Tumor necrosis factor alpha and interleukin-6 facilitate corneal lymphangiogenesis in response to herpes simplex virus 1 infection

Journal of virology, 2014-12, Vol.88 (24), p.14451-14457

2014

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Long‐term consequences of topical dexamethasone treatment during acute corneal HSV‐1 infection on the immune system

Journal of leukocyte biology, 2017-05, Vol.101 (5), p.1253-1261

2017

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Herpesvirus-Associated Lymphadenitis Distorts Fibroblastic Reticular Cell Microarchitecture and Attenuates CD8 T Cell Responses to Neurotropic Infection in Mice Lacking the STING-IFNα/β Defense Pathways

The Journal of immunology (1950), 2016-09, Vol.197 (6), p.2338-2352

2016

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A Highly Efficacious Herpes Simplex Virus 1 Vaccine Blocks Viral Pathogenesis and Prevents Corneal Immunopathology via Humoral Immunity

Journal of virology, 2016-06, Vol.90 (11), p.5514-5529

2016

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Impact of Type I Interferon on the Safety and Immunogenicity of an Experimental Live-Attenuated Herpes Simplex Virus 1 Vaccine in Mice

Journal of virology, 2017-04, Vol.91 (7)

2017

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CXCL9 and CXCL10 Expression Are Critical for Control of Genital Herpes Simplex Virus Type 2 Infection through Mobilization of HSV-Specific CTL and NK Cells to the Nervous System

The Journal of immunology (1950), 2008-01, Vol.180 (2), p.1098-1106

2008

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CD8+ T cells suppress viral replication in the cornea but contribute to VEGF-C-induced lymphatic vessel genesis

The Journal of immunology (1950), 2012-07, Vol.189 (1), p.425-432

2012

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Susceptibility of CCR5-Deficient Mice to Genital Herpes Simplex Virus Type 2 Is Linked to NK Cell Mobilization

Journal of Virology, 2007-04, Vol.81 (8), p.3704-3713

2007

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The herpes simplex virus-1 transactivator infected cell protein-4 drives VEGF-A dependent neovascularization

PLoS pathogens, 2011-10, Vol.7 (10), p.e1002278-e1002278

2011

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The use of human cornea organotypic cultures to study herpes simplex virus type 1 (HSV-1)-induced inflammation

Graefe's archive for clinical and experimental ophthalmology, 2015-10, Vol.253 (10), p.1721-1728

2015

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Complement and CD4.sup.+ T cells drive context-specific corneal sensory neuropathy

eLife, 2019-08, Vol.8

2019

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Loss of the Type I Interferon Pathway Increases Vulnerability of Mice to Genital Herpes Simplex Virus 2 Infection

Journal of Virology, 2011-02, Vol.85 (4), p.1625-1633

2011

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The current state of vaccine development for ocular HSV-1 infection

Expert review of ophthalmology, 2015-04, Vol.10 (2), p.113-126

2015

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CXCL10 expressing hematopoietic-derived cells are requisite in defense against HSV-1 infection in the nervous system of CXCL10 deficient mice

Journal of neuroimmunology, 2011-05, Vol.234 (1), p.103-108

2011

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Critical role for the oligoadenylate synthetase/RNase L pathway in response to IFN-beta during acute ocular herpes simplex virus type 1 infection

The Journal of immunology (1950), 2005-07, Vol.175 (2), p.1100-1106

2005

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Abnormal immune response of CCR5-deficient mice to ocular infection with herpes simplex virus type 1

Journal of general virology, 2006-03, Vol.87 (3), p.489-499

2006

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An increase in herpes simplex virus type 1 in the anterior segment of the eye is linked to a deficiency in NK cell infiltration in mice deficient in CXCR3

Journal of interferon & cytokine research, 2008-04, Vol.28 (4), p.245-251

2008

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ICP0 antagonizes Stat 1-dependent repression of herpes simplex virus: implications for the regulation of viral latency

Complement and CD4 + T cells drive context-specific corneal sensory neuropathy

Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency

Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells

Type I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental Malaria

Lack of neonatal Fc receptor does not diminish the efficacy of the HSV-1 0ΔNLS vaccine against ocular HSV-1 challenge

Herpes simplex type I (HSV-1) infection of the nervous system: Is an immune response a good thing?

Microglia and a functional type I IFN pathway are required to counter HSV-1-driven brain lateral ventricle enlargement and encephalitis

Tumor necrosis factor alpha and interleukin-6 facilitate corneal lymphangiogenesis in response to herpes simplex virus 1 infection

Long‐term consequences of topical dexamethasone treatment during acute corneal HSV‐1 infection on the immune system

Herpesvirus-Associated Lymphadenitis Distorts Fibroblastic Reticular Cell Microarchitecture and Attenuates CD8 T Cell Responses to Neurotropic Infection in Mice Lacking the STING-IFNα/β Defense Pathways

A Highly Efficacious Herpes Simplex Virus 1 Vaccine Blocks Viral Pathogenesis and Prevents Corneal Immunopathology via Humoral Immunity

Impact of Type I Interferon on the Safety and Immunogenicity of an Experimental Live-Attenuated Herpes Simplex Virus 1 Vaccine in Mice

CXCL9 and CXCL10 Expression Are Critical for Control of Genital Herpes Simplex Virus Type 2 Infection through Mobilization of HSV-Specific CTL and NK Cells to the Nervous System

CD8+ T cells suppress viral replication in the cornea but contribute to VEGF-C-induced lymphatic vessel genesis

Susceptibility of CCR5-Deficient Mice to Genital Herpes Simplex Virus Type 2 Is Linked to NK Cell Mobilization

The herpes simplex virus-1 transactivator infected cell protein-4 drives VEGF-A dependent neovascularization

The use of human cornea organotypic cultures to study herpes simplex virus type 1 (HSV-1)-induced inflammation

Complement and CD4.sup.+ T cells drive context-specific corneal sensory neuropathy

Loss of the Type I Interferon Pathway Increases Vulnerability of Mice to Genital Herpes Simplex Virus 2 Infection

The current state of vaccine development for ocular HSV-1 infection

CXCL10 expressing hematopoietic-derived cells are requisite in defense against HSV-1 infection in the nervous system of CXCL10 deficient mice

Critical role for the oligoadenylate synthetase/RNase L pathway in response to IFN-beta during acute ocular herpes simplex virus type 1 infection

Abnormal immune response of CCR5-deficient mice to ocular infection with herpes simplex virus type 1

An increase in herpes simplex virus type 1 in the anterior segment of the eye is linked to a deficiency in NK cell infiltration in mice deficient in CXCR3

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