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The Proliferation-Quiescence Decision Is Controlled by a Bifurcation in CDK2 Activity at Mitotic Exit

Cell, 2013-10, Vol.155 (2), p.369-383

2013

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Irreversible APC(Cdh1) Inactivation Underlies the Point of No Return for Cell-Cycle Entry

Cell, 2016-06, Vol.166 (1), p.167-180

2016

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Transient Hysteresis in CDK4/6 Activity Underlies Passage of the Restriction Point in G1

Molecular cell, 2019-11, Vol.76 (4), p.562-573.e4

2019

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Ribosome biogenesis is a downstream effector of the oncogenic U2AF1-S34F mutation

PLoS biology, 2020-11, Vol.18 (11), p.e3000920

2020

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Revealing β-TrCP activity dynamics in live cells with a genetically encoded biosensor

Nature communications, 2022-10, Vol.13 (1), p.6364-6364, Article 6364

2022

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Stress-mediated exit to quiescence restricted by increasing persistence in CDK4/6 activation

eLife, 2020-04, Vol.9

2020

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Irreversible cell cycle exit associated with senescence is mediated by constitutive MYC degradation

Cell reports (Cambridge), 2023-09, Vol.42 (9), p.113079-113079, Article 113079

2023

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Nuclear pore protein NUP210 depletion suppresses metastasis through heterochromatin-mediated disruption of tumor cell mechanical response

Nature communications, 2021-12, Vol.12 (1), p.7216-7216, Article 7216

2021

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The RepID-CRL4 ubiquitin ligase complex regulates metaphase to anaphase transition via BUB3 degradation

Nature communications, 2020-01, Vol.11 (1), p.24-24, Article 24

2020

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EMI1 switches from being a substrate to an inhibitor of APC/CCDH1 to start the cell cycle

Nature (London), 2018-06, Vol.558 (7709), p.313-317

2018

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Regulators of G-protein Signaling accelerate GPCR signaling kinetics and govern sensitivity solely by accelerating GTPase activity

Proceedings of the National Academy of Sciences - PNAS, 2010-04, Vol.107 (15), p.7066-7071

2010

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EMI1 switches from being a substrate to an inhibitor of APC/C CDH1 to start the cell cycle

Nature (London), 2018-06, Vol.558 (7709), p.313

2018

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Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal

Nature (London), 2023-07, Vol.619 (7969), p.363-370

2023

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Author Correction: Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal

Nature (London), 2024-06, Vol.630 (8018), p.E14-E14

2024

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Selective role for RGS12 as a Ras/Raf/MEK scaffold in nerve growth factor-mediated differentiation

The EMBO journal, 2007-04, Vol.26 (8), p.2029-2040

2007

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Comprehensive mapping of cell fates in microsatellite unstable cancer cells supports dual targeting of WRN and ATR

Genes & development, 2023-10, Vol.37 (19-20), p.913-928

2023

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The adapter protein FADD provides an alternate pathway for entry into the cell cycle by regulating APC/C-Cdh1 E3 ubiquitin ligase activity

The Journal of biological chemistry, 2023-06, Vol.299 (6), p.104786, Article 104786

2023

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Abrogation of the G2/M checkpoint as a chemosensitization approach for alkylating agents

Neuro-oncology (Charlottesville, Va.), 2024-06, Vol.26 (6), p.1083-1096

2024

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Cell cycle inertia underlies a bifurcation in cell fates after DNA damage

Science advances, 2021-01, Vol.7 (3)

2021

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Irreversible APCCdh1 Inactivation Underlies the Point of No Return for Cell-Cycle Entry

Cell, 2016-06, Vol.166 (1), p.167-180

2016

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Apoptosis-induced nuclear expulsion in tumor cells drives S100a4-mediated metastatic outgrowth through the RAGE pathway

Nature cancer, 2023-03, Vol.4 (3), p.419

2023

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Systematic Analysis of Essential Genes Reveals Important Regulators of G Protein Signaling

Molecular cell, 2010-06, Vol.38 (5), p.746-757

2010

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Selective Regulation of MAP Kinase Signaling by an Endomembrane Phosphatidylinositol 4-Kinase

The Journal of biological chemistry, 2011-04, Vol.286 (17), p.14852-14860

2011

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Cell Cycle-dependent Phosphorylation and Ubiquitination of a G Protein α Subunit

The Journal of biological chemistry, 2011-06, Vol.286 (23), p.20208-20216

2011

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EMI1 switches from being a substrate to an inhibitor of APC/C^sup CDH1^ to start the cell cycle

Nature (London), 2018-06, Vol.558 (7709), p.313-317Q

2018

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The Proliferation-Quiescence Decision Is Controlled by a Bifurcation in CDK2 Activity at Mitotic Exit

Irreversible APC(Cdh1) Inactivation Underlies the Point of No Return for Cell-Cycle Entry

Transient Hysteresis in CDK4/6 Activity Underlies Passage of the Restriction Point in G1

Ribosome biogenesis is a downstream effector of the oncogenic U2AF1-S34F mutation

Revealing β-TrCP activity dynamics in live cells with a genetically encoded biosensor

Stress-mediated exit to quiescence restricted by increasing persistence in CDK4/6 activation

Irreversible cell cycle exit associated with senescence is mediated by constitutive MYC degradation

Nuclear pore protein NUP210 depletion suppresses metastasis through heterochromatin-mediated disruption of tumor cell mechanical response

The RepID-CRL4 ubiquitin ligase complex regulates metaphase to anaphase transition via BUB3 degradation

EMI1 switches from being a substrate to an inhibitor of APC/CCDH1 to start the cell cycle

Regulators of G-protein Signaling accelerate GPCR signaling kinetics and govern sensitivity solely by accelerating GTPase activity

EMI1 switches from being a substrate to an inhibitor of APC/C CDH1 to start the cell cycle

Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal

Author Correction: Loss of CDK4/6 activity in S/G2 phase leads to cell cycle reversal

Selective role for RGS12 as a Ras/Raf/MEK scaffold in nerve growth factor-mediated differentiation

Comprehensive mapping of cell fates in microsatellite unstable cancer cells supports dual targeting of WRN and ATR

The adapter protein FADD provides an alternate pathway for entry into the cell cycle by regulating APC/C-Cdh1 E3 ubiquitin ligase activity

Abrogation of the G2/M checkpoint as a chemosensitization approach for alkylating agents

Cell cycle inertia underlies a bifurcation in cell fates after DNA damage

Irreversible APCCdh1 Inactivation Underlies the Point of No Return for Cell-Cycle Entry

Apoptosis-induced nuclear expulsion in tumor cells drives S100a4-mediated metastatic outgrowth through the RAGE pathway

Systematic Analysis of Essential Genes Reveals Important Regulators of G Protein Signaling

Selective Regulation of MAP Kinase Signaling by an Endomembrane Phosphatidylinositol 4-Kinase

Cell Cycle-dependent Phosphorylation and Ubiquitination of a G Protein α Subunit

EMI1 switches from being a substrate to an inhibitor of APC/C^sup CDH1^ to start the cell cycle

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