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Chemokine contribution to neuropathic pain: Respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons

Pain (Amsterdam), 2013-10, Vol.154 (10), p.2185-2197

2013

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Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice

Journal of neuroinflammation, 2016-07, Vol.13 (1), p.183-183, Article 183

2016

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MicroRNA-146a-5p attenuates neuropathic pain via suppressing TRAF6 signaling in the spinal cord

Brain, behavior, and immunity, 2015-10, Vol.49, p.119-129

2015

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Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2

Experimental neurology, 2014-11, Vol.261, p.328-336

2014

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TRAF6 Contributes to CFA-Induced Spinal Microglial Activation and Chronic Inflammatory Pain in Mice

Cellular and molecular neurobiology, 2022-07, Vol.42 (5), p.1543-1555

2022

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Descending dopaminergic pathway facilitates itch signal processing via activating spinal GRPR + neurons

EMBO reports, 2023-10, Vol.24 (10), p.e56098

2023

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Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell

Neuropsychopharmacology (New York, N.Y.), 2018-10, Vol.43 (11), p.2320-2330

2018

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Decoding gene expression signatures in mice trigeminal ganglion across trigeminal neuropathic pain stages via high-throughput sequencing

Brain research bulletin, 2022-09, Vol.187, p.122-137

2022

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Nerve Injury-Induced γH2AX Reduction in Primary Sensory Neurons Is Involved in Neuropathic Pain Processing

International journal of molecular sciences, 2023-06, Vol.24 (12), p.10148

2023

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Chemokine CCL8 and its receptor CCR5 in the spinal cord are involved in visceral pain induced by experimental colitis in mice

Brain research bulletin, 2017-10, Vol.135, p.170-178

2017

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NFAT1 Orchestrates Spinal Microglial Transcription and Promotes Microglial Proliferation via c‐MYC Contributing to Nerve Injury‐Induced Neuropathic Pain

Advanced science, 2022-09, Vol.9 (27), p.e2201300-n/a

2022

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Identification of lncRNA expression profile in the spinal cord of mice following spinal nerve ligation-induced neuropathic pain

Molecular pain, 2015-07, Vol.11, p.43-43

2015

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Chemokine Receptor CXCR3 in the Spinal Cord Contributes to Chronic Itch in Mice

Neuroscience bulletin, 2018-02, Vol.34 (1), p.54-63

2018

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Ligustilide attenuates inflammatory pain via inhibition of NFκB-mediated chemokines production in spinal astrocytes

The European journal of neuroscience, 2014-04, Vol.39 (8), p.1391-1402

2014

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Descending Modulation of Spinal Itch Transmission by Primary Somatosensory Cortex

Neuroscience bulletin, 2021-09, Vol.37 (9), p.1345-1350

2021

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Activation of astrocytes in the anterior cingulate cortex contributes to the affective component of pain in an inflammatory pain model

Brain research bulletin, 2012-01, Vol.87 (1), p.60-66

2012

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Chemokine CXCL13 activates p38 MAPK in the trigeminal ganglion after infraorbital nerve injury

Inflammation, 2017-06, Vol.40 (3), p.762-769

2017

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CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5

The Journal of clinical investigation, 2016-02, Vol.126 (2), p.745-761

2016

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TLR8 and its endogenous ligand miR-21 contribute to neuropathic pain in murine DRG

The Journal of experimental medicine, 2018-12, Vol.215 (12), p.3019-3037

2018

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TRAF6 upregulation in spinal astrocytes maintains neuropathic pain by integrating TNF-α and IL-1β signaling

Pain (Amsterdam), 2014-12, Vol.155 (12), p.2618-2629

2014

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Demethylation of G-Protein-Coupled Receptor 151 Promoter Facilitates the Binding of Krüppel-Like Factor 5 and Enhances Neuropathic Pain after Nerve Injury in Mice

The Journal of neuroscience, 2018-12, Vol.38 (49), p.10535-10551

2018

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Chemokine receptor CXCR2 in dorsal root ganglion contributes to the maintenance of inflammatory pain

Brain research bulletin, 2016-10, Vol.127, p.219-225

2016

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Promoted Interaction of C/EBPα with Demethylated Cxcr3 Gene Promoter Contributes to Neuropathic Pain in Mice

The Journal of neuroscience, 2017-01, Vol.37 (3), p.685-700

2017

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CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory pain

Scientific reports, 2016-10, Vol.6 (1), p.34836-34836, Article 34836

2016

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MicroRNA-146a-5p attenuates visceral hypersensitivity through targeting chemokine CCL8 in the spinal cord in a mouse model of colitis

Brain research bulletin, 2018-05, Vol.139, p.235-242

2018

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Chemokine contribution to neuropathic pain: Respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons

Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice

MicroRNA-146a-5p attenuates neuropathic pain via suppressing TRAF6 signaling in the spinal cord

Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2

TRAF6 Contributes to CFA-Induced Spinal Microglial Activation and Chronic Inflammatory Pain in Mice

Descending dopaminergic pathway facilitates itch signal processing via activating spinal GRPR + neurons

Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell

Decoding gene expression signatures in mice trigeminal ganglion across trigeminal neuropathic pain stages via high-throughput sequencing

Nerve Injury-Induced γH2AX Reduction in Primary Sensory Neurons Is Involved in Neuropathic Pain Processing

Chemokine CCL8 and its receptor CCR5 in the spinal cord are involved in visceral pain induced by experimental colitis in mice

NFAT1 Orchestrates Spinal Microglial Transcription and Promotes Microglial Proliferation via c‐MYC Contributing to Nerve Injury‐Induced Neuropathic Pain

Identification of lncRNA expression profile in the spinal cord of mice following spinal nerve ligation-induced neuropathic pain

Chemokine Receptor CXCR3 in the Spinal Cord Contributes to Chronic Itch in Mice

Ligustilide attenuates inflammatory pain via inhibition of NFκB-mediated chemokines production in spinal astrocytes

Descending Modulation of Spinal Itch Transmission by Primary Somatosensory Cortex

Activation of astrocytes in the anterior cingulate cortex contributes to the affective component of pain in an inflammatory pain model

Chemokine CXCL13 activates p38 MAPK in the trigeminal ganglion after infraorbital nerve injury

CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5

TLR8 and its endogenous ligand miR-21 contribute to neuropathic pain in murine DRG

TRAF6 upregulation in spinal astrocytes maintains neuropathic pain by integrating TNF-α and IL-1β signaling

Demethylation of G-Protein-Coupled Receptor 151 Promoter Facilitates the Binding of Krüppel-Like Factor 5 and Enhances Neuropathic Pain after Nerve Injury in Mice

Chemokine receptor CXCR2 in dorsal root ganglion contributes to the maintenance of inflammatory pain

Promoted Interaction of C/EBPα with Demethylated Cxcr3 Gene Promoter Contributes to Neuropathic Pain in Mice

CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory pain

MicroRNA-146a-5p attenuates visceral hypersensitivity through targeting chemokine CCL8 in the spinal cord in a mouse model of colitis

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