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Macrophages in Renal Injury and Repair

Annual review of physiology, 2017-02, Vol.79 (1), p.449-469

2017

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Tubular GM-CSF Promotes Late MCP-1/CCR2-Mediated Fibrosis and Inflammation after Ischemia/Reperfusion Injury

Journal of the American Society of Nephrology, 2019-10, Vol.30 (10), p.1825-1840

2019

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Immune-mediated tubule atrophy promotes acute kidney injury to chronic kidney disease transition

Nature communications, 2022-08, Vol.13 (1), p.4892-4892, Article 4892

2022

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Distinct Macrophage Phenotypes Contribute to Kidney Injury and Repair

Journal of the American Society of Nephrology, 2011-02, Vol.22 (2), p.317-326

2011

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Macrophage-mediated injury and repair after ischemic kidney injury

Pediatric nephrology (Berlin, West), 2015-02, Vol.30 (2), p.199-209

2015

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Stromal cells protect against acute tubular injury via an endocrine effect

Journal of the American Society of Nephrology, 2007-09, Vol.18 (9), p.2486-2496

2007

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VCAM-1 mediates proximal tubule-immune cell cross talk in failed tubule recovery during AKI-to-CKD transition

American journal of physiology. Renal physiology, 2024-10, Vol.327 (4), p.F610-F622

2024

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Biomarkers of inflammation and repair in kidney disease progression

The Journal of clinical investigation, 2021-02, Vol.131 (3), p.1-10

2021

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Mcp1 Promotes Macrophage-Dependent Cyst Expansion in Autosomal Dominant Polycystic Kidney Disease

Journal of the American Society of Nephrology, 2018-10, Vol.29 (10), p.2471-2481

2018

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GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury

Journal of the American Society of Nephrology, 2015-06, Vol.26 (6), p.1334-1345

2015

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The impact of aging on kidney repair

American journal of physiology. Renal physiology, 2008-06, Vol.294 (6), p.F1265-F1272

2008

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Recovery of Kidney Function After Acute Kidney Injury in the Elderly: A Systematic Review and Meta-analysis

American journal of kidney diseases, 2008-08, Vol.52 (2), p.262-271

2008

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Macrophages Promote Cyst Growth in Polycystic Kidney Disease

Journal of the American Society of Nephrology, 2011-10, Vol.22 (10), p.1809-1814

2011

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Identification and validation of urinary CXCL9 as a biomarker for diagnosis of acute interstitial nephritis

The Journal of clinical investigation, 2023-07, Vol.133 (13)

2023

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Arginase-1 Is Required for Macrophage-Mediated Renal Tubule Regeneration

Journal of the American Society of Nephrology, 2022-06, Vol.33 (6), p.1077-1086

2022

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The Association of COVID-19 With Acute Kidney Injury Independent of Severity of Illness: A Multicenter Cohort Study

American journal of kidney diseases, 2021-04, Vol.77 (4), p.490-499.e1

2021

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Using Imaging Mass Cytometry to Define Cell Identities and Interactions in Human Tissues

Frontiers in physiology, 2021-12, Vol.12, p.817181-817181

2021

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Zag Expression during Aging Suppresses Proliferation after Kidney Injury

Journal of the American Society of Nephrology, 2008-12, Vol.19 (12), p.2375-2383

2008

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IL‐1β Drives Production of FGF‐23 at the Onset of Chronic Kidney Disease in Mice

Journal of bone and mineral research, 2020-07, Vol.35 (7), p.1352-1362

2020

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Introduction: Understanding Human AKI

Seminars in nephrology, 2018-01, Vol.38 (1), p.1-2

Cantley, Lloyd G

2018

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Cellular maintenance and repair of the kidney

Annual review of physiology, 2010-01, Vol.72 (1), p.357-376

2010

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Breast Regression Protein-39/Chitinase 3-Like 1 Promotes Renal Fibrosis after Kidney Injury via Activation of Myofibroblasts

Journal of the American Society of Nephrology, 2017-11, Vol.28 (11), p.3218-3226

2017

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Authors' Reply: Most Arginase-1 Positive Cells Are Likely Injured S3 Proximal Tubular Cells Carrying Upregulated Phagocytotic Capacity rather than M2 Macrophages-Too Many To Be True

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11), p.2124-2125

2022

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Characterization of temporospatial distribution of renal tubular casts by nephron tracking after ischemia-reperfusion injury

American journal of physiology. Renal physiology, 2022-03, Vol.322 (3), p.F322-F334

2022

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Cyst formation and activation of the extracellular regulated kinase pathway after kidney specific inactivation of Pkd1

Human molecular genetics, 2008-06, Vol.17 (11), p.1505-1516

2008

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Macrophages in Renal Injury and Repair

Tubular GM-CSF Promotes Late MCP-1/CCR2-Mediated Fibrosis and Inflammation after Ischemia/Reperfusion Injury

Immune-mediated tubule atrophy promotes acute kidney injury to chronic kidney disease transition

Distinct Macrophage Phenotypes Contribute to Kidney Injury and Repair

Macrophage-mediated injury and repair after ischemic kidney injury

Stromal cells protect against acute tubular injury via an endocrine effect

VCAM-1 mediates proximal tubule-immune cell cross talk in failed tubule recovery during AKI-to-CKD transition

Biomarkers of inflammation and repair in kidney disease progression

Mcp1 Promotes Macrophage-Dependent Cyst Expansion in Autosomal Dominant Polycystic Kidney Disease

GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury

The impact of aging on kidney repair

Recovery of Kidney Function After Acute Kidney Injury in the Elderly: A Systematic Review and Meta-analysis

Macrophages Promote Cyst Growth in Polycystic Kidney Disease

Identification and validation of urinary CXCL9 as a biomarker for diagnosis of acute interstitial nephritis

Arginase-1 Is Required for Macrophage-Mediated Renal Tubule Regeneration

The Association of COVID-19 With Acute Kidney Injury Independent of Severity of Illness: A Multicenter Cohort Study

Using Imaging Mass Cytometry to Define Cell Identities and Interactions in Human Tissues

Zag Expression during Aging Suppresses Proliferation after Kidney Injury

IL‐1β Drives Production of FGF‐23 at the Onset of Chronic Kidney Disease in Mice

Introduction: Understanding Human AKI

Cellular maintenance and repair of the kidney

Breast Regression Protein-39/Chitinase 3-Like 1 Promotes Renal Fibrosis after Kidney Injury via Activation of Myofibroblasts

Authors' Reply: Most Arginase-1 Positive Cells Are Likely Injured S3 Proximal Tubular Cells Carrying Upregulated Phagocytotic Capacity rather than M2 Macrophages-Too Many To Be True

Characterization of temporospatial distribution of renal tubular casts by nephron tracking after ischemia-reperfusion injury

Cyst formation and activation of the extracellular regulated kinase pathway after kidney specific inactivation of Pkd1

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