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Quantification of organelle contact sites by split-GFP-based contact site sensors (SPLICS) in living cells

Nature protocols, 2021-11, Vol.16 (11), p.5287-5308

2021

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Neuronal calcium signaling: function and dysfunction

Cellular and molecular life sciences : CMLS, 2014-08, Vol.71 (15), p.2787-2814

2014

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Mitochondria Associated Membranes (MAMs): Architecture and physiopathological role

Cell calcium (Edinburgh), 2021-03, Vol.94, p.102343-102343, Article 102343

2021

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PINK1/Parkin Mediated Mitophagy, Ca2+ Signalling, and ER–Mitochondria Contacts in Parkinson’s Disease

International journal of molecular sciences, 2020-03, Vol.21 (5), p.1772

2020

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SPLICS: a split green fluorescent protein-based contact site sensor for narrow and wide heterotypic organelle juxtaposition

Cell death and differentiation, 2018-06, Vol.25 (6), p.1131-1145

2018

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α-Synuclein Controls Mitochondrial Calcium Homeostasis by Enhancing Endoplasmic Reticulum-Mitochondria Interactions

The Journal of biological chemistry, 2012-05, Vol.287 (22), p.17914-17929

2012

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SERCA2 phosphorylation at the heart of the disease

Cell calcium (Edinburgh), 2023-11, Vol.115, p.102784-102784, Article 102784

2023

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Enhanced parkin levels favor ER-mitochondria crosstalk and guarantee Ca2+ transfer to sustain cell bioenergetics

Biochimica et biophysica acta. Molecular basis of disease, 2013-04, Vol.1832 (4), p.495-508

2013

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Protein synthesis inhibition and loss of homeostatic functions in astrocytes from an Alzheimer's disease mouse model: a role for ER-mitochondria interaction

Cell death & disease, 2022-10, Vol.13 (10), p.878-878, Article 878

2022

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Mitochondrial fission links ECM mechanotransduction to metabolic redox homeostasis and metastatic chemotherapy resistance

Nature cell biology, 2022-02, Vol.24 (2), p.168-180

2022

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The Close Encounter Between Alpha-Synuclein and Mitochondria

Frontiers in neuroscience, 2018-06, Vol.12, p.388-388

2018

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plasma membrane calcium pump in health and disease

The FEBS journal, 2013-11, Vol.280 (21), p.5385-5397

2013

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Calcium signaling in Parkinson’s disease

Cell and tissue research, 2014-08, Vol.357 (2), p.439-454

2014

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Emerging (and converging) pathways in Parkinson's disease: keeping mitochondrial wellness

Biochemical and biophysical research communications, 2017-02, Vol.483 (4), p.1020-1030

2017

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An expanded palette of improved SPLICS reporters detects multiple organelle contacts in vitro and in vivo

Nature communications, 2020-11, Vol.11 (1), p.6069-6069, Article 6069

2020

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The plasma membrane calcium pumps: focus on the role in (neuro)pathology

Biochemical and biophysical research communications, 2017-02, Vol.483 (4), p.1116-1124

2017

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The Parkinson disease-related protein DJ-1 counteracts mitochondrial impairment induced by the tumour suppressor protein p53 by enhancing endoplasmic reticulum-mitochondria tethering

Human molecular genetics, 2013-06, Vol.22 (11), p.2152-2168

2013

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ER-Mitochondria Contact Sites Reporters: Strengths and Weaknesses of the Available Approaches

International journal of molecular sciences, 2020-10, Vol.21 (21), p.8157

2020

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Organelles: The Emerging Signalling Chart of Mitochondrial Dynamics

Current biology, 2018-01, Vol.28 (2), p.R73-R75

2018

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Physiological cyanide concentrations do not stimulate mitochondrial cytochrome c oxidase activity

Proceedings of the National Academy of Sciences - PNAS, 2021-09, Vol.118 (39), p.1

2021

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Ca2+ handling at the mitochondria-ER contact sites in neurodegeneration

Cell calcium (Edinburgh), 2021-09, Vol.98, p.102453-102453, Article 102453

2021

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Alpha-synuclein at the intracellular and the extracellular side: functional and dysfunctional implications

Biological chemistry, 2017-01, Vol.398 (1), p.77-100

2017

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Mitochondrial Ca2+ and neurodegeneration

Cell calcium (Edinburgh), 2012-07, Vol.52 (1), p.73-85

2012

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Play Around with mtDNA

DNA and cell biology, 2020-08, Vol.39 (8), p.1369-1369

2020

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Tau localises within mitochondrial sub-compartments and its caspase cleavage affects ER-mitochondria interactions and cellular Ca2+ handling

Biochimica et biophysica acta. Molecular basis of disease, 2018-10, Vol.1864 (10), p.3247-3256

2018

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Quantification of organelle contact sites by split-GFP-based contact site sensors (SPLICS) in living cells

Neuronal calcium signaling: function and dysfunction

Mitochondria Associated Membranes (MAMs): Architecture and physiopathological role

PINK1/Parkin Mediated Mitophagy, Ca2+ Signalling, and ER–Mitochondria Contacts in Parkinson’s Disease

SPLICS: a split green fluorescent protein-based contact site sensor for narrow and wide heterotypic organelle juxtaposition

α-Synuclein Controls Mitochondrial Calcium Homeostasis by Enhancing Endoplasmic Reticulum-Mitochondria Interactions

SERCA2 phosphorylation at the heart of the disease

Enhanced parkin levels favor ER-mitochondria crosstalk and guarantee Ca2+ transfer to sustain cell bioenergetics

Protein synthesis inhibition and loss of homeostatic functions in astrocytes from an Alzheimer's disease mouse model: a role for ER-mitochondria interaction

Mitochondrial fission links ECM mechanotransduction to metabolic redox homeostasis and metastatic chemotherapy resistance

The Close Encounter Between Alpha-Synuclein and Mitochondria

plasma membrane calcium pump in health and disease

Calcium signaling in Parkinson’s disease

Emerging (and converging) pathways in Parkinson's disease: keeping mitochondrial wellness

An expanded palette of improved SPLICS reporters detects multiple organelle contacts in vitro and in vivo

The plasma membrane calcium pumps: focus on the role in (neuro)pathology

The Parkinson disease-related protein DJ-1 counteracts mitochondrial impairment induced by the tumour suppressor protein p53 by enhancing endoplasmic reticulum-mitochondria tethering

ER-Mitochondria Contact Sites Reporters: Strengths and Weaknesses of the Available Approaches

Organelles: The Emerging Signalling Chart of Mitochondrial Dynamics

Physiological cyanide concentrations do not stimulate mitochondrial cytochrome c oxidase activity

Ca2+ handling at the mitochondria-ER contact sites in neurodegeneration

Alpha-synuclein at the intracellular and the extracellular side: functional and dysfunctional implications

Mitochondrial Ca2+ and neurodegeneration

Play Around with mtDNA

Tau localises within mitochondrial sub-compartments and its caspase cleavage affects ER-mitochondria interactions and cellular Ca2+ handling

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