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The cellular mechanisms of neuronal swelling underlying cytotoxic edema

Cell, 2015-04, Vol.161 (3), p.610-621

2015

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Pregabalin as a Pain Therapeutic: Beyond Calcium Channels

Frontiers in cellular neuroscience, 2020-04, Vol.14, p.83-83

2020

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Contributions of T-type calcium channel isoforms to neuronal firing

Channels (Austin, Tex.), 2010-11, Vol.4 (6), p.475-482

2010

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Positive allosteric modulation of type 1 cannabinoid receptors reduces spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg

Neuropharmacology, 2021-06, Vol.190, p.108553, Article 108553

2021

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The T-type calcium channel antagonist Z944 rescues impairments in crossmodal and visual recognition memory in Genetic Absence Epilepsy Rats from Strasbourg

Neurobiology of disease, 2016-10, Vol.94, p.106-115

2016

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The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation

Cell reports (Cambridge), 2019-11, Vol.29 (8), p.2422-2437.e8

2019

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Image-Based Dosimetry in Dogs and Cross-Reactivity with Human Tissues of IGF2R-Targeting Human Antibody

Pharmaceuticals (Basel, Switzerland), 2023-07, Vol.16 (7), p.979

2023

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Chronic pregabalin treatment protects against spreading depolarization and alters hippocampal synaptic characteristics in a model of familial hemiplegic migraine-type 1

Molecular brain, 2023-11, Vol.16 (1), p.76-76, Article 76

2023

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Epigallocatechin-3-gallate elicits Ca2+ spike in MCF-7 breast cancer cells: Essential role of Cav3.2 channels

Cell calcium (Edinburgh), 2014-10, Vol.56 (4), p.285-295

2014

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T-type calcium channels in burst-firing, network synchrony, and epilepsy

Biochimica et biophysica acta, 2013-07, Vol.1828 (7), p.1572-1578

2013

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The type 1 cannabinoid receptor positive allosteric modulators GAT591 and GAT593 reduce spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg

IBRO neuroscience reports, 2022-06, Vol.12, p.121-130

2022

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Dissociable changes in spike and wave discharges following exposure to injected cannabinoids and smoked cannabis in Genetic Absence Epilepsy Rats from Strasbourg

The European journal of neuroscience, 2022-02, Vol.55 (4), p.1063-1078

2022

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Brainstem spreading depolarization and cortical dynamics during fatal seizures in Cacna1a S218L mice

Brain (London, England : 1878), 2019-02, Vol.142 (2), p.412-425

2019

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Low threshold T‐type calcium channels as targets for novel epilepsy treatments

British journal of clinical pharmacology, 2014-05, Vol.77 (5), p.729-739

2014

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Splice-variant changes of the CaV3.2 T-type calcium channel mediate voltage-dependent facilitation and associate with cardiac hypertrophy and development

Channels (Austin, Tex.), 2010-09, Vol.4 (5), p.375-389

2010

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Sociability impairments in Genetic Absence Epilepsy Rats from Strasbourg: Reversal by the T-type calcium channel antagonist Z944

Experimental neurology, 2017-10, Vol.296, p.16-22

2017

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The T‐type calcium channel blocker Z944 reduces conditioned fear in Genetic Absence Epilepsy Rats from Strasbourg and the non‐epileptic control strain

The European journal of neuroscience, 2019-09, Vol.50 (6), p.3046-3059

2019

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In vivo imaging reveals that pregabalin inhibits cortical spreading depression and propagation to subcortical brain structures

Proceedings of the National Academy of Sciences - PNAS, 2017-02, Vol.114 (9), p.2401-2406

2017

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The Cacna1h mutation in the GAERS model of absence epilepsy enhances T-type Ca2+ currents by altering calnexin-dependent trafficking of Cav3.2 channels

Scientific reports, 2017-09, Vol.7 (1), p.1-13, Article 11513

2017

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The T-type calcium channel antagonist, Z944, alters social behavior in Genetic Absence Epilepsy Rats from Strasbourg

Behavioural brain research, 2019-04, Vol.361, p.54-64

2019

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T-type calcium channel blockers that attenuate thalamic burst firing and suppress absence seizures

Science translational medicine, 2012-02, Vol.4 (121), p.121ra19

2012

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Thalamocortical neurons display suppressed burst-firing due to an enhanced Ih current in a genetic model of absence epilepsy

Pflügers Archiv, 2015-06, Vol.467 (6), p.1367-1382

2015

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The Genetic Absence Epilepsy Rats from Strasbourg model of absence epilepsy exhibits alterations in fear conditioning and latent inhibition consistent with psychiatric comorbidities in humans

The European journal of neuroscience, 2016-01, Vol.43 (1), p.25-40

2016

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Voltage-gated calcium channels and disease

BioFactors (Oxford), 2011-05, Vol.37 (3), p.197-205

2011

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The T-type calcium channel antagonist Z944 disrupts prepulse inhibition in both epileptic and non-epileptic rats

Neuroscience, 2016-09, Vol.332, p.121-129

2016

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The cellular mechanisms of neuronal swelling underlying cytotoxic edema

Pregabalin as a Pain Therapeutic: Beyond Calcium Channels

Contributions of T-type calcium channel isoforms to neuronal firing

Positive allosteric modulation of type 1 cannabinoid receptors reduces spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg

The T-type calcium channel antagonist Z944 rescues impairments in crossmodal and visual recognition memory in Genetic Absence Epilepsy Rats from Strasbourg

The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation

Image-Based Dosimetry in Dogs and Cross-Reactivity with Human Tissues of IGF2R-Targeting Human Antibody

Chronic pregabalin treatment protects against spreading depolarization and alters hippocampal synaptic characteristics in a model of familial hemiplegic migraine-type 1

Epigallocatechin-3-gallate elicits Ca2+ spike in MCF-7 breast cancer cells: Essential role of Cav3.2 channels

T-type calcium channels in burst-firing, network synchrony, and epilepsy

The type 1 cannabinoid receptor positive allosteric modulators GAT591 and GAT593 reduce spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg

Dissociable changes in spike and wave discharges following exposure to injected cannabinoids and smoked cannabis in Genetic Absence Epilepsy Rats from Strasbourg

Brainstem spreading depolarization and cortical dynamics during fatal seizures in Cacna1a S218L mice

Low threshold T‐type calcium channels as targets for novel epilepsy treatments

Splice-variant changes of the CaV3.2 T-type calcium channel mediate voltage-dependent facilitation and associate with cardiac hypertrophy and development

Sociability impairments in Genetic Absence Epilepsy Rats from Strasbourg: Reversal by the T-type calcium channel antagonist Z944

The T‐type calcium channel blocker Z944 reduces conditioned fear in Genetic Absence Epilepsy Rats from Strasbourg and the non‐epileptic control strain

In vivo imaging reveals that pregabalin inhibits cortical spreading depression and propagation to subcortical brain structures

The Cacna1h mutation in the GAERS model of absence epilepsy enhances T-type Ca2+ currents by altering calnexin-dependent trafficking of Cav3.2 channels

The T-type calcium channel antagonist, Z944, alters social behavior in Genetic Absence Epilepsy Rats from Strasbourg

T-type calcium channel blockers that attenuate thalamic burst firing and suppress absence seizures

Thalamocortical neurons display suppressed burst-firing due to an enhanced Ih current in a genetic model of absence epilepsy

The Genetic Absence Epilepsy Rats from Strasbourg model of absence epilepsy exhibits alterations in fear conditioning and latent inhibition consistent with psychiatric comorbidities in humans

Voltage-gated calcium channels and disease

The T-type calcium channel antagonist Z944 disrupts prepulse inhibition in both epileptic and non-epileptic rats

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