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Homeostatic Control of Innate Lung Inflammation by Vici Syndrome Gene Epg5 and Additional Autophagy Genes Promotes Influenza Pathogenesis

Cell host & microbe, 2016-01, Vol.19 (1), p.102-113

2016

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Lung Remodeling Regions in Long-Term Coronavirus Disease 2019 Feature Basal Epithelial Cell Reprogramming

The American journal of pathology, 2023-06, Vol.193 (6), p.680-689

2023

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Lung environment determines unique phenotype of alveolar macrophages

American journal of physiology. Lung cellular and molecular physiology, 2009-06, Vol.296 (6), p.L936-L946

2009

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Basal epithelial stem cells cross an alarmin checkpoint for postviral lung disease

The Journal of clinical investigation, 2021-10, Vol.131 (19)

2021

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Viral Evasion of Innate Immune Defense: The Case of Resistance of Pandemic H1N1 Influenza A Virus to Human Mannose-Binding Proteins

Frontiers in microbiology, 2021-12, Vol.12, p.774711-774711

2021

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Antiviral Activity of the Long Chain Pentraxin PTX3 against Influenza Viruses

The Journal of immunology (1950), 2008-03, Vol.180 (5), p.3391-3398

2008

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Pandemic H1N1 influenza A viruses are resistant to the antiviral activities of innate immune proteins of the collectin and pentraxin superfamilies

The Journal of immunology (1950), 2010-10, Vol.185 (7), p.4284-4291

2010

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Surfactant proteins a and d and pulmonary host defense

Annual review of physiology, 2001-01, Vol.63 (1), p.521-554

2001

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Role of viral hemagglutinin glycosylation in anti-influenza activities of recombinant surfactant protein D

Respiratory research, 2008-09, Vol.9 (1), p.65-65, Article 65

2008

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Interactions of alpha-, beta-, and theta-defensins with influenza A virus and surfactant protein D

The Journal of immunology (1950), 2009-06, Vol.182 (12), p.7878-7887

2009

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Surfactant protein-D and pulmonary host defense

Respiratory research, 2000, Vol.1 (2), p.93-108, Article 6

Crouch, E C

2000

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Structure binding relationship of human surfactant protein D and various lipopolysaccharide inner core structures

Journal of structural biology, 2016-09, Vol.195 (3), p.387-395

2016

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Computational Analysis Reveals a Key Regulator of Cryptococcal Virulence and Determinant of Host Response

mBio, 2016-04, Vol.7 (2), p.e00313-e00316

2016

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Collectins, H-ficolin and LL-37 reduce influence viral replication in human monocytes and modulate virus-induced cytokine production

Innate immunity (London, England), 2017-01, Vol.23 (1), p.77-88

2017

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Modification of surfactant protein D by reactive oxygen-nitrogen intermediates is accompanied by loss of aggregating activity, in vitro and in vivo

The FASEB journal, 2009-05, Vol.23 (5), p.1415-1430

2009

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Molecular Mechanisms of Inhibition of Influenza by Surfactant Protein D Revealed by Large-Scale Molecular Dynamics Simulation

Biochemistry (Easton), 2013-11, Vol.52 (47), p.8527-8538

2013

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Collectins and Pulmonary Host Defense

American journal of respiratory cell and molecular biology, 1998-08, Vol.19 (2), p.177-201

Crouch, Erika C

1998

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Innate Defense against Influenza A Virus: Activity of Human Neutrophil Defensins and Interactions of Defensins with Surfactant Protein D

Journal of Immunology, 2006-06, Vol.176 (11), p.6962-6972

2006

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Human Neutrophil Defensins Increase Neutrophil Uptake of Influenza A Virus and Bacteria and Modify Virus-Induced Respiratory Burst Responses

Journal of Immunology, 2007-06, Vol.178 (12), p.8046-8052

2007

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Human Pulmonary Surfactant Protein D Binds the Extracellular Domains of Toll-like Receptors 2 and 4 through the Carbohydrate Recognition Domain by a Mechanism Different from Its Binding to Phosphatidylinositol and Lipopolysaccharide

Biochemistry (Easton), 2006-07, Vol.45 (28), p.8657-8664

2006

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Surfactant Protein D Enhances Clearance of Influenza A Virus from the Lung In Vivo

The Journal of immunology (1950), 2001-11, Vol.167 (10), p.5868-5873

2001

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S-nitrosylation of surfactant protein-D controls inflammatory function

PLoS biology, 2008-11, Vol.6 (11), p.e266-e266

2008

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The ability of pandemic influenza virus hemagglutinins to induce lower respiratory pathology is associated with decreased surfactant protein D binding

Virology (New York, N.Y.), 2011-04, Vol.412 (2), p.426-434

2011

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A correctable immune niche for epithelial stem-cell reprogramming and post-viral lung diseases

The Journal of clinical investigation, 2024-07

2024

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Homeostatic Control of Innate Lung Inflammation by Vici Syndrome Gene Epg5 and Additional Autophagy Genes Promotes Influenza Pathogenesis

Ki67 Proliferation Index as a Tool for Chemotherapy Decisions During and After Neoadjuvant Aromatase Inhibitor Treatment of Breast Cancer: Results From the American College of Surgeons Oncology Group Z1031 Trial (Alliance)

Lung Remodeling Regions in Long-Term Coronavirus Disease 2019 Feature Basal Epithelial Cell Reprogramming

Lung environment determines unique phenotype of alveolar macrophages

Basal epithelial stem cells cross an alarmin checkpoint for postviral lung disease

Viral Evasion of Innate Immune Defense: The Case of Resistance of Pandemic H1N1 Influenza A Virus to Human Mannose-Binding Proteins

Antiviral Activity of the Long Chain Pentraxin PTX3 against Influenza Viruses

Pandemic H1N1 influenza A viruses are resistant to the antiviral activities of innate immune proteins of the collectin and pentraxin superfamilies

Surfactant proteins a and d and pulmonary host defense

Role of viral hemagglutinin glycosylation in anti-influenza activities of recombinant surfactant protein D

Interactions of alpha-, beta-, and theta-defensins with influenza A virus and surfactant protein D

Surfactant protein-D and pulmonary host defense

Structure binding relationship of human surfactant protein D and various lipopolysaccharide inner core structures

Computational Analysis Reveals a Key Regulator of Cryptococcal Virulence and Determinant of Host Response

Collectins, H-ficolin and LL-37 reduce influence viral replication in human monocytes and modulate virus-induced cytokine production

Modification of surfactant protein D by reactive oxygen-nitrogen intermediates is accompanied by loss of aggregating activity, in vitro and in vivo

Molecular Mechanisms of Inhibition of Influenza by Surfactant Protein D Revealed by Large-Scale Molecular Dynamics Simulation

Collectins and Pulmonary Host Defense

Innate Defense against Influenza A Virus: Activity of Human Neutrophil Defensins and Interactions of Defensins with Surfactant Protein D

Human Neutrophil Defensins Increase Neutrophil Uptake of Influenza A Virus and Bacteria and Modify Virus-Induced Respiratory Burst Responses

Human Pulmonary Surfactant Protein D Binds the Extracellular Domains of Toll-like Receptors 2 and 4 through the Carbohydrate Recognition Domain by a Mechanism Different from Its Binding to Phosphatidylinositol and Lipopolysaccharide

Surfactant Protein D Enhances Clearance of Influenza A Virus from the Lung In Vivo

S-nitrosylation of surfactant protein-D controls inflammatory function

The ability of pandemic influenza virus hemagglutinins to induce lower respiratory pathology is associated with decreased surfactant protein D binding

A correctable immune niche for epithelial stem-cell reprogramming and post-viral lung diseases

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