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Alveolar Macrophage Apoptosis-associated Bacterial Killing Helps Prevent Murine Pneumonia

American journal of respiratory and critical care medicine, 2019-07, Vol.200 (1), p.84-97

2019

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Thr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancer

PloS one, 2012-10, Vol.7 (10), p.e47060

2012

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Serine 64 Phosphorylation Enhances the Antiapoptotic Function of Mcl-1

The Journal of biological chemistry, 2007-06, Vol.282 (25), p.18407-18417

2007

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Inhibition of Protein Phosphatase 2A (PP2A) Prevents Mcl-1 Protein Dephosphorylation at the Thr-163/Ser-159 Phosphodegron, Dramatically Reducing Expression in Mcl-1-amplified Lymphoma Cells

The Journal of biological chemistry, 2014-08, Vol.289 (32), p.21950-21959

2014

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MCL1 increases primitive thymocyte viability in female mice and promotes thymic expansion into adulthood

International immunology, 2011-10, Vol.23 (10), p.647-659

2011

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MCL1 transgenic mice exhibit a high incidence of B-cell lymphoma manifested as a spectrum of histologic subtypes

Blood, 2001-06, Vol.97 (12), p.3902-3909

2001

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Thymus Size and Age-related Thymic Involution: Early Programming, Sexual Dimorphism, Progenitors and Stroma

Aging and disease, 2012-06, Vol.3 (3), p.280-290

2012

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Impaired Mitochondrial Microbicidal Responses in Chronic Obstructive Pulmonary Disease Macrophages

American journal of respiratory and critical care medicine, 2017-10, Vol.196 (7), p.845-855

2017

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Constitutive androstane receptor (CAR) ligand, TCPOBOP, attenuates Fas‐induced murine liver injury by altering Bcl‐2 proteins

Hepatology (Baltimore, Md.), 2006-07, Vol.44 (1), p.252-262

2006

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N-terminal Truncation of Antiapoptotic MCL1, but Not G2/M-induced Phosphorylation, Is Associated with Stabilization and Abundant Expression in Tumor Cells

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.23919-23936

2007

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Mcl-1 protects eosinophils from apoptosis and exacerbates allergic airway inflammation

Thorax, 2020-07, Vol.75 (7), p.600-605

2020

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MCL1 is phosphorylated in the PEST region and stabilized upon ERK activation in viable cells, and at additional sites with cytotoxic okadaic acid or taxol

Oncogene, 2004-07, Vol.23 (31), p.5301-5315

2004

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Mcl-1 regulates effector and memory CD8 T-cell differentiation during acute viral infection

Virology (New York, N.Y.), 2016-03, Vol.490, p.75-82

2016

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Expression of the antiapoptotic MCL1 gene product is regulated by a mitogen activated protein kinase-mediated pathway triggered through microtubule disruption and protein kinase C

Oncogene, 1998-09, Vol.17 (10), p.1223-1234

1998

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MCL1 enhances the survival of CD8+ memory T Cells after viral infection

Journal of virology, 2015-02, Vol.89 (4), p.2405-2414

2015

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Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavage

Oncogene, 2004-06, Vol.23 (28), p.4818-4827

2004

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Dynamic changes in Mcl-1 expression regulate macrophage viability or commitment to apoptosis during bacterial clearance

The Journal of clinical investigation, 2005-02, Vol.115 (2), p.359-368

2005

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Exon Skipping in Mcl-1 Results in a Bcl-2 Homology Domain 3 Only Gene Product That Promotes Cell Death

The Journal of biological chemistry, 2000-07, Vol.275 (29), p.22136-22146

2000

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Mcl-1, a Bcl-2 family member, delays the death of hematopoietic cells under a variety of apoptosis-inducing conditions

Blood, 1997-01, Vol.89 (2), p.630-643

1997

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MCL1, a Gene Expressed in Programmed Myeloid Cell Differentiation, has Sequence Similarity to BCL2

Proceedings of the National Academy of Sciences - PNAS, 1993-04, Vol.90 (8), p.3516-3520

1993

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The Intracellular Distribution and Pattern of Expression of Mcl-1 Overlap with, but Are Not Identical to, Those of Bcl-2

The Journal of cell biology, 1995-03, Vol.128 (6), p.1173-1184

1995

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Mcl-1 in Transgenic Mice Promotes Survival in a Spectrum of Hematopoietic Cell Types and Immortalization in the Myeloid Lineage

Blood, 1998-11, Vol.92 (9), p.3226-3239

1998

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Myeloid Cell Leukemia 1 Is Phosphorylated through Two Distinct Pathways, One Associated with Extracellular Signal-regulated Kinase Activation and the Other with G2/M Accumulation or Protein Phosphatase 1/2A Inhibition

The Journal of biological chemistry, 2000-07, Vol.275 (28), p.21688-21694

2000

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Mcl-1 in Transgenic Mice Promotes Survival in a Spectrum of Hematopoietic Cell Types and Immortalization in the Myeloid Lineage

Blood, 1998-11, Vol.92 (9), p.3226-3239

1998

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Overexpression of Mcl-1 Attenuates Liver Injury and Fibrosis in the Bile Duct-Ligated Mouse

Digestive diseases and sciences, 2009-09, Vol.54 (9), p.1908-1917

2009

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Alveolar Macrophage Apoptosis-associated Bacterial Killing Helps Prevent Murine Pneumonia

Thr 163 phosphorylation causes Mcl-1 stabilization when degradation is independent of the adjacent GSK3-targeted phosphodegron, promoting drug resistance in cancer

Serine 64 Phosphorylation Enhances the Antiapoptotic Function of Mcl-1

Inhibition of Protein Phosphatase 2A (PP2A) Prevents Mcl-1 Protein Dephosphorylation at the Thr-163/Ser-159 Phosphodegron, Dramatically Reducing Expression in Mcl-1-amplified Lymphoma Cells

MCL1 increases primitive thymocyte viability in female mice and promotes thymic expansion into adulthood

MCL1 transgenic mice exhibit a high incidence of B-cell lymphoma manifested as a spectrum of histologic subtypes

Thymus Size and Age-related Thymic Involution: Early Programming, Sexual Dimorphism, Progenitors and Stroma

Impaired Mitochondrial Microbicidal Responses in Chronic Obstructive Pulmonary Disease Macrophages

Constitutive androstane receptor (CAR) ligand, TCPOBOP, attenuates Fas‐induced murine liver injury by altering Bcl‐2 proteins

N-terminal Truncation of Antiapoptotic MCL1, but Not G2/M-induced Phosphorylation, Is Associated with Stabilization and Abundant Expression in Tumor Cells

Mcl-1 protects eosinophils from apoptosis and exacerbates allergic airway inflammation

MCL1 is phosphorylated in the PEST region and stabilized upon ERK activation in viable cells, and at additional sites with cytotoxic okadaic acid or taxol

Mcl-1 regulates effector and memory CD8 T-cell differentiation during acute viral infection

Expression of the antiapoptotic MCL1 gene product is regulated by a mitogen activated protein kinase-mediated pathway triggered through microtubule disruption and protein kinase C

MCL1 enhances the survival of CD8+ memory T Cells after viral infection

Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavage

Dynamic changes in Mcl-1 expression regulate macrophage viability or commitment to apoptosis during bacterial clearance

Exon Skipping in Mcl-1 Results in a Bcl-2 Homology Domain 3 Only Gene Product That Promotes Cell Death

Mcl-1, a Bcl-2 family member, delays the death of hematopoietic cells under a variety of apoptosis-inducing conditions

MCL1, a Gene Expressed in Programmed Myeloid Cell Differentiation, has Sequence Similarity to BCL2

The Intracellular Distribution and Pattern of Expression of Mcl-1 Overlap with, but Are Not Identical to, Those of Bcl-2

Mcl-1 in Transgenic Mice Promotes Survival in a Spectrum of Hematopoietic Cell Types and Immortalization in the Myeloid Lineage

Myeloid Cell Leukemia 1 Is Phosphorylated through Two Distinct Pathways, One Associated with Extracellular Signal-regulated Kinase Activation and the Other with G2/M Accumulation or Protein Phosphatase 1/2A Inhibition

Mcl-1 in Transgenic Mice Promotes Survival in a Spectrum of Hematopoietic Cell Types and Immortalization in the Myeloid Lineage

Overexpression of Mcl-1 Attenuates Liver Injury and Fibrosis in the Bile Duct-Ligated Mouse

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