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Signaling pathways in the specification of arteries and veins

Arteriosclerosis, thrombosis, and vascular biology, 2014-11, Vol.34 (11), p.2372

2014

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Endothelial cells are progenitors of cardiac pericytes and vascular smooth muscle cells

Nature communications, 2016-08, Vol.7 (1), p.12422-12422, Article 12422

2016

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EndMT contributes to the onset and progression of cerebral cavernous malformations

Nature (London), 2013-06, Vol.498 (7455), p.492-496

2013

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Endothelial cell clonal expansion in the development of cerebral cavernous malformations

Nature communications, 2019-06, Vol.10 (1), p.2761-16, Article 2761

2019

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Phosphorylation of VE-cadherin is modulated by haemodynamic forces and contributes to the regulation of vascular permeability in vivo

Nature communications, 2012-11, Vol.3 (1), p.1208, Article 1208

2012

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VE-Cadherin–Mediated Epigenetic Regulation of Endothelial Gene Expression

Circulation research, 2018-01, Vol.122 (2), p.231-245

2018

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SoxF factors induce Notch1 expression via direct transcriptional regulation during early arterial development

Development (Cambridge), 2017-07, Vol.144 (14), p.2629-2639

2017

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Sulindac metabolites decrease cerebrovascular malformations in CCM3-knockout mice

Proceedings of the National Academy of Sciences - PNAS, 2015-07, Vol.112 (27), p.8421-8426

2015

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KLF4 is a key determinant in the development and progression of cerebral cavernous malformations

EMBO molecular medicine, 2016-01, Vol.8 (1), p.6-24

2016

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Fine-Tuning of Sox17 and Canonical Wnt Coordinates the Permeability Properties of the Blood-Brain Barrier

Circulation research, 2019-02, Vol.124 (4), p.511-525

2019

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Epac1 regulates integrity of endothelial cell junctions through VE-cadherin

FEBS letters, 2005-09, Vol.579 (22), p.4966-4972

2005

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Mapping endothelial-cell diversity in cerebral cavernous malformations at single-cell resolution

eLife, 2020-11, Vol.9

2020

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Wnt activation of immortalized brain endothelial cells as a tool for generating a standardized model of the blood brain barrier in vitro

PloS one, 2013-08, Vol.8 (8), p.e70233-e70233

2013

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Peg3/PW1 Is a Marker of a Subset of Vessel Associated Endothelial Progenitors

Stem cells (Dayton, Ohio), 2017-05, Vol.35 (5), p.1328-1340

2017

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Overlapping and divergent signaling pathways of N-cadherin and VE-cadherin in endothelial cells

Blood, 2012-03, Vol.119 (9), p.2159-2170

2012

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Endothelial-to-Mesenchymal Transition in Bone Marrow and Spleen of Primary Myelofibrosis

The American journal of pathology, 2017-08, Vol.187 (8), p.1879-1892

2017

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Abrogation of junctional adhesion molecule-A expression induces cell apoptosis and reduces breast cancer progression

PloS one, 2011-06, Vol.6 (6), p.e21242

2011

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Vascular endothelial growth factor induces VE-cadherin tyrosine phosphorylation in endothelial cells

Journal of cell science, 1998-07, Vol.111 ( Pt 13) (13), p.1853-1865

1998

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JAM-A promotes neutrophil chemotaxis by controlling integrin internalization and recycling

Journal of cell science, 2009-01, Vol.122 (2), p.268-277

2009

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VE-cadherin regulates endothelial actin activating Rac and increasing membrane association of Tiam

Molecular biology of the cell, 2002-04, Vol.13 (4), p.1175-1189

2002

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Increased DC trafficking to lymph nodes and contact hypersensitivity in junctional adhesion molecule-A-deficient mice

The Journal of clinical investigation, 2004-09, Vol.114 (5), p.729-738

2004

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Junctional adhesion molecule-A deficiency increases hepatic ischemia-reperfusion injury despite reduction of neutrophil transendothelial migration

Blood, 2005-07, Vol.106 (2), p.725-733

2005

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Junctional Adhesion Molecule-A-Deficient Polymorphonuclear Cells Show Reduced Diapedesis in Peritonitis and Heart Ischemia-Reperfusion Injury

Proceedings of the National Academy of Sciences - PNAS, 2005-07, Vol.102 (30), p.10634-10639

2005

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A monoclonal antibody to vascular endothelial–cadherin inhibits tumor angiogenesis without side effects on endothelial permeability

Blood, 2002-08, Vol.100 (3), p.905-911

2002

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Signaling pathways in the specification of arteries and veins

Endothelial cells are progenitors of cardiac pericytes and vascular smooth muscle cells

EndMT contributes to the onset and progression of cerebral cavernous malformations

Endothelial cell clonal expansion in the development of cerebral cavernous malformations

Phosphorylation of VE-cadherin is modulated by haemodynamic forces and contributes to the regulation of vascular permeability in vivo

VE-Cadherin–Mediated Epigenetic Regulation of Endothelial Gene Expression

SoxF factors induce Notch1 expression via direct transcriptional regulation during early arterial development

Sulindac metabolites decrease cerebrovascular malformations in CCM3-knockout mice

KLF4 is a key determinant in the development and progression of cerebral cavernous malformations

Fine-Tuning of Sox17 and Canonical Wnt Coordinates the Permeability Properties of the Blood-Brain Barrier

Epac1 regulates integrity of endothelial cell junctions through VE-cadherin

Mapping endothelial-cell diversity in cerebral cavernous malformations at single-cell resolution

Wnt activation of immortalized brain endothelial cells as a tool for generating a standardized model of the blood brain barrier in vitro

Peg3/PW1 Is a Marker of a Subset of Vessel Associated Endothelial Progenitors

Overlapping and divergent signaling pathways of N-cadherin and VE-cadherin in endothelial cells

Endothelial-to-Mesenchymal Transition in Bone Marrow and Spleen of Primary Myelofibrosis

Correction: SoxF factors induce Notch1 expression via direct transcriptional regulation during early arterial development. Development doi: 10.1242/dev.146241

Abrogation of junctional adhesion molecule-A expression induces cell apoptosis and reduces breast cancer progression

Vascular endothelial growth factor induces VE-cadherin tyrosine phosphorylation in endothelial cells

JAM-A promotes neutrophil chemotaxis by controlling integrin internalization and recycling

VE-cadherin regulates endothelial actin activating Rac and increasing membrane association of Tiam

Increased DC trafficking to lymph nodes and contact hypersensitivity in junctional adhesion molecule-A-deficient mice

Junctional adhesion molecule-A deficiency increases hepatic ischemia-reperfusion injury despite reduction of neutrophil transendothelial migration

Junctional Adhesion Molecule-A-Deficient Polymorphonuclear Cells Show Reduced Diapedesis in Peritonitis and Heart Ischemia-Reperfusion Injury

A monoclonal antibody to vascular endothelial–cadherin inhibits tumor angiogenesis without side effects on endothelial permeability

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