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EULAR/ERA-EDTA recommendations for the management of ANCA-associated vasculitis

Annals of the rheumatic diseases, 2016-09, Vol.75 (9), p.1583-1594

2016

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EULAR recommendations for the management of primary small and medium vessel vasculitis

Annals of the rheumatic diseases, 2009-03, Vol.68 (3), p.310-317

2009

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EULAR recommendations for the management of large vessel vasculitis

Annals of the rheumatic diseases, 2009-03, Vol.68 (3), p.318-323

2009

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Tissue production of pro-inflammatory cytokines (IL-1β, TNFα and IL-6) correlates with the intensity of the systemic inflammatory response and with corticosteroid requirements in giant-cell arteritis

Rheumatology (Oxford, England), 2004-03, Vol.43 (3), p.294-301

2004

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Trial of Tocilizumab in Giant-Cell Arteritis

The New England journal of medicine, 2017-07, Vol.377 (4), p.317-328

2017

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Imatinib mesylate inhibits in vitro and ex vivo biological responses related to vascular occlusion in giant cell arteritis

Annals of the rheumatic diseases, 2008-11, Vol.67 (11), p.1581-1588

2008

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2012 Revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides

Arthritis and rheumatism, 2013-01, Vol.65 (1), p.1-11

2013

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Increased expression of the endothelin system in arterial lesions from patients with giant-cell arteritis: association between elevated plasma endothelin levels and the development of ischaemic events

Annals of the rheumatic diseases, 2010-02, Vol.69 (2), p.434-442

2010

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Association between increased CCL2 (MCP-1) expression in lesions and persistence of disease activity in giant-cell arteritis

Rheumatology (Oxford, England), 2006-11, Vol.45 (11), p.1356-1363

2006

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Elevated production of interleukin-6 is associated with a lower incidence of disease-related ischemic events in patients with giant-cell arteritis: Angiogenic activity of interleukin-6 as a potential protective mechanism

Circulation (New York, N.Y.), 2003-05, Vol.107 (19), p.2428-2434

2003

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Fibronectin upregulates gelatinase B (MMP-9) and induces coordinated expression of gelatinase A (MMP-2) and its activator MT1-MMP (MMP-14) by human T lymphocyte cell lines. A process repressed through RAS/MAP kinase signaling pathways

Blood, 1999-10, Vol.94 (8), p.2754-2766

1999

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Influence of the IL17A locus in giant cell arteritis susceptibility

Annals of the rheumatic diseases, 2014-09, Vol.73 (9), p.1742-1745

2014

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Estrogen promotes angiogenic activity in human umbilical vein endothelial cells in vitro and in a murine model

Circulation (New York, N.Y.), 1995-02, Vol.91 (3), p.755-763

1995

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Identification of the PTPN22 functional variant R620W as susceptibility genetic factor for giant cell arteritis

Annals of the rheumatic diseases, 2013-11, Vol.72 (11), p.1882-1886

2013

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Tissue and serum markers of inflammation during the follow-up of patients with giant-cell arteritis-a prospective longitudinal study

Rheumatology (Oxford, England), 2011-11, Vol.50 (11), p.2061-2070

2011

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Expression of an estrogen receptor by human coronary artery and umbilical vein endothelial cells

Circulation (New York, N.Y.), 1996-09, Vol.94 (6), p.1402-1407

1996

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Estradiol enhances leukocyte binding to tumor necrosis factor (TNF)-stimulated endothelial cells via an increase in TNF-induced adhesion molecules E-selectin, intercellular adhesion molecule type 1, and vascular cell adhesion molecule type 1

The Journal of clinical investigation, 1994, Vol.93 (1), p.17-25

1994

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New therapeutic targets in giant-cell arteritis. Considerations based on the current pathogenic model and the availability of new therapeutic agents

Clinical and experimental rheumatology, 2008-05, Vol.26 (3 Suppl 49), p.S141

2008

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Gelatinase expression and proteolytic activity in giant-cell arteritis

Annals of the rheumatic diseases, 2007-11, Vol.66 (11), p.1429-1435

2007

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Mepolizumab or Placebo for Eosinophilic Granulomatosis with Polyangiitis

The New England journal of medicine, 2017-05, Vol.376 (20), p.1921-1932

2017

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Identification of haptoglobin as an angiogenic factor in sera from patients with systemic vasculitis

The Journal of clinical investigation, 1993-03, Vol.91 (3), p.977-985

1993

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Circulating soluble adhesion molecules in patients with giant cell arteritis. Correlation between soluble intercellular adhesion molecule-1 (sICAM-1) concentrations and disease activity

Annals of the rheumatic diseases, 1999-03, Vol.58 (3), p.189-192

1999

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Response to thalidomide in multiple myeloma: impact of angiogenic factors

Cytokine (Philadelphia, Pa.), 2004-05, Vol.26 (4), p.145-148

2004

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Expression of the adhesion molecule ICAM-1 in non-Hodgkin's lymphoma: relationship with tumor dissemination and prognostic importance

Journal of clinical oncology, 1998-01, Vol.16 (1), p.35-40

1998

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EULAR/ERA-EDTA recommendations for the management of ANCA-associated vasculitis

EULAR recommendations for the management of primary small and medium vessel vasculitis

EULAR recommendations for the management of large vessel vasculitis

Tissue production of pro-inflammatory cytokines (IL-1β, TNFα and IL-6) correlates with the intensity of the systemic inflammatory response and with corticosteroid requirements in giant-cell arteritis

Trial of Tocilizumab in Giant-Cell Arteritis

Imatinib mesylate inhibits in vitro and ex vivo biological responses related to vascular occlusion in giant cell arteritis

2012 Revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides

Increased expression of the endothelin system in arterial lesions from patients with giant-cell arteritis: association between elevated plasma endothelin levels and the development of ischaemic events

Association between increased CCL2 (MCP-1) expression in lesions and persistence of disease activity in giant-cell arteritis

Elevated production of interleukin-6 is associated with a lower incidence of disease-related ischemic events in patients with giant-cell arteritis: Angiogenic activity of interleukin-6 as a potential protective mechanism

EULAR points to consider in the development of classification and diagnostic criteria in systemic vasculitis

Fibronectin upregulates gelatinase B (MMP-9) and induces coordinated expression of gelatinase A (MMP-2) and its activator MT1-MMP (MMP-14) by human T lymphocyte cell lines. A process repressed through RAS/MAP kinase signaling pathways

Influence of the IL17A locus in giant cell arteritis susceptibility

Estrogen promotes angiogenic activity in human umbilical vein endothelial cells in vitro and in a murine model

Identification of the PTPN22 functional variant R620W as susceptibility genetic factor for giant cell arteritis

Tissue and serum markers of inflammation during the follow-up of patients with giant-cell arteritis-a prospective longitudinal study

Expression of an estrogen receptor by human coronary artery and umbilical vein endothelial cells

Estradiol enhances leukocyte binding to tumor necrosis factor (TNF)-stimulated endothelial cells via an increase in TNF-induced adhesion molecules E-selectin, intercellular adhesion molecule type 1, and vascular cell adhesion molecule type 1

New therapeutic targets in giant-cell arteritis. Considerations based on the current pathogenic model and the availability of new therapeutic agents

Gelatinase expression and proteolytic activity in giant-cell arteritis

Mepolizumab or Placebo for Eosinophilic Granulomatosis with Polyangiitis

Identification of haptoglobin as an angiogenic factor in sera from patients with systemic vasculitis

Circulating soluble adhesion molecules in patients with giant cell arteritis. Correlation between soluble intercellular adhesion molecule-1 (sICAM-1) concentrations and disease activity

Response to thalidomide in multiple myeloma: impact of angiogenic factors

Expression of the adhesion molecule ICAM-1 in non-Hodgkin's lymphoma: relationship with tumor dissemination and prognostic importance

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