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Tonicity-responsive microRNAs contribute to the maximal induction of osmoregulatory transcription factor OREBP in response to high-NaCl hypertonicity

Nucleic acids research, 2011-01, Vol.39 (2), p.475

2011

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Inducible nucleosome depletion at OREBP-binding-sites by hypertonic stress

PloS one, 2009-12, Vol.4 (12), p.e8435-e8435

2009

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Targeted over-expression of endothelin-1 in astrocytes leads to more severe brain damage and vasospasm after subarachnoid hemorrhage

BMC neuroscience, 2013-10, Vol.14 (1), p.131-131, Article 131

2013

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Deletion of aldose reductase leads to protection against cerebral ischemic injury

Journal of cerebral blood flow and metabolism, 2007-08, Vol.27 (8), p.1496-1509

2007

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Diabetic cataracts: mechanisms and management

Diabetes/metabolism research and reviews, 2010-03, Vol.26 (3), p.172-180

2010

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Selective over-expression of endothelin-1 in endothelial cells exacerbates inner retinal edema and neuronal death in ischemic retina

PloS one, 2011-10, Vol.6 (10), p.e26184

2011

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Sodium/myo-inositol cotransporter-1 is essential for the development and function of the peripheral nerves

The FASEB journal, 2005-11, Vol.19 (13), p.1887-1889

2005

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Endothelin-1 overexpression leads to further water accumulation and brain edema after middle cerebral artery occlusion via aquaporin 4 expression in astrocytic end-feet

Journal of Cerebral Blood Flow & Metabolism, 2005-08, Vol.25 (8), p.998-1011

2005

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Aldose reductase deficiency leads to oxidative stress-induced dopaminergic neuronal loss and autophagic abnormality in an animal model of Parkinson’s disease

Neurobiology of aging, 2017-02, Vol.50, p.119-133

2017

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Regulation of Nucleocytoplasmic Trafficking of Transcription Factor OREBP/TonEBP/NFAT5

The Journal of biological chemistry, 2006-08, Vol.281 (33), p.23870-23879

2006

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Endothelial endothelin-1 over-expression using receptor tyrosine kinase tie-1 promoter leads to more severe vascular permeability and blood brain barrier breakdown after transient middle cerebral artery occlusion

Brain research, 2009-04, Vol.1266, p.121-129

2009

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Phosphorylation by Casein Kinase 1 Regulates Tonicity-induced Osmotic Response Element-binding Protein/Tonicity Enhancer-binding Protein Nucleocytoplasmic Trafficking

The Journal of biological chemistry, 2008-06, Vol.283 (25), p.17624-17634

2008

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Aldose Reductase Deficiency Prevents Diabetes-Induced Blood-Retinal Barrier Breakdown, Apoptosis, and Glial Reactivation in the Retina of db/db Mice

Diabetes (New York, N.Y.), 2005-11, Vol.54 (11), p.3119-3125

2005

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Genetic restoration of aldose reductase to the collecting tubules restores maturation of the urine concentrating mechanism

American journal of physiology. Renal physiology, 2006-07, Vol.291 (1), p.F186-F195

2006

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Pseudolaric Acid B, a Novel Microtubule-Destabilizing Agent That Circumvents Multidrug Resistance Phenotype and Exhibits Antitumor Activity In vivo

Clinical cancer research, 2005-08, Vol.11 (16), p.6002-6011

2005

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More severe type 2 diabetes-associated ischemic stroke injury is alleviated in aldose reductase-deficient mice

Journal of neuroscience research, 2010-07, Vol.88 (9), p.2026-2034

2010

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Ion Channels in Mesenchymal Stem Cells from Rat Bone Marrow

Stem cells (Dayton, Ohio), 2006-06, Vol.24 (6), p.1519-1528

2006

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Osmotic Response Element-binding Protein (OREBP) Is an Essential Regulator of the Urine Concentrating Mechanism

The Journal of biological chemistry, 2004-11, Vol.279 (46), p.48048-48054

2004

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Aldose Reductase Regulates Hepatic Peroxisome Proliferator-activated Receptor α Phosphorylation and Activity to Impact Lipid Homeostasis

The Journal of biological chemistry, 2008-06, Vol.283 (25), p.17175-17183

2008

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Noninvasive Monitoring of Diabetes-Induced Cutaneous Nerve Fiber Loss and Hypoalgesia in thy1-YFP Transgenic Mice

Diabetes (New York, N.Y.), 2005-11, Vol.54 (11), p.3112-3118

2005

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Solution structures, dynamics, and lipid-binding of the sterile α-motif domain of the deleted in liver cancer 2

Proteins, structure, function, and bioinformatics, 2007-06, Vol.67 (4), p.1154-1166

2007

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Exchange protein activated by cAMP 1 (Epac1)‐deficient mice develop β‐cell dysfunction and metabolic syndrome

The FASEB journal, 2013-10, Vol.27 (10), p.4122-4135

2013

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Aldose Reductase–Deficient Mice Are Protected From Delayed Motor Nerve Conduction Velocity, Increased c-Jun NH2-Terminal Kinase Activation, Depletion of Reduced Glutathione, Increased Superoxide Accumulation, and DNA Damage

Diabetes (New York, N.Y.), 2006-07, Vol.55 (7), p.1946-1953

2006

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Transgenic Mice Expressing Dominant-negative Osmotic-response Element-binding Protein (OREBP) in Lens Exhibit Fiber Cell Elongation Defect Associated with Increased DNA Breaks

The Journal of biological chemistry, 2005-05, Vol.280 (20), p.19986-19991

2005

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Endothelial Cell-specific Over-expression of Endothelin- 1 Leads to More Severe Cerebral Damage following Transient Middle Cerebral Artery Occlusion

Journal of cardiovascular pharmacology, 2004-11, Vol.44 Suppl 1 (Supplement 1), p.S293-S300

2004

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Tonicity-responsive microRNAs contribute to the maximal induction of osmoregulatory transcription factor OREBP in response to high-NaCl hypertonicity

Inducible nucleosome depletion at OREBP-binding-sites by hypertonic stress

Targeted over-expression of endothelin-1 in astrocytes leads to more severe brain damage and vasospasm after subarachnoid hemorrhage

Deletion of aldose reductase leads to protection against cerebral ischemic injury

Diabetic cataracts: mechanisms and management

Selective over-expression of endothelin-1 in endothelial cells exacerbates inner retinal edema and neuronal death in ischemic retina

Sodium/myo-inositol cotransporter-1 is essential for the development and function of the peripheral nerves

Endothelin-1 overexpression leads to further water accumulation and brain edema after middle cerebral artery occlusion via aquaporin 4 expression in astrocytic end-feet

Aldose reductase deficiency leads to oxidative stress-induced dopaminergic neuronal loss and autophagic abnormality in an animal model of Parkinson’s disease

Regulation of Nucleocytoplasmic Trafficking of Transcription Factor OREBP/TonEBP/NFAT5

Endothelial endothelin-1 over-expression using receptor tyrosine kinase tie-1 promoter leads to more severe vascular permeability and blood brain barrier breakdown after transient middle cerebral artery occlusion

Phosphorylation by Casein Kinase 1 Regulates Tonicity-induced Osmotic Response Element-binding Protein/Tonicity Enhancer-binding Protein Nucleocytoplasmic Trafficking

Aldose Reductase Deficiency Prevents Diabetes-Induced Blood-Retinal Barrier Breakdown, Apoptosis, and Glial Reactivation in the Retina of db/db Mice

Genetic restoration of aldose reductase to the collecting tubules restores maturation of the urine concentrating mechanism

Pseudolaric Acid B, a Novel Microtubule-Destabilizing Agent That Circumvents Multidrug Resistance Phenotype and Exhibits Antitumor Activity In vivo

More severe type 2 diabetes-associated ischemic stroke injury is alleviated in aldose reductase-deficient mice

Ion Channels in Mesenchymal Stem Cells from Rat Bone Marrow

Osmotic Response Element-binding Protein (OREBP) Is an Essential Regulator of the Urine Concentrating Mechanism

Aldose Reductase Regulates Hepatic Peroxisome Proliferator-activated Receptor α Phosphorylation and Activity to Impact Lipid Homeostasis

Noninvasive Monitoring of Diabetes-Induced Cutaneous Nerve Fiber Loss and Hypoalgesia in thy1-YFP Transgenic Mice

Solution structures, dynamics, and lipid-binding of the sterile α-motif domain of the deleted in liver cancer 2

Exchange protein activated by cAMP 1 (Epac1)‐deficient mice develop β‐cell dysfunction and metabolic syndrome

Aldose Reductase–Deficient Mice Are Protected From Delayed Motor Nerve Conduction Velocity, Increased c-Jun NH2-Terminal Kinase Activation, Depletion of Reduced Glutathione, Increased Superoxide Accumulation, and DNA Damage

Transgenic Mice Expressing Dominant-negative Osmotic-response Element-binding Protein (OREBP) in Lens Exhibit Fiber Cell Elongation Defect Associated with Increased DNA Breaks

Endothelial Cell-specific Over-expression of Endothelin- 1 Leads to More Severe Cerebral Damage following Transient Middle Cerebral Artery Occlusion

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