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Remote ischaemic conditioning: defining critical criteria for success—report from the 11th Hatter Cardiovascular Workshop

Basic research in cardiology, 2022-12, Vol.117 (1), p.39-39, Article 39

2022

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9th Hatter Biannual Meeting: position document on ischaemia/reperfusion injury, conditioning and the ten commandments of cardioprotection

Basic Research in Cardiology, 2016-07, Vol.111 (4), p.41-41, Article 41

2016

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Glucose-lowering effect of the DPP-4 inhibitor sitagliptin after glucose and non-glucose macronutrient ingestion in non-diabetic subjects

Diabetes, obesity & metabolism, 2013-06, Vol.15 (6), p.531-537

2013

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GLP-1 signals via ERK in peripheral nerve and prevents nerve dysfunction in diabetic mice

Diabetes, obesity & metabolism, 2011-11, Vol.13 (11), p.990-1000

2011

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Neutral endopeptidase 24.11 and dipeptidyl peptidase IV are both mediators of the degradation of glucagon-like peptide 1 in the anaesthetised pig

Diabetologia, 2005-09, Vol.48 (9), p.1882-1890

2005

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Inhibitors of dipeptidyl peptidase‐4 as therapeutic agents for individuals with type 2 diabetes: a 25‐year journey

Diabetic medicine, 2020-08, Vol.37 (8), p.1230-1233

2020

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Drug development from the bench to the pharmacy: with special reference to dipeptidyl peptidase-4 inhibitor development

Diabetic medicine, 2016-06, Vol.33 (6), p.718-722

Carr, R. D.

2016

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Stimulation of insulin release by repaglinide and glibenclamide involves both common and distinct processes

Diabetes (New York, N.Y.), 1998-03, Vol.47 (3), p.345

1998

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Dissociated incretin hormone response to protein versus fat ingestion in obese subjects

Diabetes, obesity & metabolism, 2011-09, Vol.13 (9), p.863-865

2011

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Discovery and Structure−Activity Relationship of the First Non-Peptide Competitive Human Glucagon Receptor Antagonists

Journal of medicinal chemistry, 1998-12, Vol.41 (26), p.5150-5157

1998

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Reduction of beta cell mass: partial insulin secretory compensation from the residual beta cell population in the nicotinamide-streptozotocin Göttingen minipig after oral glucose in vivo and in the perfused pancreas

Diabetologia, 2004-11, Vol.47 (11), p.1873-1878

2004

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Beta-cell function and islet morphology in normal, obese, and obese beta-cell mass-reduced Göttingen minipigs

American journal of physiology: endocrinology and metabolism, 2005-02, Vol.288 (2), p.E412-E421

2005

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Loss of beta-cell mass leads to a reduction of pulse mass with normal periodicity, regularity and entrainment of pulsatile insulin secretion in Göttingen minipigs

Diabetologia, 2003-02, Vol.46 (2), p.195-202

2003

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Sensory nerve inactivation by resiniferatoxin improves insulin sensitivity in male obese Zucker rats

American journal of physiology: endocrinology and metabolism, 2005-06, Vol.288 (6), p.E1137-E1145

2005

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What Lifecycle Management Lessons Can We Learn From Pd-1 Immuno-Oncology Therapies?

Value in health, 2015-11, Vol.18 (7), p.A488-A488

2015

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Price Fluctuations Across Lifecycle: Insights From The Anti-Vegf Market

Value in health, 2015-11, Vol.18 (7), p.A428-A428

2015

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The conscious Göttingen minipig as a model for studying rapid pulsatile insulin secretion in vivo

Diabetologia, 2002-10, Vol.45 (10), p.1389-1396

2002

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Reply to: Ahren B et al. Mechanisms of action of the dipeptidyl peptidase-4 inhibitor vildagliptin in humans. Diabetes Obes Metab 2011; 13(9): 775-783 and Ahren B et al. Clinical evidence and mechanistic basis for vildagliptin's action when added to metformin. Diabetes Obes Metab 2011; 13(3): 193-203

Diabetes, obesity & metabolism, 2012-04, Vol.14 (4), p.383-384

2012

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Angiotensin (1–7) is an antagonist at the type 1 angiotensin II receptor

Journal of hypertension, 1994-12, Vol.12 (12), p.1377-1382

1994

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Plasma calcitonin gene-related peptide is increased prior to obesity, and sensory nerve desensitization by capsaicin improves oral glucose tolerance in obese Zucker rats

European journal of endocrinology, 2005-12, Vol.153 (6), p.963

2005

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Parameters of Glucose and Lipid Metabolism in the Male Göttingen Minipig: Influence of Age, Body Weight, and Breeding Family

Comparative medicine, 2001-10, Vol.51 (5), p.436-442

2001

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Inhibition of neutral endopeptidase 24.11 does not potentiate the improvement in glycemic control obtained with dipeptidyl peptidase-4 inhibition in diabetic Goto-Kakizaki rats

Hormone and metabolic research, 2009-11, Vol.41 (11), p.851

2009

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The 10th Biennial Hatter Cardiovascular Institute workshop: cellular protectionevaluating new directions in the setting of myocardial infarction, ischaemic stroke, and cardio-oncology

Basic research in cardiology, 2018-11, Vol.113 (6)

2018

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Sensory nerve desensitization by resiniferatoxin improves glucose tolerance and increases insulin secretion in Zucker Diabetic Fatty rats and is associated with reduced plasma activity of dipeptidyl peptidase IV

European journal of pharmacology, 2005-02, Vol.509 (2), p.211-217

2005

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Evidence that renal prostaglandin and thromboxane production is stimulated in chronic cyclosporine nephrotoxicity

Transplantation, 1987-02, Vol.43 (2), p.282

1987

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Remote ischaemic conditioning: defining critical criteria for success—report from the 11th Hatter Cardiovascular Workshop

9th Hatter Biannual Meeting: position document on ischaemia/reperfusion injury, conditioning and the ten commandments of cardioprotection

Glucose-lowering effect of the DPP-4 inhibitor sitagliptin after glucose and non-glucose macronutrient ingestion in non-diabetic subjects

GLP-1 signals via ERK in peripheral nerve and prevents nerve dysfunction in diabetic mice

Neutral endopeptidase 24.11 and dipeptidyl peptidase IV are both mediators of the degradation of glucagon-like peptide 1 in the anaesthetised pig

Inhibitors of dipeptidyl peptidase‐4 as therapeutic agents for individuals with type 2 diabetes: a 25‐year journey

Drug development from the bench to the pharmacy: with special reference to dipeptidyl peptidase-4 inhibitor development

Stimulation of insulin release by repaglinide and glibenclamide involves both common and distinct processes

Dissociated incretin hormone response to protein versus fat ingestion in obese subjects

Discovery and Structure−Activity Relationship of the First Non-Peptide Competitive Human Glucagon Receptor Antagonists

Reduction of beta cell mass: partial insulin secretory compensation from the residual beta cell population in the nicotinamide-streptozotocin Göttingen minipig after oral glucose in vivo and in the perfused pancreas

Beta-cell function and islet morphology in normal, obese, and obese beta-cell mass-reduced Göttingen minipigs

Loss of beta-cell mass leads to a reduction of pulse mass with normal periodicity, regularity and entrainment of pulsatile insulin secretion in Göttingen minipigs

Sensory nerve inactivation by resiniferatoxin improves insulin sensitivity in male obese Zucker rats

What Lifecycle Management Lessons Can We Learn From Pd-1 Immuno-Oncology Therapies?

Price Fluctuations Across Lifecycle: Insights From The Anti-Vegf Market

The conscious Göttingen minipig as a model for studying rapid pulsatile insulin secretion in vivo

Angiotensin (1–7) is an antagonist at the type 1 angiotensin II receptor

Plasma calcitonin gene-related peptide is increased prior to obesity, and sensory nerve desensitization by capsaicin improves oral glucose tolerance in obese Zucker rats

Parameters of Glucose and Lipid Metabolism in the Male Göttingen Minipig: Influence of Age, Body Weight, and Breeding Family

Inhibition of neutral endopeptidase 24.11 does not potentiate the improvement in glycemic control obtained with dipeptidyl peptidase-4 inhibition in diabetic Goto-Kakizaki rats

The 10th Biennial Hatter Cardiovascular Institute workshop: cellular protectionevaluating new directions in the setting of myocardial infarction, ischaemic stroke, and cardio-oncology

Sensory nerve desensitization by resiniferatoxin improves glucose tolerance and increases insulin secretion in Zucker Diabetic Fatty rats and is associated with reduced plasma activity of dipeptidyl peptidase IV

Evidence that renal prostaglandin and thromboxane production is stimulated in chronic cyclosporine nephrotoxicity

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