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Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells

Glia, 2014-09, Vol.62 (9), p.1418-1434

2014

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Type I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental Malaria

PLoS pathogens, 2016-10, Vol.12 (10), p.e1005945-e1005945

2016

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Herpes simplex virus-1 infects the olfactory bulb shortly following ocular infection and exhibits a long-term inflammatory profile in the form of effector and HSV-1-specific T cells

Journal of neuroinflammation, 2017-06, Vol.14 (1), p.124-124, Article 124

2017

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Noncognate Signals Drive Enhanced Effector CD8 + T Cell Responses through an IFNAR1-Dependent Pathway after Infection with the Prototypic Vaccine, 0ΔNLS, against Herpes Simplex Virus 1

Journal of virology, 2022-03, Vol.96 (6), p.e0172421-e0172421

2022

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HSV-1 0∆NLS vaccine elicits a robust B lymphocyte response and preserves vision without HSV-1 glycoprotein M or thymidine kinase recognition

Scientific reports, 2022-09, Vol.12 (1), p.15920-15920, Article 15920

2022

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An intact complement system dampens cornea inflammation during acute primary HSV-1 infection

Scientific reports, 2021-05, Vol.11 (1), p.10247-10247, Article 10247

2021

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Long‐term consequences of topical dexamethasone treatment during acute corneal HSV‐1 infection on the immune system

Journal of leukocyte biology, 2017-05, Vol.101 (5), p.1253-1261

2017

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The Neonatal Fc Receptor and Complement Fixation Facilitate Prophylactic Vaccine-Mediated Humoral Protection against Viral Infection in the Ocular Mucosa

The Journal of immunology (1950), 2017-09, Vol.199 (5), p.1898-1911

2017

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Tumor necrosis factor alpha and interleukin-6 facilitate corneal lymphangiogenesis in response to herpes simplex virus 1 infection

Journal of virology, 2014-12, Vol.88 (24), p.14451-14457

2014

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Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency

The Journal of immunology (1950), 2016-08, Vol.197 (4), p.1262-1275

2016

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Susceptibility of CCR5-Deficient Mice to Genital Herpes Simplex Virus Type 2 Is Linked to NK Cell Mobilization

Journal of Virology, 2007-04, Vol.81 (8), p.3704-3713

2007

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Complement Suppresses the Initial Type 1 Interferon Response to Ocular Herpes Simplex Virus Type 1 Infection in Mice

Pathogens (Basel), 2024-01, Vol.13 (1), p.74

2024

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CXCR3 Deficiency Increases Susceptibility to Genital Herpes Simplex Virus Type 2 Infection: Uncoupling of CD8+ T-Cell Effector Function but Not Migration

Journal of Virology, 2009-09, Vol.83 (18), p.9486-9501

2009

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Dysregulation of CXCR3 Signaling due to CXCL10 Deficiency Impairs the Antiviral Response to Herpes Simplex Virus 1 Infection

The Journal of immunology (1950), 2008-12, Vol.181 (11), p.7985-7993

2008

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Vaccine-induced antibodies target sequestered viral antigens to prevent ocular HSV-1 pathogenesis, preserve vision, and preempt productive neuronal infection

Mucosal immunology, 2019-05, Vol.12 (3), p.827-839

2019

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Lack of neonatal Fc receptor does not diminish the efficacy of the HSV-1 0ΔNLS vaccine against ocular HSV-1 challenge

Vaccine, 2021-04, Vol.39 (18), p.2526-2536

2021

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Herpes simplex type I (HSV-1) infection of the nervous system: Is an immune response a good thing?

Journal of neuroimmunology, 2010-03, Vol.220 (1), p.1-9

2010

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Microglia and a functional type I IFN pathway are required to counter HSV-1-driven brain lateral ventricle enlargement and encephalitis

The Journal of immunology (1950), 2013-03, Vol.190 (6), p.2807-2817

2013

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The herpes simplex virus-1 transactivator infected cell protein-4 drives VEGF-A dependent neovascularization

PLoS pathogens, 2011-10, Vol.7 (10), p.e1002278-e1002278

2011

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The use of human cornea organotypic cultures to study herpes simplex virus type 1 (HSV-1)-induced inflammation

Graefe's archive for clinical and experimental ophthalmology, 2015-10, Vol.253 (10), p.1721-1728

2015

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Herpesvirus-Associated Lymphadenitis Distorts Fibroblastic Reticular Cell Microarchitecture and Attenuates CD8 T Cell Responses to Neurotropic Infection in Mice Lacking the STING-IFNα/β Defense Pathways

The Journal of immunology (1950), 2016-09, Vol.197 (6), p.2338-2352

2016

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A Highly Efficacious Herpes Simplex Virus 1 Vaccine Blocks Viral Pathogenesis and Prevents Corneal Immunopathology via Humoral Immunity

Journal of virology, 2016-06, Vol.90 (11), p.5514-5529

2016

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Comparison of the Host Immune Response to Herpes Simplex Virus 1 (HSV-1) and HSV-2 at Two Different Mucosal Sites

Journal of Virology, 2012-07, Vol.86 (13), p.7454-7458

2012

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Loss of the Type I Interferon Pathway Increases Vulnerability of Mice to Genital Herpes Simplex Virus 2 Infection

Journal of Virology, 2011-02, Vol.85 (4), p.1625-1633

2011

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Ocular Glands Become Infected Secondarily to Infectious Keratitis and Play a Role in Corneal Resistance to Infection

Journal of virology, 2019-08, Vol.93 (16)

2019

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Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells

Type I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental Malaria

Herpes simplex virus-1 infects the olfactory bulb shortly following ocular infection and exhibits a long-term inflammatory profile in the form of effector and HSV-1-specific T cells

Noncognate Signals Drive Enhanced Effector CD8 + T Cell Responses through an IFNAR1-Dependent Pathway after Infection with the Prototypic Vaccine, 0ΔNLS, against Herpes Simplex Virus 1

HSV-1 0∆NLS vaccine elicits a robust B lymphocyte response and preserves vision without HSV-1 glycoprotein M or thymidine kinase recognition

An intact complement system dampens cornea inflammation during acute primary HSV-1 infection

Long‐term consequences of topical dexamethasone treatment during acute corneal HSV‐1 infection on the immune system

The Neonatal Fc Receptor and Complement Fixation Facilitate Prophylactic Vaccine-Mediated Humoral Protection against Viral Infection in the Ocular Mucosa

Tumor necrosis factor alpha and interleukin-6 facilitate corneal lymphangiogenesis in response to herpes simplex virus 1 infection

Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency

Susceptibility of CCR5-Deficient Mice to Genital Herpes Simplex Virus Type 2 Is Linked to NK Cell Mobilization

Complement Suppresses the Initial Type 1 Interferon Response to Ocular Herpes Simplex Virus Type 1 Infection in Mice

CXCR3 Deficiency Increases Susceptibility to Genital Herpes Simplex Virus Type 2 Infection: Uncoupling of CD8+ T-Cell Effector Function but Not Migration

Dysregulation of CXCR3 Signaling due to CXCL10 Deficiency Impairs the Antiviral Response to Herpes Simplex Virus 1 Infection

Vaccine-induced antibodies target sequestered viral antigens to prevent ocular HSV-1 pathogenesis, preserve vision, and preempt productive neuronal infection

Lack of neonatal Fc receptor does not diminish the efficacy of the HSV-1 0ΔNLS vaccine against ocular HSV-1 challenge

Herpes simplex type I (HSV-1) infection of the nervous system: Is an immune response a good thing?

Microglia and a functional type I IFN pathway are required to counter HSV-1-driven brain lateral ventricle enlargement and encephalitis

The herpes simplex virus-1 transactivator infected cell protein-4 drives VEGF-A dependent neovascularization

The use of human cornea organotypic cultures to study herpes simplex virus type 1 (HSV-1)-induced inflammation

Herpesvirus-Associated Lymphadenitis Distorts Fibroblastic Reticular Cell Microarchitecture and Attenuates CD8 T Cell Responses to Neurotropic Infection in Mice Lacking the STING-IFNα/β Defense Pathways

A Highly Efficacious Herpes Simplex Virus 1 Vaccine Blocks Viral Pathogenesis and Prevents Corneal Immunopathology via Humoral Immunity

Comparison of the Host Immune Response to Herpes Simplex Virus 1 (HSV-1) and HSV-2 at Two Different Mucosal Sites

Loss of the Type I Interferon Pathway Increases Vulnerability of Mice to Genital Herpes Simplex Virus 2 Infection

Ocular Glands Become Infected Secondarily to Infectious Keratitis and Play a Role in Corneal Resistance to Infection

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