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Long-Term Blockade of Nociceptive Nav1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Biomolecules (Basel, Switzerland), 2022-10, Vol.12 (11), p.1571

2022

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Chimaeric mice deficient in dystroglycans develop muscular dystrophy and have disrupted myoneural synapses

Nature genetics, 1999-11, Vol.23 (3), p.338-342

1999

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Nav1.6 promotes inflammation and neuronal degeneration in a mouse model of multiple sclerosis

Journal of neuroinflammation, 2019-11, Vol.16 (1), p.215-215, Article 215

2019

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Voltage‐gated sodium channel‐dependent retroaxonal modulation of photoreceptor function during post‐natal development in mice

Developmental neurobiology (Hoboken, N.J.), 2021-05, Vol.81 (4), p.353-365

2021

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Dopamine modulation of rod pathway signaling by suppression of GABAC feedback to rod-driven depolarizing bipolar cells

The European journal of neuroscience, 2015-09, Vol.42 (6), p.2258-2270

2015

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Mice Heterozygous for the Sodium Channel Scn8a (Nav1.6) Have Reduced Inflammatory Responses During EAE and Following LPS Challenge

Frontiers in immunology, 2021-03, Vol.12, p.533423

2021

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Contribution of Nav1.8 sodium channels to retinal function

Neuroscience, 2017-01, Vol.340, p.279-290

2017

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Contribution of Na v 1.8 sodium channels to retinal function

Neuroscience, 2017-01, Vol.340, p.279-290

2017

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Glial Reaction in the Spinal Cord of the Degenerating Muscle Mouse (Scn8admu)

Neurochemical research, 2015-01, Vol.40 (1), p.124-129

2015

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Contribution of retinal ganglion cells to the mouse electroretinogram

Documenta ophthalmologica, 2014-06, Vol.128 (3), p.155-168

2014

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Reduced Retinal Function in the Absence of Nav1.6

PloS one, 2012-02, Vol.7 (2), p.e31476

2012

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Hypomorphic Smn knockdown C2C12 myoblasts reveal intrinsic defects in myoblast fusion and myotube morphology

Experimental cell research, 2005-11, Vol.311 (1), p.49-61

2005

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Reduced Retinal Function in the Absence of Na(v)1.6

PloS one, 2012, Vol.7 (2), p.e31476

2012

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D1 Dopamine receptors modulate cone ON bipolar cell Nav channels to control daily rhythms in photopic vision

Chronobiology international, 2015-02, Vol.32 (1), p.48-58

2015

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Increase of CGRP Expression in Motor Endplates Within Fore and Hind Limb Muscles of the Degenerating Muscle Mouse (Scn8a dmu )

Cellular and molecular neurobiology, 2011-01, Vol.31 (1), p.155-161

2011

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Long-Term Blockade of Nociceptive Na[sub.v]1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Biomolecules (Basel, Switzerland), 2022-10, Vol.12 (11)

2022

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Increase of c-Fos and c-Jun Expression in Spinal and Cranial Motoneurons of the Degenerating Muscle Mouse (Scn8a dmu )

Cellular and molecular neurobiology, 2010-07, Vol.30 (5), p.737-742

2010

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Voltage-gated sodium channels contribute to the b-wave of the rodent electroretinogram by mediating input to rod bipolar cell GABA(c) receptors

Experimental eye research, 2013-11, Vol.116, p.279-290

2013

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Long-Term Blockade of Nociceptive Na v 1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Biomolecules (Basel, Switzerland), 2022-10, Vol.12 (11)

2022

Volltextzugriff (PDF)

Physiological Maturation of Photoreceptors Depends on the Voltage-Gated Sodium Channel NaV1.6 (Scn8a)

The Journal of neuroscience, 2005-05, Vol.25 (20), p.5046-5050

2005

Volltextzugriff (PDF)

Voltage-gated sodium channels contribute to the b-wave of the rodent electroretinogram by mediating input to rod bipolar cell GABAc receptors

Experimental eye research, 2013-11, Vol.116, p.279-290

2013

Volltextzugriff (PDF)

Contribution of Nav 1.8 sodium channels to retinal function

Neuroscience, 2016

2016

Volltextzugriff (PDF)

Dopamine modulation of rod pathway signaling by suppression of GABA C feedback to rod‐driven depolarizing bipolar cells

The European journal of neuroscience, 2015-09, Vol.42 (6), p.2258-2270

2015

Volltextzugriff (PDF)

The Dystroglycan Complex Is Necessary for Stabilization of Acetylcholine Receptor Clusters at Neuromuscular Junctions and Formation of the Synaptic Basement Membrane

The Journal of cell biology, 2001-02, Vol.152 (3), p.435-450

2001

Volltextzugriff (PDF)

Reduced Retinal Function in the Absence of Na.sub.v1.6

PloS one, 2012-02, Vol.7 (2), p.e31476

2012

Volltextzugriff (PDF)

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Long-Term Blockade of Nociceptive Nav1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Chimaeric mice deficient in dystroglycans develop muscular dystrophy and have disrupted myoneural synapses

Nav1.6 promotes inflammation and neuronal degeneration in a mouse model of multiple sclerosis

Voltage‐gated sodium channel‐dependent retroaxonal modulation of photoreceptor function during post‐natal development in mice

Dopamine modulation of rod pathway signaling by suppression of GABAC feedback to rod-driven depolarizing bipolar cells

Mice Heterozygous for the Sodium Channel Scn8a (Nav1.6) Have Reduced Inflammatory Responses During EAE and Following LPS Challenge

Contribution of Nav1.8 sodium channels to retinal function

Contribution of Na v 1.8 sodium channels to retinal function

Glial Reaction in the Spinal Cord of the Degenerating Muscle Mouse (Scn8admu)

Contribution of retinal ganglion cells to the mouse electroretinogram

Reduced Retinal Function in the Absence of Nav1.6

Hypomorphic Smn knockdown C2C12 myoblasts reveal intrinsic defects in myoblast fusion and myotube morphology

Reduced Retinal Function in the Absence of Na(v)1.6

D1 Dopamine receptors modulate cone ON bipolar cell Nav channels to control daily rhythms in photopic vision

Increase of CGRP Expression in Motor Endplates Within Fore and Hind Limb Muscles of the Degenerating Muscle Mouse (Scn8a dmu )

Long-Term Blockade of Nociceptive Na[sub.v]1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Increase of c-Fos and c-Jun Expression in Spinal and Cranial Motoneurons of the Degenerating Muscle Mouse (Scn8a dmu )

Voltage-gated sodium channels contribute to the b-wave of the rodent electroretinogram by mediating input to rod bipolar cell GABA(c) receptors

Long-Term Blockade of Nociceptive Na v 1.7 Channels Is Analgesic in Rat Models of Knee Arthritis

Physiological Maturation of Photoreceptors Depends on the Voltage-Gated Sodium Channel NaV1.6 (Scn8a)

Voltage-gated sodium channels contribute to the b-wave of the rodent electroretinogram by mediating input to rod bipolar cell GABAc receptors

Contribution of Nav 1.8 sodium channels to retinal function

Dopamine modulation of rod pathway signaling by suppression of GABA C feedback to rod‐driven depolarizing bipolar cells

The Dystroglycan Complex Is Necessary for Stabilization of Acetylcholine Receptor Clusters at Neuromuscular Junctions and Formation of the Synaptic Basement Membrane

Reduced Retinal Function in the Absence of Na.sub.v1.6

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