UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

The role of insufficient copper in lipid synthesis and fatty‐liver disease

IUBMB life, 2017-04, Vol.69 (4), p.263-270

2017

Volltextzugriff (PDF)

Copper-dependent amino oxidase 3 governs selection of metabolic fuels in adipocytes

PLoS biology, 2018-09, Vol.16 (9), p.e2006519

2018

Volltextzugriff (PDF)

Copper homeostasis

The New phytologist, 2009-06, Vol.182 (4), p.799-816

2009

Volltextzugriff (PDF)

Cellular multitasking: The dual role of human Cu-ATPases in cofactor delivery and intracellular copper balance

Archives of biochemistry and biophysics, 2008-08, Vol.476 (1), p.22-32

2008

Volltextzugriff (PDF)

Nutrition Information Brief—Copper

Advances in nutrition (Bethesda, Md.), 2022-03, Vol.13 (2), p.681-683

2022

Volltextzugriff (PDF)

Copper Toxicity Is Not Just Oxidative Damage: Zinc Systems and Insight from Wilson Disease

Biomedicines, 2021-03, Vol.9 (3), p.316

2021

Volltextzugriff (PDF)

COMMD1 and PtdIns(4,5)P2 interaction maintain ATP7B copper transporter trafficking fidelity in HepG2 cells

Journal of cell science, 2019-10, Vol.132 (19)

2019

Volltextzugriff (PDF)

COMMD1 and PtdIns(4,5)P 2 interaction maintain ATP7B copper transporter trafficking fidelity in HepG2 cells

Journal of cell science, 2019-10, Vol.132 (19)

2019

Volltextzugriff (PDF)

Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease

The Journal of nutritional biochemistry, 2015-10, Vol.26 (10), p.996-1006

2015

Volltextzugriff (PDF)

Copper modulates sex-specific fructose hepatoxicity in nonalcoholic fatty liver disease (NALFD) Wistar rat models

The Journal of nutritional biochemistry, 2020-04, Vol.78, p.108316, Article 108316

2020

Volltextzugriff (PDF)

Pattern of SARS-CoV-2 variant B.1.1.519 emergence in Alaska

Scientific reports, 2022-11, Vol.12 (1), p.20662-8, Article 20662

2022

Volltextzugriff (PDF)

High Copper Selectively Alters Lipid Metabolism and Cell Cycle Machinery in the Mouse Model of Wilson Disease

The Journal of biological chemistry, 2007-03, Vol.282 (11), p.8343-8355

2007

Volltextzugriff (PDF)

Systems biology approach to Wilson’s disease

Biometals, 2011-06, Vol.24 (3), p.455-466

2011

Volltextzugriff (PDF)

Wilson Disease at a Single Cell Level: INTRACELLULAR COPPER TRAFFICKING ACTIVATES COMPARTMENT-SPECIFIC RESPONSES IN HEPATOCYTES

The Journal of biological chemistry, 2010-10, Vol.285 (40), p.30875-30883

2010

Volltextzugriff (PDF)

Consequences of Copper Accumulation in the Livers of the Atp7b −/− (Wilson Disease Gene) Knockout Mice

The American journal of pathology, 2006-02, Vol.168 (2), p.423-434

2006

Volltextzugriff (PDF)

A Repeat Pattern of Founder Events for SARS-CoV-2 Variants in Alaska

Viruses, 2023-01, Vol.15 (1), p.222

2023

Volltextzugriff (PDF)

Host Cell Copper Transporters CTR1 and ATP7A are important for Influenza A virus replication

Virology journal, 2017-01, Vol.14 (1), p.11, Article 11

2017

Volltextzugriff (PDF)

Elevated Copper Remodels Hepatic RNA Processing Machinery in the Mouse Model of Wilson's Disease

Journal of molecular biology, 2011-02, Vol.406 (1), p.44-58

2011

Volltextzugriff (PDF)

Chronic copper treatment prevents the liver critical balance transcription response induced by acetaminophen

Journal of trace elements in medicine and biology, 2019-05, Vol.53, p.113-119

2019

Volltextzugriff (PDF)

Altered zinc balance in the Atp7b -/- mouse reveals a mechanism of copper toxicity in Wilson disease

Metallomics, 2018-11, Vol.10 (11), p.1595-1606

2018

Volltextzugriff (PDF)

Copper deficiency is an independent risk factor for mortality in patients with advanced liver disease

Hepatology communications, 2023-03, Vol.7 (3), p.e0076-e0076

2023

Volltextzugriff (PDF)

COMMD1 Forms Oligomeric Complexes Targeted to the Endocytic Membranes via Specific Interactions with Phosphatidylinositol 4,5-Bisphosphate

The Journal of biological chemistry, 2009-01, Vol.284 (1), p.696-707

2009

Volltextzugriff (PDF)

Higher plants possess two different types of ATX1-like copper chaperones

Biochemical and biophysical research communications, 2007-03, Vol.354 (2), p.385-390

2007

Volltextzugriff (PDF)

Copper Deficiency in Liver Diseases: A Case Series and Pathophysiological Considerations

Hepatology communications, 2019-08, Vol.3 (8), p.1159-1165

2019

Volltextzugriff (PDF)

Analysis of Transgenic Indian Mustard Plants for Phytoremediation of Metal-Contaminated Mine Tailings

Journal of environmental quality, 2003-03, Vol.32 (2), p.432-440

2003

Volltextzugriff (PDF)

1 – 25 von 51

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(49)
Mit Volltextzugriff
(49)

Publikationsform

Sprache

Englisch
(51)

Erscheinungsjahr

n.n

Thema

Science & Technology
(29)
Life Sciences & Biomedicine
(28)
Copper
(20)
Biochemistry & Molecular Biology
(17)
Copper - Metabolism
(15)
Humans
(13)
Animals
(11)
Copper-Transporting Atpases
(9)
Disease Models, Animal
(8)
Mice
(7)
Adenosine Triphosphatases - Genetics
(6)
Adenosine Triphosphatases - Metabolism
(6)
Cation Transport Proteins - Genetics
(6)
Homeostasis
(6)
Liver
(6)
Liver - Metabolism
(6)
Male
(6)
Cation Transport Proteins - Metabolism
(5)
Cell Biology
(5)
Female
(5)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

The role of insufficient copper in lipid synthesis and fatty‐liver disease

Copper-dependent amino oxidase 3 governs selection of metabolic fuels in adipocytes

Copper homeostasis

Cellular multitasking: The dual role of human Cu-ATPases in cofactor delivery and intracellular copper balance

Nutrition Information Brief—Copper

Copper Toxicity Is Not Just Oxidative Damage: Zinc Systems and Insight from Wilson Disease

COMMD1 and PtdIns(4,5)P2 interaction maintain ATP7B copper transporter trafficking fidelity in HepG2 cells

COMMD1 and PtdIns(4,5)P 2 interaction maintain ATP7B copper transporter trafficking fidelity in HepG2 cells

Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease

Copper modulates sex-specific fructose hepatoxicity in nonalcoholic fatty liver disease (NALFD) Wistar rat models

Pattern of SARS-CoV-2 variant B.1.1.519 emergence in Alaska

High Copper Selectively Alters Lipid Metabolism and Cell Cycle Machinery in the Mouse Model of Wilson Disease

Systems biology approach to Wilson’s disease

Wilson Disease at a Single Cell Level: INTRACELLULAR COPPER TRAFFICKING ACTIVATES COMPARTMENT-SPECIFIC RESPONSES IN HEPATOCYTES

Consequences of Copper Accumulation in the Livers of the Atp7b −/− (Wilson Disease Gene) Knockout Mice

A Repeat Pattern of Founder Events for SARS-CoV-2 Variants in Alaska

Host Cell Copper Transporters CTR1 and ATP7A are important for Influenza A virus replication

Elevated Copper Remodels Hepatic RNA Processing Machinery in the Mouse Model of Wilson's Disease

Chronic copper treatment prevents the liver critical balance transcription response induced by acetaminophen

Altered zinc balance in the Atp7b -/- mouse reveals a mechanism of copper toxicity in Wilson disease

Copper deficiency is an independent risk factor for mortality in patients with advanced liver disease

COMMD1 Forms Oligomeric Complexes Targeted to the Endocytic Membranes via Specific Interactions with Phosphatidylinositol 4,5-Bisphosphate

Higher plants possess two different types of ATX1-like copper chaperones

Copper Deficiency in Liver Diseases: A Case Series and Pathophysiological Considerations

Analysis of Transgenic Indian Mustard Plants for Phytoremediation of Metal-Contaminated Mine Tailings

Suchergebnisse filtern