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Hemopoietic-specific Sf3b1-K700E knock-in mice display the splicing defect seen in human MDS but develop anemia without ring sideroblasts

Leukemia, 2017-03, Vol.31 (3), p.720-727

2017

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The controversial role of the Hedgehog pathway in normal and malignant hematopoiesis

Leukemia, 2011-11, Vol.25 (11), p.1665-1673

2011

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Characterization of Novel Oral Splicing Modulator, H3B-8800, Identifies the Mechanistic Basis for its Preferential Lethality Towards Spliceosome-Mutant Myeloid Malignancy Models

Leukemia research, 2017-04, Vol.55, p.S11-S11

2017

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527 Is CRAF required for the maintenance of KRAS mutant non-small cell lung cancer?

European journal of cancer (1990), 2014-11, Vol.50, p.171-171

2014

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456 SF3B1 mutations induce disease relevant aberrant mRNA splicing in cancer and confer sensitivity to spliceosome inhibition

European journal of cancer (1990), 2014-11, Vol.50, p.149-149

2014

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21 SF3B1 MUTATIONS INDUCE ABERRANT SPLICING LEADING TO A BLOCK IN ERYTHROID DIFFERENTIATION AND COMPETITIVE ADVANTAGE

Leukemia research, 2015-04, Vol.39, p.S9-S9

2015

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Arsenic trioxide and thalidomide combination produces multi-lineage hematological responses in myelodysplastic syndromes patients, particularly in those with high pre-therapy EVI1 expression

Leukemia research, 2004-08, Vol.28 (8), p.791-803

2004

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68 (PB056): REM-422, a potent, selective, oral small moleculemRNA degrader of the MYB oncogene, induces regressions in mouse patient-derived xenograft models of adenoid cystic carcinoma

European journal of cancer (1990), 2024-10, Vol.211, p.114595, Article 114595

2024

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Pulsed ATRA as single therapy restores long-term remission in PML-RARα-positive acute promyelocytic leukemia patients : real time quantification of minimal residual disease. A pilot study

Leukemia, 2001-11, Vol.15 (11), p.1696-1700

2001

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Real-time quantification of different types of bcr-abl transcript in chronic myeloid leukemia

Haematologica (Roma), 2001-03, Vol.86 (3), p.252

2001

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Targeted therapies in myelodysplastic syndromes: ASH 2003 review

Seminars in hematology, 2004-04, Vol.41 (2 Suppl 4), p.13-20

2004

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Polymerase chain reaction-based detection of minimal residual disease in multiple myeloma patients receiving allogeneic stem cell transplantation

Haematologica (Roma), 2000-09, Vol.85 (9), p.930

2000

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EVI1 induces myelodysplastic syndrome in mice

The Journal of clinical investigation, 2004-09, Vol.114 (5), p.713-719

2004

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Molecular monitoring of acute myeloid leukemia associated with inv(16): threshold of CBFβ/MYH11 transcript copy number above which relapse occurs and below which continuous Complete Remission is likely

Leukemia, 2003-03, Vol.17 (3), p.650-651

2003

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20 - Characterization of Novel Oral Splicing Modulator, H3B-8800, Identifies the Mechanistic Basis for its Preferential Lethality Towards Spliceosome-Mutant Myeloid Malignancy Models

Leukemia research, 2017-04, Vol.55, p.S11-S11

2017

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Real-time quantitation of minimal residual disease in inv(16)-positive acute myeloid leukemia may indicate risk for clinical relapse and may identify patients in a curable state

Blood, 2002-01, Vol.99 (2), p.443-449

2002

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The role of EVI1 in normal and leukemic cells

Blood cells, molecules, & diseases, 2003-09, Vol.31 (2), p.206-212

2003

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PS1034 PHASE I DOSE ESCALATION CLINICAL TRIAL OF H3B‐8800, A SPLICING MODULATOR, IN PATIENTS WITH ADVANCED MYELOID MALIGNANCIES

HemaSphere, 2019-06, Vol.3 (S1), p.467-n/a

2019

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Independent Prognostic Importance of a Restrictive Left Ventricular Filling Pattern After Myocardial Infarction : An Individual Patient Meta-Analysis: Meta-Analysis Research Group in Echocardiography Acute Myocardial Infarction

Circulation (New York, N.Y.), 2008-05, Vol.117 (20), p.2591-2598

2008

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EVI1 Abrogates Interferon-α Response by Selectively Blocking PML Induction

The Journal of biological chemistry, 2005-01, Vol.280 (1), p.428-436

2005

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EVI1-MDS1/EVI1 (ecotropic viral integration site 1 (EVI1) and myelodysplastic syndrome 1 (MDS1)-EVI1)

Atlas of genetics and cytogenetics in oncology and haematology, 2011-02 (3)

2011

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Association of 3q21q26 syndrome with different RPN1/EVI1 fusion transcripts

Haematologica (Roma), 2003-11, Vol.88 (11), p.1221

2003

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AML1-ETO decreases ETO-2 (MTG16) interactions with nuclear receptor corepressor, an effect that impairs granulocyte differentiation

Cancer research (Chicago, Ill.), 2004-07, Vol.64 (13), p.4547-4554

2004

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P/CAF and GCN5 acetylate the AML1/MDS1/EVI1 fusion oncoprotein

Biochemical and biophysical research communications, 2003-08, Vol.307 (4), p.980-986

2003

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Patterns of AML1-ETO transcript expression in patients with acute myeloid leukemia and t(8;21) in complete hematologic remission

Haematologica (Roma), 2004-01, Vol.89 (1), p.103

2004

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Hemopoietic-specific Sf3b1-K700E knock-in mice display the splicing defect seen in human MDS but develop anemia without ring sideroblasts

The controversial role of the Hedgehog pathway in normal and malignant hematopoiesis

Characterization of Novel Oral Splicing Modulator, H3B-8800, Identifies the Mechanistic Basis for its Preferential Lethality Towards Spliceosome-Mutant Myeloid Malignancy Models

527 Is CRAF required for the maintenance of KRAS mutant non-small cell lung cancer?

456 SF3B1 mutations induce disease relevant aberrant mRNA splicing in cancer and confer sensitivity to spliceosome inhibition

21 SF3B1 MUTATIONS INDUCE ABERRANT SPLICING LEADING TO A BLOCK IN ERYTHROID DIFFERENTIATION AND COMPETITIVE ADVANTAGE

Arsenic trioxide and thalidomide combination produces multi-lineage hematological responses in myelodysplastic syndromes patients, particularly in those with high pre-therapy EVI1 expression

68 (PB056): REM-422, a potent, selective, oral small moleculemRNA degrader of the MYB oncogene, induces regressions in mouse patient-derived xenograft models of adenoid cystic carcinoma

Pulsed ATRA as single therapy restores long-term remission in PML-RARα-positive acute promyelocytic leukemia patients : real time quantification of minimal residual disease. A pilot study

Real-time quantification of different types of bcr-abl transcript in chronic myeloid leukemia

Targeted therapies in myelodysplastic syndromes: ASH 2003 review

Polymerase chain reaction-based detection of minimal residual disease in multiple myeloma patients receiving allogeneic stem cell transplantation

EVI1 induces myelodysplastic syndrome in mice

Molecular monitoring of acute myeloid leukemia associated with inv(16): threshold of CBFβ/MYH11 transcript copy number above which relapse occurs and below which continuous Complete Remission is likely

20 - Characterization of Novel Oral Splicing Modulator, H3B-8800, Identifies the Mechanistic Basis for its Preferential Lethality Towards Spliceosome-Mutant Myeloid Malignancy Models

Real-time quantitation of minimal residual disease in inv(16)-positive acute myeloid leukemia may indicate risk for clinical relapse and may identify patients in a curable state

The role of EVI1 in normal and leukemic cells

PS1034 PHASE I DOSE ESCALATION CLINICAL TRIAL OF H3B‐8800, A SPLICING MODULATOR, IN PATIENTS WITH ADVANCED MYELOID MALIGNANCIES

Independent Prognostic Importance of a Restrictive Left Ventricular Filling Pattern After Myocardial Infarction : An Individual Patient Meta-Analysis: Meta-Analysis Research Group in Echocardiography Acute Myocardial Infarction

EVI1 Abrogates Interferon-α Response by Selectively Blocking PML Induction

EVI1-MDS1/EVI1 (ecotropic viral integration site 1 (EVI1) and myelodysplastic syndrome 1 (MDS1)-EVI1)

Association of 3q21q26 syndrome with different RPN1/EVI1 fusion transcripts

AML1-ETO decreases ETO-2 (MTG16) interactions with nuclear receptor corepressor, an effect that impairs granulocyte differentiation

P/CAF and GCN5 acetylate the AML1/MDS1/EVI1 fusion oncoprotein

Patterns of AML1-ETO transcript expression in patients with acute myeloid leukemia and t(8;21) in complete hematologic remission

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