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Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophy

Human molecular genetics, 2005-06, Vol.14 (11), p.1539-1547

2005

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A bichromatic fluorescent reporter for cell-based screens of alternative splicing

Nucleic acids research, 2006-12, Vol.34 (22), p.e148

2006

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Redundant Pathways for Negative Feedback Regulation of Bile Acid Production

Developmental cell, 2002-06, Vol.2 (6), p.721-731

2002

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Delayed Wound Healing in Keratin 6a Knockout Mice

Molecular and Cellular Biology, 2000-07, Vol.20 (14), p.5248-5255

2000

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Quasi-Normal Cornified Cell Envelopes in Loricrin Knockout Mice Imply the Existence of a Loricrin Backup System

Journal of investigative dermatology, 2002-01, Vol.118 (1), p.102-109

2002

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Human keratin-1.bcl-2 transgenic mice aberrantly express keratin 6, exhibit reduced sensitivity to keratinocyte cell death induction, and are susceptible to skin tumor formation

Oncogene, 1998-02, Vol.16 (7), p.853-863

1998

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Identification of a major keratinocyte cell envelope protein, loricrin

Cell, 1990-06, Vol.61 (6), p.1103-1112

1990

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The Proximal Promoter of the Mouse Loricrin Gene Contains a Functional AP-1 Element and Directs Keratinocyte-specific but Not Differentiation-specific Expression

The Journal of biological chemistry, 1995-05, Vol.270 (18), p.10792-10799

1995

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Expression of a Dominant-Negative Type II Transforming Growth Factor β (TGF-β ) Receptor in the Epidermis of Transgenic Mice Blocks TGF-β -Mediated Growth Inhibition

Proceedings of the National Academy of Sciences - PNAS, 1997-03, Vol.94 (6), p.2386-2391

1997

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The mouse keratin 6 isoforms are differentially expressed in the hair follicle, footpad, tongue and activated epidermis

Differentiation (London), 1999-10, Vol.65 (2), p.119-130

1999

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Lessons from Loricrin-Deficient Mice: Compensatory Mechanisms Maintaining Skin Barrier Function in the Absence of a Major Cornified Envelope Protein

The Journal of cell biology, 2000-10, Vol.151 (2), p.389-400

2000

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Inhibition of Melanoma Growth by Adenoviral-Mediated HSV Thymidine Kinase Gene Transfer In Vivo

Journal of investigative dermatology, 1995-03, Vol.104 (3), p.313-317

1995

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Induction of epidermal hyperplasia, hyperkeratosis, and papillomas in transgenic mice by a targeted v-Ha-ras oncogene

Molecular carcinogenesis, 1993, Vol.7 (2), p.99-110

1993

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Characterization of loricrin regulation in vitro and in transgenic mice

Differentiation (London), 1999-05, Vol.64 (4), p.225-235

1999

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Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle

Differentiation (London), 1999-10, Vol.65 (2), p.97-112

1999

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Lessons from Loricrin-Deficient Mice

The Journal of cell biology, 2000-10, Vol.151 (2), p.389-400

2000

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Inhibition of Skin Development by Overexpression of Transforming Growth Factor β1in the Epidermis of Transgenic Mice

Proceedings of the National Academy of Sciences - PNAS, 1993-06, Vol.90 (11), p.5237-5241

1993

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Targeting expression of a dominant-negative retinoic acid receptor mutant in the epidermis of transgenic mice results in loss of barrier function

Genes & development, 1995-02, Vol.9 (3), p.317-329

1995

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Loricrin Expression Is Coordinated with Other Epidermal Proteins and the Appearance of Lipid Lamellar Granules in Development

Journal of investigative dermatology, 1995-03, Vol.104 (3), p.405-410

1995

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Formation of the rabbit zona pellucida and its relationship to ovarian follicular development

Developmental biology, 1984-11, Vol.106 (1), p.1,11-9,14

1984

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Genetic disorders of keratin: are scarring alopecias a sub-set?

Journal of dermatological science, 1994-07, Vol.7, p.S164-S169

1994

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Generation of a targeting construct to develop a mouse model for ablation of beta cell E-box transactivator 2, BETA2, in the beta cells

Gastroenterology (New York, N.Y. 1943), 2000-04, Vol.118 (4), p.A1527-A1527

2000

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Adenoviral-Mediated Herpes Simplex Virus-Thymidine Kinase Gene Transfer in Vivo for Treatment of Experimental Human Melanoma

Journal of investigative dermatology, 1996-06, Vol.106 (6), p.1163-1168

1996

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A transgenic mouse model that recapitulates the clinical features of both neonatal and adult forms of the skin disease epidermolytic hyperkeratosis

Differentiation (London), 1996-12, Vol.61 (2), p.129-139

1996

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Hyperplasia, hyperkeratosis and benign tumor production in transgenic mice by a targeted v-fos oncogene suggest a role for fos in epidermal differentiation and neoplasia

Oncogene, 1993-08, Vol.8 (8), p.2145-2157

1993

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Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophy

A bichromatic fluorescent reporter for cell-based screens of alternative splicing

Redundant Pathways for Negative Feedback Regulation of Bile Acid Production

Delayed Wound Healing in Keratin 6a Knockout Mice

Quasi-Normal Cornified Cell Envelopes in Loricrin Knockout Mice Imply the Existence of a Loricrin Backup System

Human keratin-1.bcl-2 transgenic mice aberrantly express keratin 6, exhibit reduced sensitivity to keratinocyte cell death induction, and are susceptible to skin tumor formation

Identification of a major keratinocyte cell envelope protein, loricrin

The Proximal Promoter of the Mouse Loricrin Gene Contains a Functional AP-1 Element and Directs Keratinocyte-specific but Not Differentiation-specific Expression

Expression of a Dominant-Negative Type II Transforming Growth Factor β (TGF-β ) Receptor in the Epidermis of Transgenic Mice Blocks TGF-β -Mediated Growth Inhibition

The mouse keratin 6 isoforms are differentially expressed in the hair follicle, footpad, tongue and activated epidermis

Lessons from Loricrin-Deficient Mice: Compensatory Mechanisms Maintaining Skin Barrier Function in the Absence of a Major Cornified Envelope Protein

Inhibition of Melanoma Growth by Adenoviral-Mediated HSV Thymidine Kinase Gene Transfer In Vivo

Induction of epidermal hyperplasia, hyperkeratosis, and papillomas in transgenic mice by a targeted v-Ha-ras oncogene

Characterization of loricrin regulation in vitro and in transgenic mice

Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle

Lessons from Loricrin-Deficient Mice

Inhibition of Skin Development by Overexpression of Transforming Growth Factor β1in the Epidermis of Transgenic Mice

Targeting expression of a dominant-negative retinoic acid receptor mutant in the epidermis of transgenic mice results in loss of barrier function

Loricrin Expression Is Coordinated with Other Epidermal Proteins and the Appearance of Lipid Lamellar Granules in Development

Formation of the rabbit zona pellucida and its relationship to ovarian follicular development

Genetic disorders of keratin: are scarring alopecias a sub-set?

Generation of a targeting construct to develop a mouse model for ablation of beta cell E-box transactivator 2, BETA2, in the beta cells

Adenoviral-Mediated Herpes Simplex Virus-Thymidine Kinase Gene Transfer in Vivo for Treatment of Experimental Human Melanoma

A transgenic mouse model that recapitulates the clinical features of both neonatal and adult forms of the skin disease epidermolytic hyperkeratosis

Hyperplasia, hyperkeratosis and benign tumor production in transgenic mice by a targeted v-fos oncogene suggest a role for fos in epidermal differentiation and neoplasia

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