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Drosophila as a model to explore secondary injury cascades after traumatic brain injury

Biomedicine & pharmacotherapy, 2021-10, Vol.142, p.112079-112079, Article 112079

2021

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Vulnerable Parkin Loss-of-Function Drosophila Dopaminergic Neurons Have Advanced Mitochondrial Aging, Mitochondrial Network Loss and Transiently Reduced Autophagosome Recruitment

Frontiers in cellular neuroscience, 2018-02, Vol.12, p.39-39

2018

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Nicotine Has a Therapeutic Window of Effectiveness in a Drosophila melanogaster Model of Parkinson’s Disease

Parkinson's disease, 2022-07, Vol.2022, p.1-11

2022

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Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease

1st ed. 2016 edition., 2016

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Folic Acid Improves Parkin-Null Drosophila Phenotypes and Transiently Reduces Vulnerable Dopaminergic Neuron Mitochondrial Hydrogen Peroxide Levels and Glutathione Redox Equilibrium

Antioxidants, 2022-10, Vol.11 (10), p.2068

2022

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Parkin loss-of-function pathology: Premature neuronal senescence induced by high levels of reactive oxygen species?

Mechanisms of ageing and development, 2017-01, Vol.161 (Pt A), p.112-120

Buhlman, Lori M.

2017

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3D quantification of autophagy activation and autophagosome-to-mitochondria recruitment in a Drosophila model of Parkinson’s disease

STAR protocols, 2021-06, Vol.2 (2), p.100408-100408, Article 100408

2021

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Mild traumatic brain injury in Drosophila melanogaster alters reactive oxygen and nitrogen species in a sex-dependent manner

Experimental neurology, 2024-02, Vol.372, p.114621-114621, Article 114621

2024

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Effects of postnatal maturation on muscarinic acetylcholine receptor distribution in hypoglossal motorneurons

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Traumatic brain injury in a Drosophila upregulates nitric oxide synthase associated with increased acute behavioral deficits and decreased survival time

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Nicotine increases lifespan and rescues olfactory and motor deficits in a Drosophila model of Parkinson's disease

Behavioural brain research, 2013-09, Vol.253, p.95-102

2013

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α6 subunit‐containing nicotinic receptors mediate low‐dose ethanol effects on ventral tegmental area neurons and ethanol reward

Addiction biology, 2018-09, Vol.23 (5), p.1079-1093

2018

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Mitochondrial ROS and Apoptosis

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.1-23

2016

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Folic Acid Improves Parkin-Null IDrosophila/I Phenotypes and Transiently Reduces Vulnerable Dopaminergic Neuron Mitochondrial Hydrogen Peroxide Levels and Glutathione Redox Equilibrium

Antioxidants, 2022-10, Vol.11 (10)

2022

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Toxin-Mediated Complex I Inhibition and Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.115-137

2016

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Dopamine Metabolism and Reactive Oxygen Species Production

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.25-47

2016

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Delivery of Biologically Active Molecules to Mitochondria

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.255-267

2016

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Parkinson’s Disease-Associated Mutations Affect Mitochondrial Function

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.139-158

2016

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Transcription Modulation of Mitochondrial Function and Related Pathways as a Therapeutic Opportunity in Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.231-253

2016

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PARKIN/PINK1 Pathway for the Selective Isolation and Degradation of Impaired Mitochondria

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.159-182

2016

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The Consequences of Damaged Mitochondrial DNA

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.49-61

2016

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Altering Mitochondrial Fusion and Fission Protein Levels Rescues Parkin and PINK1 Loss-of-Function Phenotypes

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.207-218

2016

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Ion-Catalyzed Reactive Oxygen Species in Sporadic Models of Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.75-113

2016

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Early Nicotine Exposure Is Protective in Familial and Idiopathic Models of Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.219-229

2016

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Mitochondrial Therapeutic Approaches in Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 2016, p.183-205

2016

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Drosophila as a model to explore secondary injury cascades after traumatic brain injury

Vulnerable Parkin Loss-of-Function Drosophila Dopaminergic Neurons Have Advanced Mitochondrial Aging, Mitochondrial Network Loss and Transiently Reduced Autophagosome Recruitment

Nicotine Has a Therapeutic Window of Effectiveness in a Drosophila melanogaster Model of Parkinson’s Disease

Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease

Folic Acid Improves Parkin-Null Drosophila Phenotypes and Transiently Reduces Vulnerable Dopaminergic Neuron Mitochondrial Hydrogen Peroxide Levels and Glutathione Redox Equilibrium

Parkin loss-of-function pathology: Premature neuronal senescence induced by high levels of reactive oxygen species?

3D quantification of autophagy activation and autophagosome-to-mitochondria recruitment in a Drosophila model of Parkinson’s disease

Mild traumatic brain injury in Drosophila melanogaster alters reactive oxygen and nitrogen species in a sex-dependent manner

Effects of postnatal maturation on muscarinic acetylcholine receptor distribution in hypoglossal motorneurons

Traumatic brain injury in a Drosophila upregulates nitric oxide synthase associated with increased acute behavioral deficits and decreased survival time

Nicotine increases lifespan and rescues olfactory and motor deficits in a Drosophila model of Parkinson's disease

α6 subunit‐containing nicotinic receptors mediate low‐dose ethanol effects on ventral tegmental area neurons and ethanol reward

Mitochondrial ROS and Apoptosis

Folic Acid Improves Parkin-Null IDrosophila/I Phenotypes and Transiently Reduces Vulnerable Dopaminergic Neuron Mitochondrial Hydrogen Peroxide Levels and Glutathione Redox Equilibrium

Toxin-Mediated Complex I Inhibition and Parkinson’s Disease

Dopamine Metabolism and Reactive Oxygen Species Production

Delivery of Biologically Active Molecules to Mitochondria

Parkinson’s Disease-Associated Mutations Affect Mitochondrial Function

Transcription Modulation of Mitochondrial Function and Related Pathways as a Therapeutic Opportunity in Parkinson’s Disease

PARKIN/PINK1 Pathway for the Selective Isolation and Degradation of Impaired Mitochondria

The Consequences of Damaged Mitochondrial DNA

Altering Mitochondrial Fusion and Fission Protein Levels Rescues Parkin and PINK1 Loss-of-Function Phenotypes

Ion-Catalyzed Reactive Oxygen Species in Sporadic Models of Parkinson’s Disease

Early Nicotine Exposure Is Protective in Familial and Idiopathic Models of Parkinson’s Disease

Mitochondrial Therapeutic Approaches in Parkinson’s Disease

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