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Emerging Concepts in Defective Macrophage Phagocytosis in Cystic Fibrosis

International journal of molecular sciences, 2022-07, Vol.23 (14), p.7750

2022

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Innate and Adaptive Immunity in Cystic Fibrosis

Clinics in chest medicine, 2016-03, Vol.37 (1), p.17-29

2016

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A highly efficient and faithful MDS patient-derived xenotransplantation model for pre-clinical studies

Nature communications, 2019-01, Vol.10 (1), p.366-366, Article 366

2019

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Pharmacological modulation of the AKT/microRNA-199a-5p/CAV1 pathway ameliorates cystic fibrosis lung hyper-inflammation

Nature communications, 2015-02, Vol.6 (1), p.6221-6221, Article 6221

2015

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Combined liver-cytokine humanization comes to the rescue of circulating human red blood cells

Science (American Association for the Advancement of Science), 2021-03, Vol.371 (6533), p.1019-1025

2021

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Targeting the Heme Oxygenase 1/Carbon Monoxide Pathway to Resolve Lung Hyper-Inflammation and Restore a Regulated Immune Response in Cystic Fibrosis

Frontiers in pharmacology, 2020-07, Vol.11, p.1059-1059

2020

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Abnormal trafficking and degradation of TLR4 underlie the elevated inflammatory response in cystic fibrosis

The Journal of immunology (1950), 2011-06, Vol.186 (12), p.6990-6998

2011

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Prior Influenza Infection Mitigates SARS-CoV-2 Disease in Syrian Hamsters

Viruses, 2024-02, Vol.16 (2), p.246

2024

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The carbon monoxide releasing molecule CORM-2 attenuates Pseudomonas aeruginosa biofilm formation

PloS one, 2012-04, Vol.7 (4), p.e35499

2012

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Ezrin links CFTR to TLR4 signaling to orchestrate anti-bacterial immune response in macrophages

Scientific reports, 2017-09, Vol.7 (1), p.10882-11, Article 10882

2017

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Lack of a Fusion Requirement for Development of Bone Marrow-Derived Epithelia

Science (American Association for the Advancement of Science), 2004-07, Vol.305 (5680), p.90-93

2004

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SRF is required for neutrophil migration in response to inflammation

Blood, 2014-05, Vol.123 (19), p.3027-3036

2014

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Recruitment of monocytes primed to express heme oxygenase-1 ameliorates pathological lung inflammation in cystic fibrosis

Experimental and Molecular Medicine, 2022, 54(0), , pp.1-14

2022

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Cellular Innate Immunity: An Old Game with New Players

Journal of innate immunity, 2017-01, Vol.9 (2), p.111-125

2017

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Cystic Fibrosis Lung Immunity: The Role of the Macrophage

Journal of innate immunity, 2016, Vol.8 (6), p.550-563

2016

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Nonhematopoietic Cells are the Primary Source of Bone Marrow-Derived Lung Epithelial Cells

Stem cells (Dayton, Ohio), 2012-03, Vol.30 (3), p.491-499

2012

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Update on Innate and Adaptive Immunity in Cystic Fibrosis

Clinics in chest medicine, 2022-12, Vol.43 (4), p.603-615

2022

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Role for MKL1 in megakaryocytic maturation

Blood, 2009-03, Vol.113 (12), p.2826-2834

2009

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Single-Cell Transcriptional Archetypes of Airway Inflammation in Cystic Fibrosis

American journal of respiratory and critical care medicine, 2020-11, Vol.202 (10), p.1419-1429

2020

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Human neutrophil development and functionality are enabled in a humanized mouse model

Proceedings of the National Academy of Sciences - PNAS, 2022-10, Vol.119 (43), p.e2121077119-e2121077119

2022

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Recruited monocytes/macrophages drive pulmonary neutrophilic inflammation and irreversible lung tissue remodeling in cystic fibrosis

Cell reports (Cambridge), 2022-12, Vol.41 (11), p.111797-111797, Article 111797

2022

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Assessment of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Activity in CFTR-Null Mice after Bone Marrow Transplantation

Proceedings of the National Academy of Sciences - PNAS, 2006-02, Vol.103 (8), p.2965-2970

2006

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Surfactant protein C dampens inflammation by decreasing JAK/STAT activation during lung repair

American journal of physiology. Lung cellular and molecular physiology, 2018-05, Vol.314 (5), p.L882-L892

2018

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Macrophages Directly Contribute to the Exaggerated Inflammatory Response in Cystic Fibrosis Transmembrane Conductance Regulator-/- Mice

American journal of respiratory cell and molecular biology, 2009-03, Vol.40 (3), p.295-304

2009

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Very Small Embryonic‐Like Stem Cells from the Murine Bone Marrow Differentiate into Epithelial Cells of the Lung

Stem cells (Dayton, Ohio), 2013-12, Vol.31 (12), p.2759-2766

2013

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Emerging Concepts in Defective Macrophage Phagocytosis in Cystic Fibrosis

Innate and Adaptive Immunity in Cystic Fibrosis

A highly efficient and faithful MDS patient-derived xenotransplantation model for pre-clinical studies

Pharmacological modulation of the AKT/microRNA-199a-5p/CAV1 pathway ameliorates cystic fibrosis lung hyper-inflammation

Combined liver-cytokine humanization comes to the rescue of circulating human red blood cells

Targeting the Heme Oxygenase 1/Carbon Monoxide Pathway to Resolve Lung Hyper-Inflammation and Restore a Regulated Immune Response in Cystic Fibrosis

Abnormal trafficking and degradation of TLR4 underlie the elevated inflammatory response in cystic fibrosis

Prior Influenza Infection Mitigates SARS-CoV-2 Disease in Syrian Hamsters

The carbon monoxide releasing molecule CORM-2 attenuates Pseudomonas aeruginosa biofilm formation

Ezrin links CFTR to TLR4 signaling to orchestrate anti-bacterial immune response in macrophages

Lack of a Fusion Requirement for Development of Bone Marrow-Derived Epithelia

SRF is required for neutrophil migration in response to inflammation

Recruitment of monocytes primed to express heme oxygenase-1 ameliorates pathological lung inflammation in cystic fibrosis

Cellular Innate Immunity: An Old Game with New Players

Cystic Fibrosis Lung Immunity: The Role of the Macrophage

Nonhematopoietic Cells are the Primary Source of Bone Marrow-Derived Lung Epithelial Cells

Update on Innate and Adaptive Immunity in Cystic Fibrosis

Role for MKL1 in megakaryocytic maturation

Single-Cell Transcriptional Archetypes of Airway Inflammation in Cystic Fibrosis

Human neutrophil development and functionality are enabled in a humanized mouse model

Recruited monocytes/macrophages drive pulmonary neutrophilic inflammation and irreversible lung tissue remodeling in cystic fibrosis

Assessment of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Activity in CFTR-Null Mice after Bone Marrow Transplantation

Surfactant protein C dampens inflammation by decreasing JAK/STAT activation during lung repair

Macrophages Directly Contribute to the Exaggerated Inflammatory Response in Cystic Fibrosis Transmembrane Conductance Regulator-/- Mice

Very Small Embryonic‐Like Stem Cells from the Murine Bone Marrow Differentiate into Epithelial Cells of the Lung

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