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Cleaning Up: ER-Associated Degradation to the Rescue

Cell, 2012-12, Vol.151 (6), p.1163-1167

Brodsky, Jeffrey L.

2012

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Protein quality control in the secretory pathway

The Journal of cell biology, 2019-10, Vol.218 (10), p.3171-3187

2019

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The evolving role of ubiquitin modification in endoplasmic reticulum-associated degradation

Biochemical journal, 2017-02, Vol.474 (4), p.445-469

2017

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The delicate balance between secreted protein folding and endoplasmic reticulum-associated degradation in human physiology

Physiological reviews, 2012-04, Vol.92 (2), p.537-576

2012

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Chaperoning Endoplasmic Reticulum-Associated Degradation (ERAD) and Protein Conformational Diseases

Cold Spring Harbor perspectives in biology, 2019-08, Vol.11 (8), p.a033928

2019

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Regulation of CFTR Biogenesis by the Proteostatic Network and Pharmacological Modulators

International journal of molecular sciences, 2020-01, Vol.21 (2), p.452

2020

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The degradation pathway of a model misfolded protein is determined by aggregation propensity

Molecular biology of the cell, 2018-06, Vol.29 (12), p.1422-1434

2018

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Recognition and Retrotranslocation of Misfolded Proteins from the Endoplasmic Reticulum

Traffic (Copenhagen, Denmark), 2008-06, Vol.9 (6), p.861-870

2008

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Adapting to stress - chaperome networks in cancer

Nature reviews. Cancer, 2018-09, Vol.18 (9), p.562-575

2018

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The generation of detergent-insoluble clipped fragments from an ERAD substrate in mammalian cells

Scientific reports, 2023-12, Vol.13 (1), p.21508-21508, Article 21508

2023

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From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

Molecular biology of the cell, 2016-02, Vol.27 (3), p.424-433

2016

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The protective and destructive roles played by molecular chaperones during ERAD (endoplasmic-reticulum-associated degradation)

Biochemical journal, 2007-06, Vol.404 (3), p.353-363

Brodsky, Jeffrey L

2007

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Targeting protein quality control pathways in breast cancer

BMC biology, 2017-11, Vol.15 (1), p.109-109, Article 109

2017

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The Targeting of Native Proteins to the Endoplasmic Reticulum-Associated Degradation (ERAD) Pathway: An Expanding Repertoire of Regulated Substrates

Biomolecules (Basel, Switzerland), 2021-08, Vol.11 (8), p.1185

2021

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Discovery and validation of a new class of small molecule Toll-like receptor 4 (TLR4) inhibitors

PloS one, 2013-06, Vol.8 (6), p.e65779-e65779

2013

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How early studies on secreted and membrane protein quality control gave rise to the ER associated degradation (ERAD) pathway: The early history of ERAD

Biochimica et biophysica acta, 2013-11, Vol.1833 (11), p.2447-2457

2013

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Dissecting the ER-Associated Degradation of a Misfolded Polytopic Membrane Protein

Cell, 2008-01, Vol.132 (1), p.101-112

2008

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Hsp70 Targets a Cytoplasmic Quality Control Substrate to the San1p Ubiquitin Ligase

The Journal of biological chemistry, 2013-06, Vol.288 (25), p.18506-18520

2013

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Substrate ubiquitination retains misfolded membrane proteins in the endoplasmic reticulum for degradation

Cell reports (Cambridge), 2021-09, Vol.36 (12), p.109717-109717, Article 109717

2021

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Predicting protein targets for drug-like compounds using transcriptomics

PLoS computational biology, 2018-12, Vol.14 (12), p.e1006651-e1006651

2018

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The HSP70 modulator MAL3-101 inhibits Merkel cell carcinoma

PloS one, 2014-04, Vol.9 (4), p.e92041

2014

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Can modulators of apolipoproteinB biogenesis serve as an alternate target for cholesterol-lowering drugs?

Biochimica et biophysica acta. Molecular and cell biology of lipids, 2018-07, Vol.1863 (7), p.762-771

2018

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Genome mining yields putative disease-associated ROMK variants with distinct defects

PLoS genetics, 2023-11, Vol.19 (11), p.e1011051-e1011051

2023

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Complementary computational and experimental evaluation of missense variants in the ROMK potassium channel

PLoS computational biology, 2020-04, Vol.16 (4), p.e1007749

2020

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Cleaning Up: ER-Associated Degradation to the Rescue

Protein quality control in the secretory pathway

The evolving role of ubiquitin modification in endoplasmic reticulum-associated degradation

The delicate balance between secreted protein folding and endoplasmic reticulum-associated degradation in human physiology

Chaperoning Endoplasmic Reticulum-Associated Degradation (ERAD) and Protein Conformational Diseases

Regulation of CFTR Biogenesis by the Proteostatic Network and Pharmacological Modulators

The degradation pathway of a model misfolded protein is determined by aggregation propensity

Recognition and Retrotranslocation of Misfolded Proteins from the Endoplasmic Reticulum

Adapting to stress - chaperome networks in cancer

The generation of detergent-insoluble clipped fragments from an ERAD substrate in mammalian cells

From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

The protective and destructive roles played by molecular chaperones during ERAD (endoplasmic-reticulum-associated degradation)

Targeting protein quality control pathways in breast cancer

The Targeting of Native Proteins to the Endoplasmic Reticulum-Associated Degradation (ERAD) Pathway: An Expanding Repertoire of Regulated Substrates

Discovery and validation of a new class of small molecule Toll-like receptor 4 (TLR4) inhibitors

How early studies on secreted and membrane protein quality control gave rise to the ER associated degradation (ERAD) pathway: The early history of ERAD

Dissecting the ER-Associated Degradation of a Misfolded Polytopic Membrane Protein

Hsp70 Targets a Cytoplasmic Quality Control Substrate to the San1p Ubiquitin Ligase

Substrate ubiquitination retains misfolded membrane proteins in the endoplasmic reticulum for degradation

Predicting protein targets for drug-like compounds using transcriptomics

The ER membrane protein complex interacts cotranslationally to enable biogenesis of multipass membrane proteins

The HSP70 modulator MAL3-101 inhibits Merkel cell carcinoma

Can modulators of apolipoproteinB biogenesis serve as an alternate target for cholesterol-lowering drugs?

Genome mining yields putative disease-associated ROMK variants with distinct defects

Complementary computational and experimental evaluation of missense variants in the ROMK potassium channel

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