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Histological parameters for the quantitative assessment of muscular dystrophy in the mdx-mouse

Neuromuscular disorders : NMD, 2004-10, Vol.14 (10), p.675-682

2004

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Introduction of Telemedicine in a Prehospital Emergency Care Setting: A Pilot Study

International journal of telemedicine and applications, 2023-03, Vol.2023, p.1171401-8

2023

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Structure and Function of Purified Monoclonal Antibody Dimers Induced by Different Stress Conditions

Pharmaceutical research, 2012-08, Vol.29 (8), p.2047-2059

2012

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Detailed Analytical Characterization of a Bispecific IgG1 CrossMab Antibody of the Knob-into-Hole Format Applying Various Stress Conditions Revealed Pronounced Stability

ACS omega, 2022-02, Vol.7 (4), p.3671-3679

2022

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Loss of skeletal muscle strength by ablation of the sarcoplasmic reticulum protein JP45

Proceedings of the National Academy of Sciences - PNAS, 2007-12, Vol.104 (50), p.20108-20113

2007

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Effect of calpain and proteasome inhibition on Ca²⁺-dependent proteolysis and muscle histopathology in the mdx mouse

The FASEB journal, 2008-12, Vol.22 (12), p.4190-4200

2008

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Utrophin is a calpain substrate in muscle cells

Muscle & nerve, 2006-06, Vol.33 (6), p.753-759

2006

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The Ets Transcription Factor GABP Is Required for Postsynaptic Differentiation In Vivo

The Journal of neuroscience, 2000-08, Vol.20 (16), p.5989-5996

2000

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Novel cell-penetrating α-keto-amide calpain inhibitors as potential treatment for muscular dystrophy

Bioorganic & medicinal chemistry letters, 2005-12, Vol.15 (23), p.5176-5181

2005

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Glucocorticoid-mediated regulation of utrophin levels in human muscle fibers

Neuromuscular disorders : NMD, 2002-10, Vol.12, p.S95-S104

2002

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Effect of calpain and proteasome inhibition on Ca 2+ ‐dependent proteolysis and muscle histopathology in the mdx mouse

The FASEB journal, 2008-12, Vol.22 (12), p.4190-4200

2008

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A newly identified chromosomal microdeletion and an N-box mutation of the AChRε gene cause a congenital myasthenic syndrome

Brain (London, England : 1878), 2002-05, Vol.125 (Pt 5), p.1005-1013

2002

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Transcriptional activation of the utrophin promoter B by a constitutively active Ets-transcription factor

Neuromuscular disorders : NMD, 2003-02, Vol.13 (2), p.143-150

2003

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A newly identified chromosomal microdeletion and an N-box mutation of the AChR[epsilon] gene cause a congenital myasthenic syndrome

Brain (London, England : 1878), 2002-05, Vol.125 (5), p.1005

2002

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Histological parameters for the quantitative assessment of muscular dystrophy in the mdx-mouse

Introduction of Telemedicine in a Prehospital Emergency Care Setting: A Pilot Study

Structure and Function of Purified Monoclonal Antibody Dimers Induced by Different Stress Conditions

Detailed Analytical Characterization of a Bispecific IgG1 CrossMab Antibody of the Knob-into-Hole Format Applying Various Stress Conditions Revealed Pronounced Stability

Loss of skeletal muscle strength by ablation of the sarcoplasmic reticulum protein JP45

Effect of calpain and proteasome inhibition on Ca²⁺-dependent proteolysis and muscle histopathology in the mdx mouse

Utrophin is a calpain substrate in muscle cells

The Ets Transcription Factor GABP Is Required for Postsynaptic Differentiation In Vivo

Novel cell-penetrating α-keto-amide calpain inhibitors as potential treatment for muscular dystrophy

Glucocorticoid-mediated regulation of utrophin levels in human muscle fibers

Effect of calpain and proteasome inhibition on Ca 2+ ‐dependent proteolysis and muscle histopathology in the mdx mouse

A newly identified chromosomal microdeletion and an N-box mutation of the AChRε gene cause a congenital myasthenic syndrome

Transcriptional activation of the utrophin promoter B by a constitutively active Ets-transcription factor

A newly identified chromosomal microdeletion and an N-box mutation of the AChR[epsilon] gene cause a congenital myasthenic syndrome

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