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MIV-802, a uridine nucleotide prodrug, is a more potent inhibitor of hepatitis C virus genotype 3 replication than sofosbuvir

Journal of hepatology, 2017, Vol.66 (1), p.S322-S323

2017

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318 THE HEPATITIS C VIRUS ENHANCES ACTIVATION OF THE EGF-RECEPTOR AND AKT-ACTIVITY BY NS3/4A-MEDIATED CLEAVAGE OF THE T-CELL PROTEIN TYROSINE PHOSPHATASE

Journal of hepatology, 2009, Vol.50, p.S124-S124

2009

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Long‐term follow‐up of successful hepatitis C virus therapy: waning immune responses and disappearance of liver disease are consistent with cure

Alimentary pharmacology & therapeutics, 2015-03, Vol.41 (6), p.532-543

2015

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The hepatitis C virus and immune evasion: non-structural 3/4A transgenic mice are resistant to lethal tumour necrosis factor α mediated liver disease

Gut, 2006-10, Vol.55 (10), p.1475-1483

2006

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THU-302 - MIV-802, a uridine nucleotide prodrug, is a more potent inhibitor of hepatitis C virus genotype 3 replication than sofosbuvir

Journal of hepatology, 2017, Vol.66 (1), p.S322-S323

2017

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The role of chemokines in hepatitis C virus-mediated liver disease

International journal of molecular sciences, 2014-03, Vol.15 (3), p.4747-4779

2014

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Biodistribution of fluorescence-labelled EGF protein from slow release NanoZolid depots in mouse

International journal of pharmaceutics, 2021-05, Vol.601, p.120588-120588, Article 120588

2021

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Long-term follow-up after cure from chronic hepatitis C virus infection shows occult hepatitis and a risk of hepatocellular carcinoma in noncirrhotic patients

European journal of gastroenterology & hepatology, 2019-04, Vol.31 (4), p.506-513

2019

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Lack of Association Between Interleukin 28B Polymorphism and Vertical Transmission of Hepatitis C

Journal of pediatric gastroenterology and nutrition, 2017-12, Vol.65 (6), p.608-612

2017

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TCR-redirected human T cells inhibit hepatitis C virus replication: hepatotoxic potential is linked to antigen specificity and functional avidity

The Journal of immunology (1950), 2012-11, Vol.189 (9), p.4510-4519

2012

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Hepatitis C Virus Nonstructural 3/4A Protein Dampens Inflammation and Contributes to Slow Fibrosis Progression during Chronic Fibrosis In Vivo

PloS one, 2015-06, Vol.10 (6), p.e0128466-e0128466

2015

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The hepatitis C virus and immune evasion: non-structural 3/4A transgenic mice are resistant to lethal tumour necrosis factor mediated liver disease

Gut, 2006-10, Vol.55 (10), p.1475-1483

Frelin, L

2006

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Neonatal Exposure to Hepatitis C Virus Antigens in Uninfected Children Born to Infected Mothers

Journal of pediatric gastroenterology and nutrition, 2018-01, Vol.66 (1), p.106-111

2018

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Die Aktivierung der MKK6/p38MAPK-Kaskade ist essentiell für die Stabilisierung der Suppressor of Cytokine Signaling 3 mRNA durch TNF-alpha in Makrophagen

Zeitschrift für Gastroenterologie, 2006, Vol.44 (8)

2006

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Hepatitis C virus non-structural 3/4A protein interferes with intrahepatic interferon-γ production

Gut, 2012-04, Vol.61 (4), p.589-596

2012

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Hepatitis C virus (HCV) employs multiple strategies to subvert the host innate antiviral response

Biological chemistry, 2008-10, Vol.389 (10), p.1283-1298

2008

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Heterologous T cells can help restore function in dysfunctional hepatitis C virus nonstructural 3/4A-specific T cells during therapeutic vaccination

The Journal of immunology (1950), 2011-05, Vol.186 (9), p.5107-5118

2011

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Cleavage of the T cell protein tyrosine phosphatase by the hepatitis C virus nonstructural 3/4A protease induces a Th1 to Th2 shift reversible by ribavirin therapy

The Journal of immunology (1950), 2014-02, Vol.192 (4), p.1671-1680

2014

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Interplay between host cell and hepatitis C virus in regulating viral replication

Biological chemistry, 2009-10, Vol.390 (10), p.1013-1032

2009

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Nonstructural 3/4A protease of hepatitis C virus activates epithelial growth factor–induced signal transduction by cleavage of the T‐cell protein tyrosine phosphatase

Hepatology (Baltimore, Md.), 2009-06, Vol.49 (6), p.1810-1820

2009

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Hepatitis C Virus-Mediated Modulation of Cellular Immunity

Archivum Immunologiae et Therapiae Experimentalis, 2012-10, Vol.60 (5), p.315-329

2012

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Modifikation TNF-alpha induzierter Signalvermittlung durch das NS3/4A-Protein des Hepatitis C Virus in NS3/4A transgenen Mäusen

Zeitschrift für Gastroenterologie, 2006, Vol.44 (1)

2006

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Die Aktivierung der MKK6/p38 Kaskade ist essentiell für die Stabilisierung der Suppressor of Cytokine Signaling 3 mRNA durch TNF-alpha

Zeitschrift für Gastroenterologie, 2006, Vol.44 (1)

2006

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Anti–tumor necrosis factor α treatment promotes apoptosis and prevents liver regeneration in a transgenic mouse model of chronic hepatitis C

Hepatology (Baltimore, Md.), 2010-11, Vol.52 (5), p.1553-1563

2010

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Containing “The Great Houdini” of viruses: Combining direct acting antivirals with the host immune response for the treatment of chronic hepatitis C

Drug resistance updates, 2013-07, Vol.16 (3), p.60-67

2013

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MIV-802, a uridine nucleotide prodrug, is a more potent inhibitor of hepatitis C virus genotype 3 replication than sofosbuvir

318 THE HEPATITIS C VIRUS ENHANCES ACTIVATION OF THE EGF-RECEPTOR AND AKT-ACTIVITY BY NS3/4A-MEDIATED CLEAVAGE OF THE T-CELL PROTEIN TYROSINE PHOSPHATASE

Long‐term follow‐up of successful hepatitis C virus therapy: waning immune responses and disappearance of liver disease are consistent with cure

The hepatitis C virus and immune evasion: non-structural 3/4A transgenic mice are resistant to lethal tumour necrosis factor α mediated liver disease

THU-302 - MIV-802, a uridine nucleotide prodrug, is a more potent inhibitor of hepatitis C virus genotype 3 replication than sofosbuvir

The role of chemokines in hepatitis C virus-mediated liver disease

Biodistribution of fluorescence-labelled EGF protein from slow release NanoZolid depots in mouse

Long-term follow-up after cure from chronic hepatitis C virus infection shows occult hepatitis and a risk of hepatocellular carcinoma in noncirrhotic patients

Lack of Association Between Interleukin 28B Polymorphism and Vertical Transmission of Hepatitis C

TCR-redirected human T cells inhibit hepatitis C virus replication: hepatotoxic potential is linked to antigen specificity and functional avidity

Hepatitis C Virus Nonstructural 3/4A Protein Dampens Inflammation and Contributes to Slow Fibrosis Progression during Chronic Fibrosis In Vivo

The hepatitis C virus and immune evasion: non-structural 3/4A transgenic mice are resistant to lethal tumour necrosis factor mediated liver disease

Neonatal Exposure to Hepatitis C Virus Antigens in Uninfected Children Born to Infected Mothers

Die Aktivierung der MKK6/p38MAPK-Kaskade ist essentiell für die Stabilisierung der Suppressor of Cytokine Signaling 3 mRNA durch TNF-alpha in Makrophagen

Hepatitis C virus non-structural 3/4A protein interferes with intrahepatic interferon-γ production

Hepatitis C virus (HCV) employs multiple strategies to subvert the host innate antiviral response

Heterologous T cells can help restore function in dysfunctional hepatitis C virus nonstructural 3/4A-specific T cells during therapeutic vaccination

Cleavage of the T cell protein tyrosine phosphatase by the hepatitis C virus nonstructural 3/4A protease induces a Th1 to Th2 shift reversible by ribavirin therapy

Interplay between host cell and hepatitis C virus in regulating viral replication

Nonstructural 3/4A protease of hepatitis C virus activates epithelial growth factor–induced signal transduction by cleavage of the T‐cell protein tyrosine phosphatase

Hepatitis C Virus-Mediated Modulation of Cellular Immunity

Modifikation TNF-alpha induzierter Signalvermittlung durch das NS3/4A-Protein des Hepatitis C Virus in NS3/4A transgenen Mäusen

Die Aktivierung der MKK6/p38 Kaskade ist essentiell für die Stabilisierung der Suppressor of Cytokine Signaling 3 mRNA durch TNF-alpha

Anti–tumor necrosis factor α treatment promotes apoptosis and prevents liver regeneration in a transgenic mouse model of chronic hepatitis C

Containing “The Great Houdini” of viruses: Combining direct acting antivirals with the host immune response for the treatment of chronic hepatitis C

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