UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Propofol causes vasodilation in vivo via TRPA1 ion channels: role of nitric oxide and BKCa channels

PloS one, 2015-04, Vol.10 (4), p.e0122189-e0122189

2015

Volltextzugriff (PDF)

TRPA1 ion channel stimulation enhances cardiomyocyte contractile function via a CaMKII-dependent pathway

Channels (Austin, Tex.), 2017-11, Vol.11 (6), p.587-603

2017

Volltextzugriff (PDF)

TRPA1 and TRPV1 contribute to propofol-mediated antagonism of U46619-induced constriction in murine coronary arteries

PloS one, 2017-06, Vol.12 (6), p.e0180106-e0180106

2017

Volltextzugriff (PDF)

Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunction

Basic research in cardiology, 2016-03, Vol.111 (2), p.21-21, Article 21

2016

Volltextzugriff (PDF)

Impaired capsaicin-induced relaxation of coronary arteries in a porcine model of the metabolic syndrome

American journal of physiology. Heart and circulatory physiology, 2008-06, Vol.294 (6), p.H2489-H2496

2008

Volltextzugriff (PDF)

H2O2-induced redox-sensitive coronary vasodilation is mediated by 4-aminopyridine-sensitive K+ channels

American journal of physiology. Heart and circulatory physiology, 2006-11, Vol.291 (5), p.H2473-H2482

2006

Volltextzugriff (PDF)

Endothelin-mediated in vivo pressor responses following TRPV1 activation

American journal of physiology. Heart and circulatory physiology, 2011-09, Vol.301 (3), p.H1135-H1142

2011

Volltextzugriff (PDF)

TRPA1 is functionally co-expressed with TRPV1 in cardiac muscle: Co-localization at z-discs, costameres and intercalated discs

Channels (Austin, Tex.), 2016-09, Vol.10 (5), p.395-409

2016

Volltextzugriff (PDF)

Mechanisms of Coronary Dysfunction in Obesity and Insulin Resistance

Microcirculation (New York, N.Y. 1994), 2007-06, Vol.14 (4-5), p.317-338

2007

Volltextzugriff (PDF)

PKCα mediates acetylcholine-induced activation of TRPV4-dependent calcium influx in endothelial cells

American journal of physiology. Heart and circulatory physiology, 2011-09, Vol.301 (3), p.H757-H765

2011

Volltextzugriff (PDF)

4-Hydroxynonenal dependent alteration of TRPV1-mediated coronary microvascular signaling

Free radical biology & medicine, 2016-12, Vol.101, p.10-19

2016

Volltextzugriff (PDF)

Exercise training decreases store-operated Ca2+entry associated with metabolic syndrome and coronary atherosclerosis

Cardiovascular research, 2010-02, Vol.85 (3), p.631-640

2010

Volltextzugriff (PDF)

Disruption of TRPV1-mediated coupling of coronary blood flow to cardiac metabolism in diabetic mice: role of nitric oxide and BK channels

American journal of physiology. Heart and circulatory physiology, 2012-07, Vol.303 (2), p.H216-H223

2012

Volltextzugriff (PDF)

Impaired function of coronary BKCa channels in metabolic syndrome

American journal of physiology. Heart and circulatory physiology, 2009-11, Vol.297 (5), p.H1629-H1637

2009

Volltextzugriff (PDF)

H2O2 activates redox- and 4-aminopyridine-sensitive Kv channels in coronary vascular smooth muscle

American journal of physiology. Heart and circulatory physiology, 2007-03, Vol.292 (3), p.H1404-H1411

2007

Volltextzugriff (PDF)

Short-term exercise training prevents micro- and macrovascular disease following coronary stenting

Journal of applied physiology (1985), 2010-06, Vol.108 (6), p.1766-1774

2010

Volltextzugriff (PDF)

Propofol restores transient receptor potential vanilloid receptor subtype-1 sensitivity via activation of transient receptor potential ankyrin receptor subtype-1 in sensory neurons

Anesthesiology (Philadelphia), 2011-05, Vol.114 (5), p.1169-1179

2011

Volltextzugriff (PDF)

Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

American journal of physiology. Heart and circulatory physiology, 2010-04, Vol.298 (4), p.H1182-H1189

2010

Volltextzugriff (PDF)

Penitrem A as a tool for understanding the role of large conductance Ca(2+)/voltage-sensitive K(+) channels in vascular function

The Journal of pharmacology and experimental therapeutics, 2012-08, Vol.342 (2), p.453

2012

Volltextzugriff (PDF)

Voltage-dependent K+ channels regulate the duration of reactive hyperemia in the canine coronary circulation

American journal of physiology. Heart and circulatory physiology, 2008-05, Vol.294 (5), p.H2371-H2381

2008

Volltextzugriff (PDF)

Propofol restores TRPV1 sensitivity via a TRPA1‐, nitric oxide synthase‐dependent activation of PKCε

Pharmacology research & perspectives, 2015-08, Vol.3 (4), p.e00153-n/a

2015

Volltextzugriff (PDF)

Endogenous Adipose-Derived Factors Diminish Coronary Endothelial Function via Inhibition of Nitric Oxide Synthase

Microcirculation (New York, N.Y. 1994), 2008-07, Vol.15 (5), p.417-426

2008

Volltextzugriff (PDF)

Reduced molecular expression of K+ channel proteins in vascular smooth muscle from rats made hypertensive with N{omega}-nitro-L-arginine

American journal of physiology. Heart and circulatory physiology, 2005-09, Vol.289 (3), p.H1277

2005

Volltextzugriff (PDF)

C-reactive protein does not relax vascular smooth muscle: effects mediated by sodium azide in commercially available preparations

American journal of physiology. Heart and circulatory physiology, 2005-04, Vol.288 (4), p.H1786-H1795

2005

Volltextzugriff (PDF)

Nitric oxide synthase-inhibition hypertension is associated with altered endothelial cyclooxygenase function

American journal of physiology. Heart and circulatory physiology, 2004-12, Vol.287 (6), p.H2394-H2401

2004

Volltextzugriff (PDF)

1 – 25 von 83

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(83)
Mit Volltextzugriff
(81)

Publikationsform

Artikel
(83)

Sprache

Englisch
(83)

Erscheinungsjahr

n.n

Menü

Weitere Dienste

Einstellungen

Propofol causes vasodilation in vivo via TRPA1 ion channels: role of nitric oxide and BKCa channels

TRPA1 ion channel stimulation enhances cardiomyocyte contractile function via a CaMKII-dependent pathway

TRPA1 and TRPV1 contribute to propofol-mediated antagonism of U46619-induced constriction in murine coronary arteries

Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunction

Impaired capsaicin-induced relaxation of coronary arteries in a porcine model of the metabolic syndrome

H2O2-induced redox-sensitive coronary vasodilation is mediated by 4-aminopyridine-sensitive K+ channels

Endothelin-mediated in vivo pressor responses following TRPV1 activation

TRPA1 is functionally co-expressed with TRPV1 in cardiac muscle: Co-localization at z-discs, costameres and intercalated discs

Mechanisms of Coronary Dysfunction in Obesity and Insulin Resistance

PKCα mediates acetylcholine-induced activation of TRPV4-dependent calcium influx in endothelial cells

4-Hydroxynonenal dependent alteration of TRPV1-mediated coronary microvascular signaling

Exercise training decreases store-operated Ca2+entry associated with metabolic syndrome and coronary atherosclerosis

Disruption of TRPV1-mediated coupling of coronary blood flow to cardiac metabolism in diabetic mice: role of nitric oxide and BK channels

Impaired function of coronary BKCa channels in metabolic syndrome

H2O2 activates redox- and 4-aminopyridine-sensitive Kv channels in coronary vascular smooth muscle

Short-term exercise training prevents micro- and macrovascular disease following coronary stenting

Propofol restores transient receptor potential vanilloid receptor subtype-1 sensitivity via activation of transient receptor potential ankyrin receptor subtype-1 in sensory neurons

Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

Penitrem A as a tool for understanding the role of large conductance Ca(2+)/voltage-sensitive K(+) channels in vascular function

Voltage-dependent K+ channels regulate the duration of reactive hyperemia in the canine coronary circulation

Propofol restores TRPV1 sensitivity via a TRPA1‐, nitric oxide synthase‐dependent activation of PKCε

Endogenous Adipose-Derived Factors Diminish Coronary Endothelial Function via Inhibition of Nitric Oxide Synthase

Reduced molecular expression of K+ channel proteins in vascular smooth muscle from rats made hypertensive with N{omega}-nitro-L-arginine

C-reactive protein does not relax vascular smooth muscle: effects mediated by sodium azide in commercially available preparations

Nitric oxide synthase-inhibition hypertension is associated with altered endothelial cyclooxygenase function

Suchergebnisse filtern