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Early IFNγ-Mediated and Late Perforin-Mediated Suppression of Pathogenic CD4 T Cell Responses Are Both Required for Inhibition of Demyelinating Disease by CNS-Specific Autoregulatory CD8 T Cells

Frontiers in immunology, 2018-10, Vol.9, p.2336

2018

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IL-12-Induced Immune Suppressive Deficit During CD8+ T-Cell Differentiation

Frontiers in immunology, 2020-10, Vol.11, p.568630-568630

2020

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Suppression of autoimmune demyelinating disease by preferential stimulation of CNS-specific CD8 T cells using Listeria-encoded neuroantigen

Scientific reports, 2017-05, Vol.7 (1), p.1519-11, Article 1519

2017

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Therapeutic intervention in relapsing autoimmune demyelinating disease through induction of myelin-specific regulatory CD8 T cell responses

Journal of translational autoimmunity (Online), 2019-12, Vol.2, p.100010, Article 100010

2019

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Novel B cell-dependent multiple sclerosis model using extracellular domains of myelin proteolipid protein

Scientific reports, 2020-03, Vol.10 (1), p.5011-5011, Article 5011

2020

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Immune Autoregulatory CD8 T Cells Require IFN-γ Responsiveness to Optimally Suppress Central Nervous System Autoimmunity

The Journal of immunology (1950), 2020-07, Vol.205 (2), p.359-368

2020

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A Functionally Distinct CXCR3 + /IFN-γ + /IL-10 + Subset Defines Disease-Suppressive Myelin-Specific CD8 T Cells

The Journal of immunology (1950), 2021-03, Vol.206 (6), p.1151-1160

2021

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Neuroantigen-specific CD8 T cells inhibit ongoing demyelinating disease by autoregulating CD4 T cell responses using temporally distinct IFNγ- and perforin-dependent mechanisms

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.163-163.2

2018

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Myelin-specific CD8 T cells require IFNγ responsiveness to optimally suppress CNS autoimmunity

The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.238-238.6

2020

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Novel B cell-dependent multiple sclerosis model using extracellular domains of myelin proteolipid protein

The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.218-218.18

2020

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A functionally distinct CXCR3+/IFNγ+/IL-10+ subset defines disease-suppressive CNS-specific CD8 T cells

The Journal of immunology (1950), 2021-02, Vol.206 (6), p.1151-1160

2021

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Immune autoregulatory CD8 T cells require IFNγ responsiveness to optimally suppress CNS autoimmunity

The Journal of immunology (1950), 2020-06, Vol.205 (2), p.359-368

2020

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CNS-specific autoregulatory CD8 T cells rely on IFNγ signaling for optimal suppression of pathogenic CD4 T cell responses during inhibition of demyelinating disease

The Journal of immunology (1950), 2019-05, Vol.202 (1_Supplement), p.115-115.9

2019

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Neuroantigen-specific regulatory CD8+ T cell responses as a therapy for relapsing autoimmune demyelinating disease

The Journal of immunology (1950), 2019-05, Vol.202 (1_Supplement), p.193-193.11

2019

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Induction of neuroantigen-specific CD8+ T cell responses as a treatment for relapsing autoimmune demyelinating disease

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.121-121.1

2018

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The Role of Myelin-Specific Regulatory CD8 T Cells in Relapsing-Remitting Demyelinating Disease

Brate, Ashley A

2019

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Altered expression of SIRPγ on the T-cells of relapsing remitting multiple sclerosis and type 1 diabetes patients could potentiate effector responses from T-cells

PloS one, 2020-08, Vol.15 (8), p.e0238070-e0238070

2020

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Novel role for T-helper 17 signature cytokine, IL-17, in inducing CD4 effector resistance to immune suppression

The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.228-228.3

2020

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Altered expression of SIRP[gamma] on the T-cells of relapsing remitting multiple sclerosis and type 1 diabetes patients could potentiate effector responses from T-cells

PloS one, 2020-08, Vol.15 (8), p.e0238070

2020

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321 Single mRNA constructs encoding multiple linked antigens allow for a multiantigen-specific CD8+ T cell response driven by SQZ® eAPCs

Journal for immunotherapy of cancer, 2022-11, Vol.10 (Suppl 2), p.A337-A337

2022

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Listeria monocytogenes -encoded expression of a neuroantigen induces CD8 T-cell-mediated inhibition of autoimmune demyelinating disease

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.186-186.29

2016

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Effects of Disease Causing Point Mutations on Actin's Resilience

Biophysical journal, 2013-01, Vol.104 (2), p.648a-648a

2013

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Large Scale Opening of the N Terminal Actin-Binding Domain of Dystrophin Detected by Dipolar Electron-Electron Resonance (DEER)

Biophysical journal, 2013-01, Vol.104 (2), p.648a-648a

2013

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Early IFNγ-Mediated and Late Perforin-Mediated Suppression of Pathogenic CD4 T Cell Responses Are Both Required for Inhibition of Demyelinating Disease by CNS-Specific Autoregulatory CD8 T Cells

IL-12-Induced Immune Suppressive Deficit During CD8+ T-Cell Differentiation

Suppression of autoimmune demyelinating disease by preferential stimulation of CNS-specific CD8 T cells using Listeria-encoded neuroantigen

Therapeutic intervention in relapsing autoimmune demyelinating disease through induction of myelin-specific regulatory CD8 T cell responses

Novel B cell-dependent multiple sclerosis model using extracellular domains of myelin proteolipid protein

Immune Autoregulatory CD8 T Cells Require IFN-γ Responsiveness to Optimally Suppress Central Nervous System Autoimmunity

A Functionally Distinct CXCR3 + /IFN-γ + /IL-10 + Subset Defines Disease-Suppressive Myelin-Specific CD8 T Cells

Neuroantigen-specific CD8 T cells inhibit ongoing demyelinating disease by autoregulating CD4 T cell responses using temporally distinct IFNγ- and perforin-dependent mechanisms

Myelin-specific CD8 T cells require IFNγ responsiveness to optimally suppress CNS autoimmunity

Novel B cell-dependent multiple sclerosis model using extracellular domains of myelin proteolipid protein

A functionally distinct CXCR3+/IFNγ+/IL-10+ subset defines disease-suppressive CNS-specific CD8 T cells

Immune autoregulatory CD8 T cells require IFNγ responsiveness to optimally suppress CNS autoimmunity

CNS-specific autoregulatory CD8 T cells rely on IFNγ signaling for optimal suppression of pathogenic CD4 T cell responses during inhibition of demyelinating disease

Neuroantigen-specific regulatory CD8+ T cell responses as a therapy for relapsing autoimmune demyelinating disease

Induction of neuroantigen-specific CD8+ T cell responses as a treatment for relapsing autoimmune demyelinating disease

The Role of Myelin-Specific Regulatory CD8 T Cells in Relapsing-Remitting Demyelinating Disease

Altered expression of SIRPγ on the T-cells of relapsing remitting multiple sclerosis and type 1 diabetes patients could potentiate effector responses from T-cells

Novel role for T-helper 17 signature cytokine, IL-17, in inducing CD4 effector resistance to immune suppression

Altered expression of SIRP[gamma] on the T-cells of relapsing remitting multiple sclerosis and type 1 diabetes patients could potentiate effector responses from T-cells

321 Single mRNA constructs encoding multiple linked antigens allow for a multiantigen-specific CD8+ T cell response driven by SQZ® eAPCs

Listeria monocytogenes -encoded expression of a neuroantigen induces CD8 T-cell-mediated inhibition of autoimmune demyelinating disease

Effects of Disease Causing Point Mutations on Actin's Resilience

Large Scale Opening of the N Terminal Actin-Binding Domain of Dystrophin Detected by Dipolar Electron-Electron Resonance (DEER)

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