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IFITM3 restricts influenza A virus entry by blocking the formation of fusion pores following virus-endosome hemifusion

PLoS pathogens, 2014-04, Vol.10 (4), p.e1004048-e1004048

2014

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The IFITMs Inhibit Zika Virus Replication

Cell reports (Cambridge), 2016-06, Vol.15 (11), p.2323-2330

2016

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Identification of Zika Virus and Dengue Virus Dependency Factors using Functional Genomics

Cell reports (Cambridge), 2016-06, Vol.16 (1), p.232-246

2016

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Frizzled proteins are colonic epithelial receptors for C. difficile toxin B

Nature (London), 2016-10, Vol.538 (7625), p.350-355

2016

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IFITMs Restrict the Replication of Multiple Pathogenic Viruses

Journal of molecular biology, 2013-12, Vol.425 (24), p.4937-4955

2013

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Frequent Loss of IRF2 in Cancers Leads to Immune Evasion through Decreased MHC Class I Antigen Presentation and Increased PD-L1 Expression

The Journal of immunology (1950), 2019-10, Vol.203 (7), p.1999-2010

2019

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Identification of Host Proteins Required for HIV Infection Through a Functional Genomic Screen

Science (American Association for the Advancement of Science), 2008-02, Vol.319 (5865), p.921-926

2008

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The IFITM Proteins Mediate Cellular Resistance to Influenza A H1N1 Virus, West Nile Virus, and Dengue Virus

Cell, 2009-12, Vol.139 (7), p.1243-1254

2009

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Direct Visualization of HIV-1 Replication Intermediates Shows that Capsid and CPSF6 Modulate HIV-1 Intra-nuclear Invasion and Integration

Cell reports (Cambridge), 2015-11, Vol.13 (8), p.1717-1731

2015

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Reactivation of Latent HIV-1 by Inhibition of BRD4

Cell reports (Cambridge), 2012-10, Vol.2 (4), p.807-816

2012

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Interferon-induced transmembrane protein 3 blocks fusion of sensitive but not resistant viruses by partitioning into virus-carrying endosomes

PLoS pathogens, 2019-01, Vol.15 (1), p.e1007532-e1007532

2019

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IFITM3 inhibits influenza A virus infection by preventing cytosolic entry

PLoS pathogens, 2011-10, Vol.7 (10), p.e1002337

2011

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genome-wide genetic screen for host factors required for hepatitis C virus propagation

Proceedings of the National Academy of Sciences - PNAS, 2009-09, Vol.106 (38), p.16410-16415

2009

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Distinct patterns of IFITM-mediated restriction of filoviruses, SARS coronavirus, and influenza A virus

PLoS pathogens, 2011-01, Vol.7 (1), p.e1001258-e1001258

2011

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The CD225 Domain of IFITM3 Is Required for both IFITM Protein Association and Inhibition of Influenza A Virus and Dengue Virus Replication

Journal of Virology, 2013-07, Vol.87 (14), p.7837-7852

2013

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Dysfunctional HIV-Specific CD8+ T Cell Proliferation Is Associated with Increased Caspase-8 Activity and Mediated by Necroptosis

Immunity (Cambridge, Mass.), 2014-12, Vol.41 (6), p.1001-1012

2014

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Amphotericin B Increases Influenza A Virus Infection by Preventing IFITM3-Mediated Restriction

Cell reports (Cambridge), 2013-11, Vol.5 (4), p.895-908

2013

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ADAM9 promotes type I interferon-mediated innate immunity during encephalomyocarditis virus infection

Nature communications, 2024-05, Vol.15 (1), p.4153-14, Article 4153

2024

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RNASEK Is a V-ATPase-Associated Factor Required for Endocytosis and the Replication of Rhinovirus, Influenza A Virus, and Dengue Virus

Cell reports (Cambridge), 2015-08, Vol.12 (5), p.850-863

2015

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TNPO3 Is Required for HIV-1 Replication after Nuclear Import but prior to Integration and Binds the HIV-1 Core

Journal of Virology, 2012-05, Vol.86 (10), p.5931-5936

2012

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Exploiting glycan topography for computational design of Env glycoprotein antigenicity

PLoS computational biology, 2018-04, Vol.14 (4), p.e1006093-e1006093

2018

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Comprehensive Identification of Host Modulators of HIV-1 Replication using Multiple Orthologous RNAi Reagents

Cell reports (Cambridge), 2014-10, Vol.9 (2), p.752-766

2014

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A Disintegrin and Metalloproteinase 9 Domain (ADAM9) Is a Major Susceptibility Factor in the Early Stages of Encephalomyocarditis Virus Infection

mBio, 2019-02, Vol.10 (1)

2019

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Inhibition of HIV-1 Integration in Ex Vivo-Infected CD4 T Cells from Elite Controllers

Journal of Virology, 2011-09, Vol.85 (18), p.9646-9650

2011

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Association of TRIM22 with the Type 1 Interferon Response and Viral Control during Primary HIV-1 Infection

Journal of Virology, 2011-01, Vol.85 (1), p.208-216

2011

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IFITM3 restricts influenza A virus entry by blocking the formation of fusion pores following virus-endosome hemifusion

The IFITMs Inhibit Zika Virus Replication

Identification of Zika Virus and Dengue Virus Dependency Factors using Functional Genomics

Frizzled proteins are colonic epithelial receptors for C. difficile toxin B

IFITMs Restrict the Replication of Multiple Pathogenic Viruses

Frequent Loss of IRF2 in Cancers Leads to Immune Evasion through Decreased MHC Class I Antigen Presentation and Increased PD-L1 Expression

Identification of Host Proteins Required for HIV Infection Through a Functional Genomic Screen

The IFITM Proteins Mediate Cellular Resistance to Influenza A H1N1 Virus, West Nile Virus, and Dengue Virus

Direct Visualization of HIV-1 Replication Intermediates Shows that Capsid and CPSF6 Modulate HIV-1 Intra-nuclear Invasion and Integration

Reactivation of Latent HIV-1 by Inhibition of BRD4

Interferon-induced transmembrane protein 3 blocks fusion of sensitive but not resistant viruses by partitioning into virus-carrying endosomes

IFITM3 inhibits influenza A virus infection by preventing cytosolic entry

genome-wide genetic screen for host factors required for hepatitis C virus propagation

Distinct patterns of IFITM-mediated restriction of filoviruses, SARS coronavirus, and influenza A virus

The CD225 Domain of IFITM3 Is Required for both IFITM Protein Association and Inhibition of Influenza A Virus and Dengue Virus Replication

Dysfunctional HIV-Specific CD8+ T Cell Proliferation Is Associated with Increased Caspase-8 Activity and Mediated by Necroptosis

Amphotericin B Increases Influenza A Virus Infection by Preventing IFITM3-Mediated Restriction

ADAM9 promotes type I interferon-mediated innate immunity during encephalomyocarditis virus infection

RNASEK Is a V-ATPase-Associated Factor Required for Endocytosis and the Replication of Rhinovirus, Influenza A Virus, and Dengue Virus

TNPO3 Is Required for HIV-1 Replication after Nuclear Import but prior to Integration and Binds the HIV-1 Core

Exploiting glycan topography for computational design of Env glycoprotein antigenicity

Comprehensive Identification of Host Modulators of HIV-1 Replication using Multiple Orthologous RNAi Reagents

A Disintegrin and Metalloproteinase 9 Domain (ADAM9) Is a Major Susceptibility Factor in the Early Stages of Encephalomyocarditis Virus Infection

Inhibition of HIV-1 Integration in Ex Vivo-Infected CD4 T Cells from Elite Controllers

Association of TRIM22 with the Type 1 Interferon Response and Viral Control during Primary HIV-1 Infection

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