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The Role of ClpB in Bacterial Stress Responses and Virulence

Frontiers in molecular biosciences, 2021-04, Vol.8, p.668910-668910

2021

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Francisella tularensis IglG Belongs to a Novel Family of PAAR-Like T6SS Proteins and Harbors a Unique N-terminal Extension Required for Virulence

PLoS pathogens, 2016-09, Vol.12 (9), p.e1005821-e1005821

2016

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Exploring the Diversity Within the Genus Francisella – An Integrated Pan-Genome and Genome-Mining Approach

Frontiers in microbiology, 2020-08, Vol.11, p.1928-1928

2020

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DotU and VgrG, core components of type VI secretion systems, are essential for Francisella LVS pathogenicity

PloS one, 2012-04, Vol.7 (4), p.e34639

2012

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Unique substrates secreted by the type VI secretion system of Francisella tularensis during intramacrophage infection

PloS one, 2012-11, Vol.7 (11), p.e50473

2012

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A Conserved α-Helix Essential for a Type VI Secretion-Like System of Francisella tularensis

Journal of Bacteriology, 2009-04, Vol.191 (8), p.2431-2446

2009

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The type IV pili component PilO is a virulence determinant of Francisella novicida

PloS one, 2022-01, Vol.17 (1), p.e0261938-e0261938

2022

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The Role of the Francisella Tularensis Pathogenicity Island in Type VI Secretion, Intracellular Survival, and Modulation of Host Cell Signaling

Frontiers in microbiology, 2010-01, Vol.1, p.136-136

2010

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Importance of PdpC, IglC, IglI, and IglG for Modulation of a Host Cell Death Pathway Induced by Francisella tularensis

Infection and Immunity, 2013-06, Vol.81 (6), p.2076-2084

2013

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IglG and IglI of the Francisella Pathogenicity Island Are Important Virulence Determinants of Francisella tularensis LVS

Infection and Immunity, 2011-09, Vol.79 (9), p.3683-3696

2011

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A functional VipA-VipB interaction is required for the type VI secretion system activity of Vibrio cholerae O1 strain A1552

BMC microbiology, 2013-05, Vol.13 (1), p.96-96

2013

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Microinjection of Francisella tularensis and Listeria monocytogenes reveals the importance of bacterial and host factors for successful replication

Infection and immunity, 2015-08, Vol.83 (8), p.3233-3242

2015

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A mutagenesis-based approach identifies amino acids in the N-terminal part of Francisella tularensis IglE that critically control Type VI system-mediated secretion

Virulence, 2017-08, Vol.8 (6), p.821-847

2017

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Twin Arginine Translocation System Is Essential for Virulence of Yersinia pseudotuberculosis

Infection and Immunity, 2006-03, Vol.74 (3), p.1768-1776

2006

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The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infection

BMC microbiology, 2013-01, Vol.13 (1), p.20-20

2013

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Impact of the N-Terminal Secretor Domain on YopD Translocator Function in Yersinia pseudotuberculosis Type III Secretion

Journal of Bacteriology, 2011-12, Vol.193 (23), p.6683-6700

2011

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Diminished LcrV Secretion Attenuates Yersinia pseudotuberculosis Virulence

Journal of Bacteriology, 2007-12, Vol.189 (23), p.8417-8429

2007

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The Amino-Terminal Part of the Needle-Tip Translocator LcrV of Yersinia pseudotuberculosis Is Required for Early Targeting of YopH and In vivo Virulence

Frontiers in cellular and infection microbiology, 2016, Vol.6, p.175-175

2016

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The YopD Translocator of Yersinia pseudotuberculosis Is a Multifunctional Protein Comprised of Discrete Domains

Journal of Bacteriology, 2004-07, Vol.186 (13), p.4110-4123

2004

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Mapping of a YscY Binding Domain within the LcrH Chaperone That Is Required for Regulation of Yersinia Type III Secretion

Journal of Bacteriology, 2005-11, Vol.187 (22), p.7738-7752

2005

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PcrH of Pseudomonas aeruginosa Is Essential for Secretion and Assembly of the Type III Translocon

The Journal of infectious diseases, 2003-12, Vol.188 (12), p.1909-1921

2003

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Comparative Analysis of Type III Effector Translocation by Yersinia pseudotuberculosis Expressing Native LcrV or PcrV from Pseudomonas aeruginosa

The Journal of infectious diseases, 2003-07, Vol.188 (2), p.239-249

2003

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Twin arginine translocation in Yersinia

Advances in experimental medicine and biology, 2007, Vol.603, p.258

2007

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Dissection of homologous translocon operons reveals a distinct role for YopD in type III secretion by Yersinia pseudotuberculosis

Microbiology (Society for General Microbiology), 2003-09, Vol.149 (Pt 9), p.2615-2626

2003

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YopD Self-assembly and Binding to LcrV Facilitate Type III Secretion Activity by Yersinia pseudotuberculosis

The Journal of biological chemistry, 2010-08, Vol.285 (33), p.25269-25284

2010

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The Role of ClpB in Bacterial Stress Responses and Virulence

Francisella tularensis IglG Belongs to a Novel Family of PAAR-Like T6SS Proteins and Harbors a Unique N-terminal Extension Required for Virulence

Exploring the Diversity Within the Genus Francisella – An Integrated Pan-Genome and Genome-Mining Approach

DotU and VgrG, core components of type VI secretion systems, are essential for Francisella LVS pathogenicity

Unique substrates secreted by the type VI secretion system of Francisella tularensis during intramacrophage infection

A Conserved α-Helix Essential for a Type VI Secretion-Like System of Francisella tularensis

The type IV pili component PilO is a virulence determinant of Francisella novicida

The Role of the Francisella Tularensis Pathogenicity Island in Type VI Secretion, Intracellular Survival, and Modulation of Host Cell Signaling

Importance of PdpC, IglC, IglI, and IglG for Modulation of a Host Cell Death Pathway Induced by Francisella tularensis

IglG and IglI of the Francisella Pathogenicity Island Are Important Virulence Determinants of Francisella tularensis LVS

A functional VipA-VipB interaction is required for the type VI secretion system activity of Vibrio cholerae O1 strain A1552

Microinjection of Francisella tularensis and Listeria monocytogenes reveals the importance of bacterial and host factors for successful replication

A mutagenesis-based approach identifies amino acids in the N-terminal part of Francisella tularensis IglE that critically control Type VI system-mediated secretion

Twin Arginine Translocation System Is Essential for Virulence of Yersinia pseudotuberculosis

The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infection

Impact of the N-Terminal Secretor Domain on YopD Translocator Function in Yersinia pseudotuberculosis Type III Secretion

Diminished LcrV Secretion Attenuates Yersinia pseudotuberculosis Virulence

The Amino-Terminal Part of the Needle-Tip Translocator LcrV of Yersinia pseudotuberculosis Is Required for Early Targeting of YopH and In vivo Virulence

The YopD Translocator of Yersinia pseudotuberculosis Is a Multifunctional Protein Comprised of Discrete Domains

Mapping of a YscY Binding Domain within the LcrH Chaperone That Is Required for Regulation of Yersinia Type III Secretion

PcrH of Pseudomonas aeruginosa Is Essential for Secretion and Assembly of the Type III Translocon

Comparative Analysis of Type III Effector Translocation by Yersinia pseudotuberculosis Expressing Native LcrV or PcrV from Pseudomonas aeruginosa

Twin arginine translocation in Yersinia

Dissection of homologous translocon operons reveals a distinct role for YopD in type III secretion by Yersinia pseudotuberculosis

YopD Self-assembly and Binding to LcrV Facilitate Type III Secretion Activity by Yersinia pseudotuberculosis

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