UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Misfolded proteins activate factor XII in humans, leading to kallikrein formation without initiating coagulation

The Journal of clinical investigation, 2008-09, Vol.118 (9), p.3208-3218

2008

Volltextzugriff (PDF)

High Levels of Coagulation Factor XI as a Risk Factor for Venous Thrombosis

The New England journal of medicine, 2000-03, Vol.342 (10), p.696-701

2000

Volltextzugriff (PDF)

Physiological responses to protein aggregates: Fibrinolysis, coagulation and inflammation (new roles for old factors)

FEBS letters, 2009-08, Vol.583 (16), p.2691-2699

2009

Volltextzugriff (PDF)

Thrombin activatable fibrinolysis inhibitor (TAFI)-How does thrombin regulate fibrinolysis?

Annals of medicine (Helsinki), 2006-01, Vol.38 (6), p.378-388

2006

Volltextzugriff (PDF)

Effect of second- and third-generation oral contraceptives on the protein C system in the absence or presence of the factor VLeiden mutation: a randomized trial

Blood, 2004-02, Vol.103 (3), p.927-933

2004

Volltextzugriff (PDF)

Identification of thrombin activatable fibrinolysis inhibitor (TAFI) in human platelets

Blood, 2003-06, Vol.101 (12), p.4844-4846

2003

Volltextzugriff (PDF)

Regulation of fibrinolysis in plasma by TAFI and protein C is dependent on the concentration of thrombomodulin

Thrombosis and haemostasis, 2001-01, Vol.85 (1), p.5-11

2001

Volltextzugriff (PDF)

Regulation of fibrinolysis by thrombin activatable fibrinolysis inhibitor, an unstable carboxypeptidase B that unites the pathways of coagulation and fibrinolysis

Arteriosclerosis, thrombosis, and vascular biology, 2006-11, Vol.26 (11), p.2445-2453

2006

Volltextzugriff (PDF)

The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1035-1039

2001

Volltextzugriff (PDF)

Thrombin-Activatable Fibrinolysis Inhibitor (TAFI, Plasma Procarboxypeptidase B, Procarboxypeptidase R, Procarboxypeptidase U)

Thrombosis research, 2001-03, Vol.101 (5), p.329-354

2001

Volltextzugriff (PDF)

Plasmin-Mediated Activation and Inactivation of Thrombin-Activatable Fibrinolysis Inhibitor

Biochemistry (Easton), 2002-05, Vol.41 (21), p.6688-6696

2002

Volltextzugriff (PDF)

Oral Contraceptives and the Risk of Venous Thrombosis

The New England journal of medicine, 2001-05, Vol.344 (20), p.1527-1535

2001

Volltextzugriff (PDF)

Generation and Characterization of a Highly Stable Form of Activated Thrombin-activable Fibrinolysis Inhibitor

The Journal of biological chemistry, 2004-02, Vol.279 (8), p.6620-6628

2004

Volltextzugriff (PDF)

Plasma protein S contains zinc essential for efficient activated protein C-independent anticoagulant activity and binding to factor Xa, but not for efficient binding to tissue factor pathway inhibitor

The FASEB journal, 2009-07, Vol.23 (7), p.2244-2253

2009

Volltextzugriff (PDF)

Factor XI enhances fibrin generation and inhibits fibrinolysis in a coagulation model initiated by surface-coated tissue factor

Blood coagulation & fibrinolysis, 2006-06, Vol.17 (4), p.251-257

2006

Volltextzugriff (PDF)

Thrombin Activatable Fibrinolysis Inhibitor (TAFI) at the Interface between Coagulation and Fibrinolysis

Pathophysiology of haemostasis and thrombosis, 2003-01, Vol.33 (5-6), p.375-381

2003

Volltextzugriff (PDF)

Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1057-1064

2001

Volltextzugriff (PDF)

The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1040-1046

2001

Volltextzugriff (PDF)

Thrombin-Activatable Fibrinolysis Inhibitor Deficiency in Cirrhosis Is Not Associated With Increased Plasma Fibrinolysis

Gastroenterology (New York, N.Y. 1943), 2001-07, Vol.121 (1), p.131-139

2001

Volltextzugriff (PDF)

A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.15-21

2000

Volltextzugriff (PDF)

Low-dose oral contraceptives and acquired resistance to activated protein C: a randomised cross-over study

The Lancet (British edition), 1999-12, Vol.354 (9195), p.2036-2040

1999

Volltextzugriff (PDF)

Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

Thrombosis and haemostasis, 1998-11, Vol.80 (5), p.829-835

1998

Volltextzugriff (PDF)

Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.4-8

2000

Volltextzugriff (PDF)

C4b-binding protein inhibits the factor V-dependent but not the factor V-independent cofactor activity of protein S in the activated protein C-mediated inactivation of factor VIIIa

Thrombosis and haemostasis, 2001-05, Vol.85 (5), p.761-765

2001

Volltextzugriff (PDF)

Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.9-14

2000

Volltextzugriff (PDF)

1 – 25 von 146

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(138)
Mit Volltextzugriff
(103)

Publikationsform

Sprache

Englisch
(146)
Japanisch
(29)

Erscheinungsjahr

n.n

Thema

Humans
(114)
Science & Technology
(107)
Life Sciences & Biomedicine
(105)
Biological And Medical Sciences
(76)
Hematology
(58)
Fundamental And Applied Biological Sciences. Psychology
(49)
Molecular And Cellular Biology
(37)
Blood Coagulation. Blood Cells
(35)
Cardiovascular System & Cardiology
(35)
Peripheral Vascular Disease
(34)
Medical Sciences
(30)
Animals
(26)
Adult
(22)
Protein Binding
(22)
Thrombin - Metabolism
(22)
Biochemistry & Molecular Biology
(21)
Female
(21)
Kinetics
(21)
Blood Coagulation
(20)
Fibrinolysis
(19)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Misfolded proteins activate factor XII in humans, leading to kallikrein formation without initiating coagulation

High Levels of Coagulation Factor XI as a Risk Factor for Venous Thrombosis

Physiological responses to protein aggregates: Fibrinolysis, coagulation and inflammation (new roles for old factors)

Thrombin activatable fibrinolysis inhibitor (TAFI)-How does thrombin regulate fibrinolysis?

Effect of second- and third-generation oral contraceptives on the protein C system in the absence or presence of the factor VLeiden mutation: a randomized trial

Identification of thrombin activatable fibrinolysis inhibitor (TAFI) in human platelets

Regulation of fibrinolysis in plasma by TAFI and protein C is dependent on the concentration of thrombomodulin

Regulation of fibrinolysis by thrombin activatable fibrinolysis inhibitor, an unstable carboxypeptidase B that unites the pathways of coagulation and fibrinolysis

The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Thrombin-Activatable Fibrinolysis Inhibitor (TAFI, Plasma Procarboxypeptidase B, Procarboxypeptidase R, Procarboxypeptidase U)

Plasmin-Mediated Activation and Inactivation of Thrombin-Activatable Fibrinolysis Inhibitor

Oral Contraceptives and the Risk of Venous Thrombosis

Generation and Characterization of a Highly Stable Form of Activated Thrombin-activable Fibrinolysis Inhibitor

Plasma protein S contains zinc essential for efficient activated protein C-independent anticoagulant activity and binding to factor Xa, but not for efficient binding to tissue factor pathway inhibitor

Factor XI enhances fibrin generation and inhibits fibrinolysis in a coagulation model initiated by surface-coated tissue factor

Thrombin Activatable Fibrinolysis Inhibitor (TAFI) at the Interface between Coagulation and Fibrinolysis

Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Thrombin-Activatable Fibrinolysis Inhibitor Deficiency in Cirrhosis Is Not Associated With Increased Plasma Fibrinolysis

A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Low-dose oral contraceptives and acquired resistance to activated protein C: a randomised cross-over study

Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

C4b-binding protein inhibits the factor V-dependent but not the factor V-independent cofactor activity of protein S in the activated protein C-mediated inactivation of factor VIIIa

Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Suchergebnisse filtern