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High Levels of Coagulation Factor XI as a Risk Factor for Venous Thrombosis

The New England journal of medicine, 2000-03, Vol.342 (10), p.696-701

2000

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Thrombin‐activatable fibrinolysis inhibitor (TAFI, plasma procarboxypeptidase B, procarboxypeptidase R, procarboxypeptidase U)

Journal of thrombosis and haemostasis, 2003-07, Vol.1 (7), p.1566-1574

2003

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Low-dose oral contraceptives and acquired resistance to activated protein C: a randomised cross-over study

The Lancet (British edition), 1999-12, Vol.354 (9195), p.2036-2040

1999

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A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.15

2000

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Oral Contraceptives and the Risk of Venous Thrombosis

The New England journal of medicine, 2001-05, Vol.344 (20), p.1527-1535

2001

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Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.4

2000

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Thrombin-Activatable Fibrinolysis Inhibitor Deficiency in Cirrhosis Is Not Associated With Increased Plasma Fibrinolysis

Gastroenterology (New York, N.Y. 1943), 2001-07, Vol.121 (1), p.131-139

2001

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Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.9

2000

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Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

Thrombosis and haemostasis, 1998-11, Vol.80 (5), p.829

1998

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The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1035

2001

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Effects of second and third generation oral contraceptives and their respective progestagens on the coagulation system in the absence or presence of the factor V Leiden mutation

Thrombosis and haemostasis, 2002-02, Vol.87 (2), p.199

2002

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Thrombin-mediated activation of factor XI results in a thrombin-activatable fibrinolysis inhibitor-dependent inhibition of fibrinolysis

The Journal of clinical investigation, 1997-05, Vol.99 (10), p.2323-2327

1997

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Impaired healing of cutaneous wounds and colonic anastomoses in mice lacking thrombin‐activatable fibrinolysis inhibitor

Journal of thrombosis and haemostasis, 2003-10, Vol.1 (10), p.2087-2096

2003

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Decreased plasma sensitivity to activated protein C by oral contraceptives is associated with decreases in plasma glucosylceramide

Journal of thrombosis and haemostasis, 2005-05, Vol.3 (5), p.935-938

2005

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Feedback Activation of Factor XI by Thrombin in Plasma Results in Additional Formation of Thrombin That Protects Fibrin Clots From Fibrinolysis

Blood, 1995-10, Vol.86 (8), p.3035-3042

1995

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Human protein S inhibits prothrombinase complex activity on endothelial cells and platelets via direct interactions with factors Va and Xa

The Journal of biological chemistry, 1994-08, Vol.269 (33), p.21051-21058

1994

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Enhancement of rabbit jugular vein thrombolysis by neutralization of factor XI. In vivo evidence for a role of factor XI as an anti-fibrinolytic factor

The Journal of clinical investigation, 1998-01, Vol.101 (1), p.10-14

1998

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Antiphospholipid Antibodies Directed Against a Combination of Phospholipids With Prothrombin, Protein C, or Protein S: An Explanation for Their Pathogenic Mechanism?

Blood, 1993-05, Vol.81 (10), p.2618-2625

1993

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Aging, physical conditioning, and exercise-induced changes in hemostatic factors and reaction products

Journal of applied physiology (1985), 2000-05, Vol.88 (5), p.1558-1564

2000

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Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1057

2001

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The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1040

2001

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Acute inhibitory effect of alcohol on fibrinolysis

European journal of clinical investigation, 2001-02, Vol.31 (2), p.164-170

2001

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Unraveling the mystery of von Willebrand factor

Journal of thrombosis and haemostasis, 2006-03, Vol.4 (3), p.489-495

2006

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Reduced activity of TAFI (thrombin‐activatable fibrinolysis inhibitor) in acute promyelocytic leukaemia

British journal of haematology, 2000-03, Vol.108 (3), p.518-523

2000

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Factor XI dependent and independent activation of thrombin activatable fibrinolysis inhibitor (TAFI) in plasma associated with clot formation

Thrombosis and haemostasis, 1999-12, Vol.82 (6), p.1703

1999

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High Levels of Coagulation Factor XI as a Risk Factor for Venous Thrombosis

Thrombin‐activatable fibrinolysis inhibitor (TAFI, plasma procarboxypeptidase B, procarboxypeptidase R, procarboxypeptidase U)

Low-dose oral contraceptives and acquired resistance to activated protein C: a randomised cross-over study

A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Oral Contraceptives and the Risk of Venous Thrombosis

Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

Thrombin-Activatable Fibrinolysis Inhibitor Deficiency in Cirrhosis Is Not Associated With Increased Plasma Fibrinolysis

Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Effects of second and third generation oral contraceptives and their respective progestagens on the coagulation system in the absence or presence of the factor V Leiden mutation

Thrombin-mediated activation of factor XI results in a thrombin-activatable fibrinolysis inhibitor-dependent inhibition of fibrinolysis

Impaired healing of cutaneous wounds and colonic anastomoses in mice lacking thrombin‐activatable fibrinolysis inhibitor

Decreased plasma sensitivity to activated protein C by oral contraceptives is associated with decreases in plasma glucosylceramide

Feedback Activation of Factor XI by Thrombin in Plasma Results in Additional Formation of Thrombin That Protects Fibrin Clots From Fibrinolysis

Human protein S inhibits prothrombinase complex activity on endothelial cells and platelets via direct interactions with factors Va and Xa

Enhancement of rabbit jugular vein thrombolysis by neutralization of factor XI. In vivo evidence for a role of factor XI as an anti-fibrinolytic factor

Antiphospholipid Antibodies Directed Against a Combination of Phospholipids With Prothrombin, Protein C, or Protein S: An Explanation for Their Pathogenic Mechanism?

Aging, physical conditioning, and exercise-induced changes in hemostatic factors and reaction products

Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Acute inhibitory effect of alcohol on fibrinolysis

Unraveling the mystery of von Willebrand factor

Reduced activity of TAFI (thrombin‐activatable fibrinolysis inhibitor) in acute promyelocytic leukaemia

Factor XI dependent and independent activation of thrombin activatable fibrinolysis inhibitor (TAFI) in plasma associated with clot formation

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