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An anti-RANKL treatment reduces muscle inflammation and dysfunction and strengthens bone in dystrophic mice

Human molecular genetics, 2019-09, Vol.28 (18), p.3101-3112

2019

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Utrophin haploinsufficiency does not worsen the functional performance, resistance to eccentric contractions and force production of dystrophic mice

PloS one, 2018-06, Vol.13 (6), p.e0198408-e0198408

2018

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Muscle RANK is a key regulator of Ca2+ storage, SERCA activity, and function of fast-twitch skeletal muscles

American Journal of Physiology: Cell Physiology, 2016-04, Vol.310 (8), p.C663-C672

2016

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Targeting the Muscle-Bone Unit: Filling Two Needs with One Deed in the Treatment of Duchenne Muscular Dystrophy

Current osteoporosis reports, 2018-10, Vol.16 (5), p.541-553

2018

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Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophy

Acta neuropathologica communications, 2018-04, Vol.6 (1), p.31-31, Article 31

2018

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A short-term statin treatment changes the contractile properties of fast-twitch skeletal muscles

BMC musculoskeletal disorders, 2016-10, Vol.17 (1), p.449-449, Article 449

2016

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Osteoprotegerin and β2 -Agonists Mitigate Muscular Dystrophy in Slow- and Fast-Twitch Skeletal Muscles

The American journal of pathology, 2017-03, Vol.187 (3), p.498-504

2017

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Muscle-specific deletion of SLK/Stk2 enhances p38 activity and myogenesis in mdx mice

Biochimica et biophysica acta. Molecular cell research, 2021-02, Vol.1868 (2), p.118917-118917, Article 118917

2021

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Motor hypertonia and lack of locomotor coordination in mutant mice lacking DSCAM

Journal of neurophysiology, 2016-03, Vol.115 (3), p.1355-1371

2016

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Detection of S-100β Protein in Plasma and Urine After a Mild Traumatic Brain Injury

Canadian journal of neurological sciences, 2019-09, Vol.46 (5), p.599-602

2019

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Macrophage depletion reduces cell proliferation and extracellular matrix accumulation but increases the ultimate tensile strength of injured Achilles tendons

Journal of orthopaedic research, 2014-02, Vol.32 (2), p.279-285

2014

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Blockade of receptor activator of nuclear factor‐κB signaling greatly improves SERCA activity and contractility in atrophic and dystrophic skeletal muscles (731.7)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Osteoprotegerin and β 2 -Agonists Mitigate Muscular Dystrophy in Slow- and Fast-Twitch Skeletal Muscles

The American journal of pathology, 2017-03, Vol.187 (3), p.498

2017

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Muscle RANK is a key regulator of Ca^sup 2+^ storage, SERCA activity, and function of fast-twitch skeletal muscles

American Journal of Physiology: Cell Physiology, 2016-04, Vol.310 (8), p.C663

2016

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Muscle RANK is a key regulator of Ca 2+ storage, SERCA activity, and function of fast-twitch skeletal muscles

American Journal of Physiology: Cell Physiology, 2016-04, Vol.310 (8), p.C663-C672

2016

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Muscle RANK is a key regulator of Ca super( 2+) storage, SERCA activity, and function of fast-twitch skeletal muscles

American Journal of Physiology: Cell Physiology, 2016-04, Vol.310 (8), p.C663-C663

2016

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L’intolérance musculaire aux statines

M.S. Médecine sciences, 2015-12, Vol.31 (12), p.1109-1114

2015

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Physiological role of receptor activator nuclear factor-kB (RANK) in denervation-induced muscle atrophy and dysfunction

Receptors & clinical investigation, 2016-05, Vol.3 (2), p.e1323-1-e1323-6

2016

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Statin intolerance and associated muscular dysfunctions

M.S. Médecine sciences, 2015-12, Vol.31 (12), p.1109

2015

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An anti-RANKL treatment reduces muscle inflammation and dysfunction and strengthens bone in dystrophic mice

Utrophin haploinsufficiency does not worsen the functional performance, resistance to eccentric contractions and force production of dystrophic mice

Muscle RANK is a key regulator of Ca2+ storage, SERCA activity, and function of fast-twitch skeletal muscles

Targeting the Muscle-Bone Unit: Filling Two Needs with One Deed in the Treatment of Duchenne Muscular Dystrophy

Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophy

A short-term statin treatment changes the contractile properties of fast-twitch skeletal muscles

Osteoprotegerin and β2 -Agonists Mitigate Muscular Dystrophy in Slow- and Fast-Twitch Skeletal Muscles

Muscle-specific deletion of SLK/Stk2 enhances p38 activity and myogenesis in mdx mice

Motor hypertonia and lack of locomotor coordination in mutant mice lacking DSCAM

Detection of S-100β Protein in Plasma and Urine After a Mild Traumatic Brain Injury

Macrophage depletion reduces cell proliferation and extracellular matrix accumulation but increases the ultimate tensile strength of injured Achilles tendons

Blockade of receptor activator of nuclear factor‐κB signaling greatly improves SERCA activity and contractility in atrophic and dystrophic skeletal muscles (731.7)

Osteoprotegerin and β 2 -Agonists Mitigate Muscular Dystrophy in Slow- and Fast-Twitch Skeletal Muscles

Muscle RANK is a key regulator of Ca^sup 2+^ storage, SERCA activity, and function of fast-twitch skeletal muscles

Muscle RANK is a key regulator of Ca 2+ storage, SERCA activity, and function of fast-twitch skeletal muscles

Muscle RANK is a key regulator of Ca super( 2+) storage, SERCA activity, and function of fast-twitch skeletal muscles

L’intolérance musculaire aux statines

Physiological role of receptor activator nuclear factor-kB (RANK) in denervation-induced muscle atrophy and dysfunction

Statin intolerance and associated muscular dysfunctions

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