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Identification of a Novel Amino Acid Motif Responsible for Peptide Aggregation in vitro

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Cytotoxic Effect of Synthetic Peptides on Normal and Cancerous Cells

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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In Vitro FRET‐Based Helicase Activity Assay as a Drug Screening Tool

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Alzheimer amyloid beta inhibition of Eg5/kinesin 5 reduces neurotrophin and/or transmitter receptor function

Neurobiology of aging, 2014-08, Vol.35 (8), p.1839-1849

2014

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The Hsp90 Cochaperone, FKBP51, Increases Tau Stability and Polymerizes Microtubules

The Journal of neuroscience, 2010-01, Vol.30 (2), p.591-599

2010

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Chromosome Instability and Mosaic Aneuploidy in Neurodegenerative and Neurodevelopmental Disorders

Frontiers in genetics, 2019-11, Vol.10, p.1092-1092

2019

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The N Terminus of the Retinoblastoma Protein Inhibits DNA Replication via a Bipartite Mechanism Disrupted in Partially Penetrant Retinoblastomas

Molecular and cellular biology, 2016-03, Vol.36 (5), p.832-845

2016

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Alzheimer Aβ disrupts the mitotic spindle and directly inhibits mitotic microtubule motors

Cell cycle (Georgetown, Tex.), 2011-05, Vol.10 (9), p.1397-1410

2011

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Imbalance of Hsp70 family variants fosters tau accumulation

The FASEB journal, 2013-04, Vol.27 (4), p.1450-1459

2013

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Hsc70 Rapidly Engages Tau after Microtubule Destabilization

The Journal of biological chemistry, 2010-05, Vol.285 (22), p.16798-16805

2010

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A novel role for Cdk1/cyclin B in regulating B-raf activation at mitosis

Molecular biology of the cell, 2008-07, Vol.19 (7), p.2907-2915

2008

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Retinoblastoma‐derived peptides directly inhibit the CMG helicase and impair proliferation of cancer cells

The FASEB journal, 2018-04, Vol.32 (S1), p.531.22-531.22

2018

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Molecular Dynamic Simulation Approach Provides Insights into the Tumorigenesis of the N Terminal Portion of the Retinoblastoma Protein

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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B-Raf Is Critical For MAPK Activation during Mitosis and Is Regulated in an M Phase-dependent Manner in Xenopus Egg Extracts

The Journal of biological chemistry, 2006-08, Vol.281 (32), p.22586-22596

2006

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The active Hsc70/tau complex can be exploited to enhance tau turnover without damaging microtubule dynamics

Human molecular genetics, 2015-07, Vol.24 (14), p.3971-3981

2015

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Alzheimer Aβ inhibition of eg5/kin5 reduces neurotrophin/transmitter receptor function

Neurobiology of aging, 2014-02, Vol.35 (8), p.1839-1849

2014

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Aβ INHIBITION OF KINESIN 5 DISRUPTS THE LOCALIZATION AND FUNCTION OF MEMBRANE PROTEINS: IMPLICATIONS FOR NEURONAL RESPONSES TO NEUROTROPHINS, NEUROTRANSMITTERS, GLUCOSE, AND LIPIDS IN AD

Alzheimer's & dementia, 2014-07, Vol.10 (4), p.P474-P474

2014

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P2‐011: Aβ INHIBITION OF KINESIN 5 DISRUPTS THE LOCALIZATION AND FUNCTION OF MEMBRANE PROTEINS: IMPLICATIONS FOR NEURONAL RESPONSES TO NEUROTROPHINS, NEUROTRANSMITTERS, GLUCOSE, AND LIPIDS IN AD

Alzheimer's & dementia, 2014-07, Vol.10 (4S_Part_12), p.P474-P474

2014

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P3‐036: Aß inhibition of kinesin 5 induces neurotransmitter and neurotrophin receptor mislocalization and dysfunction

Alzheimer's & dementia, 2012-07, Vol.8 (4S_Part_13), p.P468-P468

2012

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Aß inhibition of kinesin 5 induces neurotransmitter and neurotrophin receptor mislocalization and dysfunction

Alzheimer's & dementia, 2012-07, Vol.8 (4), p.P468-P468

2012

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Analysis of DNA replication associated chromatin decondensation: in vivo assay for understanding chromatin remodeling mechanisms of selected proteins

Methods in molecular biology (Clifton, N.J.), 2015, Vol.1288, p.289

2015

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P4‐324: Aβ inhibits specific kinesin motors involved in both mitosis and neuronal function; potential implications for neurogenesis and neuroplasticity in Alzheimer's disease and Down syndrome

Alzheimer's & dementia, 2011-07, Vol.7 (4S_Part_24), p.e24-e24

2011

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Aβ inhibits specific kinesin motors involved in both mitosis and neuronal function; potential implications for neurogenesis and neuroplasticity in Alzheimer's disease and Down syndrome

Alzheimer's & dementia, 2011-07, Vol.7 (4), p.e24-e24

2011

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Alzheimer Aβ Induces Chromosome Mis-segregation, Microtubule Defects, Receptor Mis-localization, and Inhibition of Mitotic/Neuronal Kinesins: Implications for Neurogenesis and Plasticity in Neurodegenrative Diseases

Alzheimer's & dementia, 2010-07, Vol.6 (4), p.e22-e22

2010

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P4‐005: Alzheimer Aβ Induces Chromosome Mis‐segregation, Microtubule Defects, Receptor Mis‐localization, and Inhibition of Mitotic/Neuronal Kinesins: Implications for Neurogenesis and Plasticity in Neurodegenrative Diseases

Alzheimer's & dementia, 2010-07, Vol.6 (4S_Part_20), p.e22-e22

2010

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Identification of a Novel Amino Acid Motif Responsible for Peptide Aggregation in vitro

Cytotoxic Effect of Synthetic Peptides on Normal and Cancerous Cells

In Vitro FRET‐Based Helicase Activity Assay as a Drug Screening Tool

Alzheimer amyloid beta inhibition of Eg5/kinesin 5 reduces neurotrophin and/or transmitter receptor function

The Hsp90 Cochaperone, FKBP51, Increases Tau Stability and Polymerizes Microtubules

Chromosome Instability and Mosaic Aneuploidy in Neurodegenerative and Neurodevelopmental Disorders

The N Terminus of the Retinoblastoma Protein Inhibits DNA Replication via a Bipartite Mechanism Disrupted in Partially Penetrant Retinoblastomas

Alzheimer Aβ disrupts the mitotic spindle and directly inhibits mitotic microtubule motors

Imbalance of Hsp70 family variants fosters tau accumulation

Hsc70 Rapidly Engages Tau after Microtubule Destabilization

A novel role for Cdk1/cyclin B in regulating B-raf activation at mitosis

Retinoblastoma‐derived peptides directly inhibit the CMG helicase and impair proliferation of cancer cells

Molecular Dynamic Simulation Approach Provides Insights into the Tumorigenesis of the N Terminal Portion of the Retinoblastoma Protein

B-Raf Is Critical For MAPK Activation during Mitosis and Is Regulated in an M Phase-dependent Manner in Xenopus Egg Extracts

The active Hsc70/tau complex can be exploited to enhance tau turnover without damaging microtubule dynamics

Alzheimer Aβ inhibition of eg5/kin5 reduces neurotrophin/transmitter receptor function

Aβ INHIBITION OF KINESIN 5 DISRUPTS THE LOCALIZATION AND FUNCTION OF MEMBRANE PROTEINS: IMPLICATIONS FOR NEURONAL RESPONSES TO NEUROTROPHINS, NEUROTRANSMITTERS, GLUCOSE, AND LIPIDS IN AD

P2‐011: Aβ INHIBITION OF KINESIN 5 DISRUPTS THE LOCALIZATION AND FUNCTION OF MEMBRANE PROTEINS: IMPLICATIONS FOR NEURONAL RESPONSES TO NEUROTROPHINS, NEUROTRANSMITTERS, GLUCOSE, AND LIPIDS IN AD

P3‐036: Aß inhibition of kinesin 5 induces neurotransmitter and neurotrophin receptor mislocalization and dysfunction

Aß inhibition of kinesin 5 induces neurotransmitter and neurotrophin receptor mislocalization and dysfunction

Analysis of DNA replication associated chromatin decondensation: in vivo assay for understanding chromatin remodeling mechanisms of selected proteins

P4‐324: Aβ inhibits specific kinesin motors involved in both mitosis and neuronal function; potential implications for neurogenesis and neuroplasticity in Alzheimer's disease and Down syndrome

Aβ inhibits specific kinesin motors involved in both mitosis and neuronal function; potential implications for neurogenesis and neuroplasticity in Alzheimer's disease and Down syndrome

Alzheimer Aβ Induces Chromosome Mis-segregation, Microtubule Defects, Receptor Mis-localization, and Inhibition of Mitotic/Neuronal Kinesins: Implications for Neurogenesis and Plasticity in Neurodegenrative Diseases

P4‐005: Alzheimer Aβ Induces Chromosome Mis‐segregation, Microtubule Defects, Receptor Mis‐localization, and Inhibition of Mitotic/Neuronal Kinesins: Implications for Neurogenesis and Plasticity in Neurodegenrative Diseases

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