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Nitric oxide formation by lymphatic bulb and valves is a major regulatory component of lymphatic pumping

American journal of physiology. Heart and circulatory physiology, 2011-11, Vol.301 (5), p.H1897-H1906

2011

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Phasic contractions of rat mesenteric lymphatics increase basal and phasic nitric oxide generation in vivo

American journal of physiology. Heart and circulatory physiology, 2009-10, Vol.297 (4), p.H1319-H1328

2009

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Rapid and Slow Nitric Oxide Responses During Conducted Vasodilation in the In Vivo Intestine and Brain Cortex Microvasculatures

Microcirculation (New York, N.Y. 1994), 2011-11, Vol.18 (8), p.623-634

GLENN BOHLEN, H.

2011

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Cerebral microvascular dilation during hypotension and decreased oxygen tension: a role for nNOS

American journal of physiology. Heart and circulatory physiology, 2007-10, Vol.293 (4), p.H2193-H2201

2007

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Abnormal nitric oxide production in aged rat mesenteric arteries is mediated by NAD(P)H oxidase-derived peroxide

American journal of physiology. Heart and circulatory physiology, 2009-12, Vol.297 (6), p.H2227-H2233

2009

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High concentration of glucose inhibits glomerular endothelial eNOS through a PKC mechanism

American journal of physiology. Renal physiology, 2004-09, Vol.287 (3), p.F384-F392

2004

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Is the Real In Vivo Nitric Oxide Concentration Pico or Nano Molar? Influence of Electrode Size on Unstirred Layers and NO Consumption

Microcirculation (New York, N.Y. 1994), 2013-01, Vol.20 (1), p.30-41

Bohlen, H. Glenn

2013

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Nitric oxide production by mouse renal tubules can be increased by a sodium-dependent mechanism

Nitric oxide, 2007-08, Vol.17 (1), p.33-43

2007

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A perpetual switching system in pulmonary capillaries

Journal of applied physiology (1985), 2019-02, Vol.126 (2), p.494-501

2019

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Periadventitial adipose tissue impairs coronary endothelial function via PKC-beta-dependent phosphorylation of nitric oxide synthase

American journal of physiology. Heart and circulatory physiology, 2009-07, Vol.297 (1), p.H460-H465

2009

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NAD(P)H oxidase-derived peroxide mediates elevated basal and impaired flow-induced NO production in SHR mesenteric arteries in vivo

American journal of physiology. Heart and circulatory physiology, 2008-09, Vol.295 (3), p.H1008-H1016

2008

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Transport of extracellular L-arginine via cationic amino acid transporter is required during in vivo endothelial nitric oxide production

American journal of physiology. Heart and circulatory physiology, 2005-10, Vol.289 (4), p.H1381-H1390

2005

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Metalloproteinases Damage the Insulin Receptor to Cause Insulin Resistance in Spontaneously Hypertensive Rats

Hypertension (Dallas, Tex. 1979), 2008-08, Vol.52 (2), p.215-217

Bohlen, H Glenn

2008

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Impaired compensation to femoral artery ligation in diet-induced obese mice is primarily mediated via suppression of collateral growth by Nox2 and p47phox

American journal of physiology. Heart and circulatory physiology, 2015-10, Vol.309 (7), p.H1207-H1217

2015

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Cerebral microvascular nNOS responds to lowered oxygen tension through a bumetanide-sensitive cotransporter and sodium-calcium exchanger

American journal of physiology. Heart and circulatory physiology, 2008-05, Vol.294 (5), p.H2166-H2173

2008

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Extracellular Arginine Rapidly Dilates In Vivo Intestinal Arteries and Arterioles Through a Nitric Oxide Mechanism

Microcirculation (New York, N.Y. 1994), 2008-02, Vol.15 (2), p.123-135

2008

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Endogenous Adipose-Derived Factors Diminish Coronary Endothelial Function via Inhibition of Nitric Oxide Synthase

Microcirculation (New York, N.Y. 1994), 2008-07, Vol.15 (5), p.417-426

2008

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Mechanisms for early microvascular injury in obesity and type II diabetes

Current hypertension reports, 2004-02, Vol.6 (1), p.60-65

Bohlen, H Glenn

2004

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Multiple Mechanisms Of Early Hyperglycaemic Injury Of The Rat Intestinal Microcirculation

Clinical and Experimental Pharmacology and Physiology, 2002-01, Vol.29 (1-2), p.138-142

2002

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Sodium channels are required during in vivo sodium chloride hyperosmolarity to stimulate increase in intestinal endothelial nitric oxide production

American journal of physiology. Heart and circulatory physiology, 2005-01, Vol.288 (1), p.H89-H95

2005

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Periadventitial adipose tissue impairs coronary endothelial function via PKC-{beta}-dependent phosphorylation of nitric oxide synthase

American journal of physiology. Heart and circulatory physiology, 2009-07, Vol.297 (1), p.H460

2009

Volltextzugriff (PDF)

Transport of extracellular L-arginine via cationic amino acid transporter is required during in vivo endothelial nitric oxide production

American journal of physiology. Heart and circulatory physiology, 2005-10, Vol.58 (4), p.H1381-H1390

2005

Volltextzugriff (PDF)

Sodium channels are required during in vivo sodium chloride hyperosmolarity to stimulate increase in intestinal endothelial nitric oxide production

American journal of physiology. Heart and circulatory physiology, 2005, Vol.57 (1), p.H89-H95

2005

Volltextzugriff (PDF)

Independent Regulation of Periarteriolar and Perivenular Nitric Oxide Mechanisms in the In Vivo Hamster Cheek Pouch Microvasculature

Microcirculation (New York, N.Y. 1994), 2009-05, Vol.16 (4), p.323-330

2009

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High concentration of glucose inhibits glomerular endothelial eNOS through a PKC mechanism

American journal of physiology. Renal physiology, 2004-09, Vol.56 (3), p.F384-F392

2004

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Nitric oxide formation by lymphatic bulb and valves is a major regulatory component of lymphatic pumping

Phasic contractions of rat mesenteric lymphatics increase basal and phasic nitric oxide generation in vivo

Rapid and Slow Nitric Oxide Responses During Conducted Vasodilation in the In Vivo Intestine and Brain Cortex Microvasculatures

Cerebral microvascular dilation during hypotension and decreased oxygen tension: a role for nNOS

Abnormal nitric oxide production in aged rat mesenteric arteries is mediated by NAD(P)H oxidase-derived peroxide

High concentration of glucose inhibits glomerular endothelial eNOS through a PKC mechanism

Is the Real In Vivo Nitric Oxide Concentration Pico or Nano Molar? Influence of Electrode Size on Unstirred Layers and NO Consumption

Nitric oxide production by mouse renal tubules can be increased by a sodium-dependent mechanism

A perpetual switching system in pulmonary capillaries

Periadventitial adipose tissue impairs coronary endothelial function via PKC-beta-dependent phosphorylation of nitric oxide synthase

NAD(P)H oxidase-derived peroxide mediates elevated basal and impaired flow-induced NO production in SHR mesenteric arteries in vivo

Transport of extracellular L-arginine via cationic amino acid transporter is required during in vivo endothelial nitric oxide production

Metalloproteinases Damage the Insulin Receptor to Cause Insulin Resistance in Spontaneously Hypertensive Rats

Impaired compensation to femoral artery ligation in diet-induced obese mice is primarily mediated via suppression of collateral growth by Nox2 and p47phox

Cerebral microvascular nNOS responds to lowered oxygen tension through a bumetanide-sensitive cotransporter and sodium-calcium exchanger

Extracellular Arginine Rapidly Dilates In Vivo Intestinal Arteries and Arterioles Through a Nitric Oxide Mechanism

Endogenous Adipose-Derived Factors Diminish Coronary Endothelial Function via Inhibition of Nitric Oxide Synthase

Mechanisms for early microvascular injury in obesity and type II diabetes

Multiple Mechanisms Of Early Hyperglycaemic Injury Of The Rat Intestinal Microcirculation

Sodium channels are required during in vivo sodium chloride hyperosmolarity to stimulate increase in intestinal endothelial nitric oxide production

Periadventitial adipose tissue impairs coronary endothelial function via PKC-{beta}-dependent phosphorylation of nitric oxide synthase

Transport of extracellular L-arginine via cationic amino acid transporter is required during in vivo endothelial nitric oxide production

Sodium channels are required during in vivo sodium chloride hyperosmolarity to stimulate increase in intestinal endothelial nitric oxide production

Independent Regulation of Periarteriolar and Perivenular Nitric Oxide Mechanisms in the In Vivo Hamster Cheek Pouch Microvasculature

High concentration of glucose inhibits glomerular endothelial eNOS through a PKC mechanism

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