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Krebs-cycle-deficient hereditary cancer syndromes are defined by defects in homologous-recombination DNA repair

Nature genetics, 2018-08, Vol.50 (8), p.1086-1092

2018

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Tumor Microvessel Density as a Prognostic Marker in High-Risk Renal Cell Carcinoma Patients Treated on ECOG-ACRIN E2805

Clinical cancer research, 2018-01, Vol.24 (1), p.217-223

2018

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Genomic Heterogeneity and the Small Renal Mass

Clinical cancer research, 2018-09, Vol.24 (17), p.4137-4144

2018

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KRAS alleles: The LCS6 3′UTR variant and KRAS coding sequence mutations in the NCI-60 panel

Cell cycle (Georgetown, Tex.), 2012-01, Vol.11 (2), p.361-366

2012

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The KRAS-variant and miRNA expression in RTOG endometrial cancer clinical trials 9708 and 9905

PloS one, 2014-04, Vol.9 (4), p.e94167-e94167

2014

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MP04-20 CHARACTERIZATION OF MITOCHONDRIAL DYSFUNCTION IN MODELS OF RENAL ONCOCYTOMA

The Journal of urology, 2023-04, Vol.209 (Supplement 4)

2023

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A KRAS-Variant in Ovarian Cancer Acts as a Genetic Marker of Cancer Risk

Cancer research (Chicago, Ill.), 2010-08, Vol.70 (16), p.6509-6515

2010

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Microvessel density as a prognostic marker in high-risk renal cell carcinoma

Journal of clinical oncology, 2017-05, Vol.35 (15_suppl), p.4565-4565

2017

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Krebs Cycle-Deficient Hereditary Cancer Syndromes are Defined by Homologous Recombination DNA Repair Defects

Nature genetics, 2018-07, Vol.50 (8), p.1086-1092

2018

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MP71-12 GENOMIC CHARACTERIZATION OF SARCOMATOID TRANSFORMATION IN CLEAR CELL RENAL CELL CARCINOMA

The Journal of urology, 2016-04, Vol.195 (4), p.e920-e920

2016

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KRAS alleles: the LCS6 3'UTR variant and KRAS coding sequence mutations in the NCI-60 panel

Cell cycle (Georgetown, Tex.), 2012-01, Vol.11 (2), p.361-366

2012

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MET Inhibition in Clear Cell Renal Cell Carcinoma

Journal of Cancer, 2016-01, Vol.7 (10), p.1205-1214

2016

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The KRAS-Variant and miRNA Expression in RTOG Endometrial Cancer Clinical Trials 9708 and 9905: e94167

PloS one, 2014-04, Vol.9 (4)

2014

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Regulation of NF-kappaB activity by the class I histone deacetylases, HDAC1, HDAC2, HDAC3 corepressors and the spermidine/spermine N1 acetyltransferase 2 (SSAT2) coactivator

Boeke, Marta

2008

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Spermidine/Spermine N1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-κB and cooperates with CBP and P/CAF to enhance NF-κB-dependent transcription

Biochimica et biophysica acta. Gene structure and expression, 2006-10, Vol.1759 (10), p.470-477

2006

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Spermidine/Spermine N1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-kappaB and cooperates with CBP and P/CAF to enhance NF-kappaB-dependent transcription

Biochimica et biophysica acta, 2006-10, Vol.1759 (10), p.470

2006

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Spermidine/Spermine N 1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-κB and cooperates with CBP and P/CAF to enhance NF-κB-dependent transcription

Biochimica et biophysica acta. Gene structure and expression, 2006-10, Vol.1759 (10), p.470-477

2006

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Widespread Transcriptional Scanning in the Testis Modulates Gene Evolution Rates

Cell, 2020-01, Vol.180 (2), p.248-262.e21

2020

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Krebs-cycle-deficient hereditary cancer syndromes are defined by defects in homologous-recombination DNA repair

Tumor Microvessel Density as a Prognostic Marker in High-Risk Renal Cell Carcinoma Patients Treated on ECOG-ACRIN E2805

Genomic Heterogeneity and the Small Renal Mass

KRAS alleles: The LCS6 3′UTR variant and KRAS coding sequence mutations in the NCI-60 panel

The KRAS-variant and miRNA expression in RTOG endometrial cancer clinical trials 9708 and 9905

MP04-20 CHARACTERIZATION OF MITOCHONDRIAL DYSFUNCTION IN MODELS OF RENAL ONCOCYTOMA

A KRAS-Variant in Ovarian Cancer Acts as a Genetic Marker of Cancer Risk

Microvessel density as a prognostic marker in high-risk renal cell carcinoma

Krebs Cycle-Deficient Hereditary Cancer Syndromes are Defined by Homologous Recombination DNA Repair Defects

MP71-12 GENOMIC CHARACTERIZATION OF SARCOMATOID TRANSFORMATION IN CLEAR CELL RENAL CELL CARCINOMA

KRAS alleles: the LCS6 3'UTR variant and KRAS coding sequence mutations in the NCI-60 panel

MET Inhibition in Clear Cell Renal Cell Carcinoma

The KRAS-Variant and miRNA Expression in RTOG Endometrial Cancer Clinical Trials 9708 and 9905: e94167

Regulation of NF-kappaB activity by the class I histone deacetylases, HDAC1, HDAC2, HDAC3 corepressors and the spermidine/spermine N1 acetyltransferase 2 (SSAT2) coactivator

Spermidine/Spermine N1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-κB and cooperates with CBP and P/CAF to enhance NF-κB-dependent transcription

Spermidine/Spermine N1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-kappaB and cooperates with CBP and P/CAF to enhance NF-kappaB-dependent transcription

Spermidine/Spermine N 1-Acetyltransferase 2 (SSAT2) functions as a coactivator for NF-κB and cooperates with CBP and P/CAF to enhance NF-κB-dependent transcription

Widespread Transcriptional Scanning in the Testis Modulates Gene Evolution Rates

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