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Defining the Functional Role of NaV1.7 in Human Nociception

Neuron (Cambridge, Mass.), 2019-03, Vol.101 (5), p.905-919.e8

2019

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Phosphorylation of the Cav3.2 T-type calcium channel directly regulates its gating properties

Proceedings of the National Academy of Sciences - PNAS, 2015-11, Vol.112 (44), p.13705-13710

2015

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CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism

EBioMedicine, 2016-11, Vol.13 (C), p.225-236

2016

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Production of UCP1 a membrane protein from the inner mitochondrial membrane using the cell free expression system in the presence of a fluorinated surfactant

Biochimica et biophysica acta, 2012-03, Vol.1818 (3), p.798-805

2012

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Defining the Functional Role of Na V 1.7 in Human Nociception

Neuron (Cambridge, Mass.), 2019-03, Vol.101 (5), p.905

2019

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Rare NaV1.7 variants associated with painful diabetic peripheral neuropathy

Pain (Amsterdam), 2018-03, Vol.159 (3), p.469-480

2018

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A Recurrent Mutation in CACNA1G Alters Cav3.1 T-Type Calcium-Channel Conduction and Causes Autosomal-Dominant Cerebellar Ataxia

American journal of human genetics, 2015-11, Vol.97 (5), p.726-737

2015

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CACNA1H missense mutations associated with amyotrophic lateral sclerosis alter Cav3.2 T-type calcium channel activity and reticular thalamic neuron firing

Channels (Austin, Tex.), 2016-11, Vol.10 (6), p.466-477

2016

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A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca2+ channels

Scientific reports, 2019-10, Vol.9 (1), p.1-14, Article 15642

2019

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The Novel Activity of Carbamazepine as an Activation Modulator Extends from Na V 1.7 Mutations to the Na V 1.8-S242T Mutant Channel from a Patient with Painful Diabetic Neuropathy

Molecular pharmacology, 2018-11, Vol.94 (5), p.1256-1269

2018

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CACNA1H missense mutations associated with amyotrophic lateral sclerosis alter Ca v 3.2 T-type calcium channel activity and reticular thalamic neuron firing

Channels (Austin, Tex.), 2016-11, Vol.10 (6), p.466-477

2016

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Crystallization of the membrane protein hVDAC1 produced in cell-free system

Biochimica et biophysica acta, 2010-08, Vol.1798 (8), p.1540-1546

2010

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Inhibition of Cav3.2 T-type Calcium Channels by Its Intracellular I-II Loop

The Journal of biological chemistry, 2015-06, Vol.290 (26), p.16168-16176

2015

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The Novel Activity of Carbamazepine as an Activation Modulator Extends from NaV1.7 Mutations to the NaV1.8-S242T Mutant Channel from a Patient with Painful Diabetic Neuropathy

Molecular pharmacology, 2018-11, Vol.94 (5), p.1256-1269

2018

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Whole-genome sequencing of patients with rare diseases in a national health system

Nature (London), 2020-07, Vol.583 (7814), p.96-102

2020

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Whole-genome sequencing of patients with rare diseases in a national health system

Nature (London), 2020-07, Vol.583 (7814), p.96-102

2020

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A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca 2+ channels

Scientific reports, 2019-10, Vol.9 (1), p.15642

2019

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An iPSC model of hereditary sensory neuropathy-1 reveals L-serine-responsive deficits in neuronal ganglioside composition and axoglial interactions

Cell reports. Medicine, 2021-07, Vol.2 (7), p.100345, Article 100345

2021

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Simple Method for the Reconstitution of Membrane Proteins into Giant Unilamellar Vesicles

The Journal of membrane biology, 2010-02, Vol.233 (1-3), p.85-92

2010

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Investigating genotype–phenotype relationship of extreme neuropathic pain disorders in a UK national cohort

Brain communications, 2023, Vol.5 (2), p.fcad037

2023

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Assaying the proton transport and regulation of UCP1 using solid supported membranes

European biophysics journal, 2012-08, Vol.41 (8), p.675-679

2012

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Single-step production of functional OEP24 proteoliposomes

Protein expression and purification, 2010, Vol.69 (1), p.106-111

2010

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Structural approaches of the mitochondrial carrier family

Methods in molecular biology (Clifton, N.J.), 2010, Vol.654, p.105

2010

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Impaired Transport of Nucleotides in a Mitochondrial Carrier Explains Severe Human Genetic Diseases

ACS chemical biology, 2012-07, Vol.7 (7), p.1164-1169

2012

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Defining the Functional Role of NaV1.7 in Human Nociception

Phosphorylation of the Cav3.2 T-type calcium channel directly regulates its gating properties

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism

Production of UCP1 a membrane protein from the inner mitochondrial membrane using the cell free expression system in the presence of a fluorinated surfactant

Defining the Functional Role of Na V 1.7 in Human Nociception

Rare NaV1.7 variants associated with painful diabetic peripheral neuropathy

A Recurrent Mutation in CACNA1G Alters Cav3.1 T-Type Calcium-Channel Conduction and Causes Autosomal-Dominant Cerebellar Ataxia

CACNA1H missense mutations associated with amyotrophic lateral sclerosis alter Cav3.2 T-type calcium channel activity and reticular thalamic neuron firing

A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca2+ channels

The Novel Activity of Carbamazepine as an Activation Modulator Extends from Na V 1.7 Mutations to the Na V 1.8-S242T Mutant Channel from a Patient with Painful Diabetic Neuropathy

CACNA1H missense mutations associated with amyotrophic lateral sclerosis alter Ca v 3.2 T-type calcium channel activity and reticular thalamic neuron firing

Crystallization of the membrane protein hVDAC1 produced in cell-free system

Inhibition of Cav3.2 T-type Calcium Channels by Its Intracellular I-II Loop

The Novel Activity of Carbamazepine as an Activation Modulator Extends from NaV1.7 Mutations to the NaV1.8-S242T Mutant Channel from a Patient with Painful Diabetic Neuropathy

Whole-genome sequencing of patients with rare diseases in a national health system

Whole-genome sequencing of patients with rare diseases in a national health system

A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca 2+ channels

An iPSC model of hereditary sensory neuropathy-1 reveals L-serine-responsive deficits in neuronal ganglioside composition and axoglial interactions

Simple Method for the Reconstitution of Membrane Proteins into Giant Unilamellar Vesicles

Investigating genotype–phenotype relationship of extreme neuropathic pain disorders in a UK national cohort

Assaying the proton transport and regulation of UCP1 using solid supported membranes

Single-step production of functional OEP24 proteoliposomes

Structural approaches of the mitochondrial carrier family

Impaired Transport of Nucleotides in a Mitochondrial Carrier Explains Severe Human Genetic Diseases

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