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Viral entry mechanisms: human papillomavirus and a long journey from extracellular matrix to the nucleus

The FEBS journal, 2009-12, Vol.276 (24), p.7206-7216

2009

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PML nuclear body-residing proteins sequentially associate with HPV genome after infectious nuclear delivery

PLoS pathogens, 2019-02, Vol.15 (2), p.e1007590-e1007590

2019

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Multiple Heparan Sulfate Binding Site Engagements Are Required for the Infectious Entry of Human Papillomavirus Type 16

Journal of Virology, 2013-11, Vol.87 (21), p.11426-11437

2013

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Target cell cyclophilins facilitate human papillomavirus type 16 infection

PLoS pathogens, 2009-07, Vol.5 (7), p.e1000524-e1000524

2009

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Cyclophilins Facilitate Dissociation of the Human Papillomavirus Type 16 Capsid Protein L1 from the L2/DNA Complex following Virus Entry

Journal of Virology, 2012-09, Vol.86 (18), p.9875-9887

2012

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A new cell culture model to genetically dissect the complete human papillomavirus life cycle

PLoS pathogens, 2018-03, Vol.14 (3), p.e1006846-e1006846

2018

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Incoming human papillomavirus 16 genome is lost in PML protein‐deficient HaCaT keratinocytes

Cellular microbiology, 2017-05, Vol.19 (5), p.e12708-n/a

2017

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Human papillomavirus species-specific interaction with the basement membrane-resident non-heparan sulfate receptor

Viruses, 2014-12, Vol.6 (12), p.4856-4879

2014

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The cytoskeleton in papillomavirus infection

Viruses, 2011-03, Vol.3 (3), p.260-271

2011

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Human Papillomavirus Entry: Hiding in a Bubble

Journal of virology, 2016-09, Vol.90 (18), p.8032-8035

2016

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The nuclear retention signal of HPV16 L2 protein is essential for incoming viral genome to transverse the trans-Golgi network

Virology (New York, N.Y.), 2014-06, Vol.458, p.93-105

2014

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Human Papillomavirus Genome Copy Number Is Maintained by S-Phase Amplification, Genome Loss to the Cytosol during Mitosis, and Degradation in G1 Phase

Journal of virology, 2023-02, Vol.97 (2), p.e0187922-e0187922

2023

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Human Papillomavirus Genome Copy Number Is Maintained by S-Phase Amplification, Genome Loss to the Cytosol during Mitosis, and Degradation in G 1 Phase

Journal of virology, 2023-02, Vol.97 (2), p.e0187922

2023

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Correction for Bienkowska-Haba et al., "Genome-Wide Transcriptome Analysis of Human Papillomavirus 16-Infected Primary Keratinocytes Reveals Subtle Perturbations Mostly Due To E7 Protein Expression"

Journal of virology, 2022-11, Vol.96 (22), p.e0165922-e0165922

2022

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Experimental Support for Human Papillomavirus Genome Amplification Early after Infectious Delivery

Journal of virology, 2023-06, Vol.97 (6), p.e0021423-e0021423

2023

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Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus

Virus research, 2017-03, Vol.231, p.1-9

2017

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Genome-Wide Transcriptome Analysis of Human Papillomavirus 16-Infected Primary Keratinocytes Reveals Subtle Perturbations Mostly due to E7 Protein Expression

Journal of virology, 2020-01, Vol.94 (3)

2020

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Human Papillomavirus Major Capsid Protein L1 Remains Associated with the Incoming Viral Genome throughout the Entry Process

Journal of virology, 2017-08, Vol.91 (16)

2017

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Incoming human papillomavirus type 16 genome resides in a vesicular compartment throughout mitosis

Proceedings of the National Academy of Sciences - PNAS, 2016-05, Vol.113 (22), p.6289-6294

2016

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Topography of the Human Papillomavirus Minor Capsid Protein L2 during Vesicular Trafficking of Infectious Entry

Journal of virology, 2015-10, Vol.89 (20), p.10442-10452

2015

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Retinoblastoma Protein Is Required for Epstein-Barr Virus Replication in Differentiated Epithelia

Journal of virology, 2023-02, Vol.97 (2), p.e0103222-e0103222

2023

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Epstein-Barr virus stably confers an invasive phenotype to epithelial cells through reprogramming of the WNT pathway

Oncotarget, 2018-02, Vol.9 (12), p.10417-10435

2018

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Structural Basis of Oligosaccharide Receptor Recognition by Human Papillomavirus

The Journal of biological chemistry, 2011-01, Vol.286 (4), p.2617-2624

2011

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Target Cell Cyclophilins Facilitate Human Papillomavirus Type 16 Infection: e1000524

PLoS pathogens, 2009-07, Vol.5 (7)

2009

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Nitric oxide production by pulmonary leukocytes from induced sputum in patients with asthma and its effect on epithelial cell viability

Archivum Immunologiae et Therapiae Experimentalis, 2006-06, Vol.54 (3), p.201-207

2006

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Viral entry mechanisms: human papillomavirus and a long journey from extracellular matrix to the nucleus

PML nuclear body-residing proteins sequentially associate with HPV genome after infectious nuclear delivery

Multiple Heparan Sulfate Binding Site Engagements Are Required for the Infectious Entry of Human Papillomavirus Type 16

Target cell cyclophilins facilitate human papillomavirus type 16 infection

Cyclophilins Facilitate Dissociation of the Human Papillomavirus Type 16 Capsid Protein L1 from the L2/DNA Complex following Virus Entry

A new cell culture model to genetically dissect the complete human papillomavirus life cycle

Incoming human papillomavirus 16 genome is lost in PML protein‐deficient HaCaT keratinocytes

Human papillomavirus species-specific interaction with the basement membrane-resident non-heparan sulfate receptor

The cytoskeleton in papillomavirus infection

Human Papillomavirus Entry: Hiding in a Bubble

The nuclear retention signal of HPV16 L2 protein is essential for incoming viral genome to transverse the trans-Golgi network

Human Papillomavirus Genome Copy Number Is Maintained by S-Phase Amplification, Genome Loss to the Cytosol during Mitosis, and Degradation in G1 Phase

Human Papillomavirus Genome Copy Number Is Maintained by S-Phase Amplification, Genome Loss to the Cytosol during Mitosis, and Degradation in G 1 Phase

Correction for Bienkowska-Haba et al., "Genome-Wide Transcriptome Analysis of Human Papillomavirus 16-Infected Primary Keratinocytes Reveals Subtle Perturbations Mostly Due To E7 Protein Expression"

Experimental Support for Human Papillomavirus Genome Amplification Early after Infectious Delivery

Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus

Genome-Wide Transcriptome Analysis of Human Papillomavirus 16-Infected Primary Keratinocytes Reveals Subtle Perturbations Mostly due to E7 Protein Expression

Human Papillomavirus Major Capsid Protein L1 Remains Associated with the Incoming Viral Genome throughout the Entry Process

Incoming human papillomavirus type 16 genome resides in a vesicular compartment throughout mitosis

Topography of the Human Papillomavirus Minor Capsid Protein L2 during Vesicular Trafficking of Infectious Entry

Retinoblastoma Protein Is Required for Epstein-Barr Virus Replication in Differentiated Epithelia

Epstein-Barr virus stably confers an invasive phenotype to epithelial cells through reprogramming of the WNT pathway

Structural Basis of Oligosaccharide Receptor Recognition by Human Papillomavirus

Target Cell Cyclophilins Facilitate Human Papillomavirus Type 16 Infection: e1000524

Nitric oxide production by pulmonary leukocytes from induced sputum in patients with asthma and its effect on epithelial cell viability

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