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Characterization of new-generation aminoglycoside promoting premature termination codon readthrough in cancer cells

RNA biology, 2017-03, Vol.14 (3), p.378-388

2017

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Statistical analysis of readthrough levels for nonsense mutations in mammalian cells reveals a major determinant of response to gentamicin

PLoS genetics, 2012-03, Vol.8 (3), p.e1002608

2012

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Readthrough of premature termination codons in the adenomatous polyposis coli gene restores its biological activity in human cancer cells

PloS one, 2011-08, Vol.6 (8), p.e24125-e24125

2011

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In vitro prediction of stop-codon suppression by intravenous gentamicin in patients with cystic fibrosis: a pilot study

BMC medicine, 2007-03, Vol.5 (1), p.5-5

2007

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Factors influencing readthrough therapy for frequent cystic fibrosis premature termination codons

ERJ open research, 2018-01, Vol.4 (1), p.80-2017

2018

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Different Modes of Stop Codon Restriction by the Stylonychia and Paramecium eRF1 Translation Termination Factors

Proceedings of the National Academy of Sciences - PNAS, 2007-06, Vol.104 (26), p.10824-10829

2007

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Drug-induced readthrough of premature stop codons leads to the stabilization of laminin α2 chain mRNA in CMD myotubes

The journal of gene medicine, 2008-02, Vol.10 (2), p.217-224

2008

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Increased expression of tryptophan and tyrosine tRNAs elevates stop codon readthrough of reporter systems in human cell lines

Nucleic acids research, 2021-05, Vol.49 (9), p.5202-5215

2021

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Sense from nonsense: therapies for premature stop codon diseases

Trends in molecular medicine, 2012-11, Vol.18 (11), p.679-688

2012

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Rescue of non-sense mutated p53 tumor suppressor gene by aminoglycosides

Nucleic acids research, 2011-04, Vol.39 (8), p.3350-3362

2011

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2-Guanidino-quinazoline promotes the readthrough of nonsense mutations underlying human genetic diseases

Proceedings of the National Academy of Sciences - PNAS, 2022-08, Vol.119 (35)

2022

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2-Guanidino-quinazoline promotes the readthrough of nonsense mutations underlying human genetic diseases

Proceedings of the National Academy of Sciences - PNAS, 2022-08, Vol.119 (35), p.1-e2122004119

2022

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In vitro rescue of nonsense variations by drug-induced readthrough of premature termination codons in progressive familial intrahepatic cholestasis type 2

Journal of hepatology, 2022-07, Vol.77, p.S514

2022

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Towards a computational model for −1 eukaryotic frameshifting sites

Bioinformatics, 2003-02, Vol.19 (3), p.327-335

2003

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TLN468 changes the pattern of tRNA used to read through premature termination codons in CFTR

Journal of cystic fibrosis, 2024-08

2024

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Pharmacological Premature Termination Codon Readthrough of ABCB11 in Bile Salt Export Pump Deficiency: An In Vitro Study

Hepatology (Baltimore, Md.), 2021-04, Vol.73 (4), p.1449-1463

2021

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The U UGA C sequence provides a favorable context to ELX-02 induced CFTR readthrough

Journal of cystic fibrosis, 2023-05, Vol.22 (3), p.560-563

2023

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Translational errors: from yeast to new therapeutic targets: Translational fidelity

FEMS yeast research, 2010-12, Vol.10 (8), p.1070-1082

2010

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Translational errors: from yeast to new therapeutic targets

FEMS yeast research, 2010-12, Vol.10 (8), p.1070-1082

2010

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Drug-induced readthrough of premature stop codons leads to the stabilization of laminin alpha 2 chain mRNA in CMD myotubes

The journal of gene medicine, 2008-02, Vol.10 (2), p.217-224

2008

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La réactivation par translecture du gène p53 possédant une mutation non-sens induit l’apoptose de cellules cancéreuses

M.S. Médecine sciences, 2011-06, Vol.27 (6-7), p.585-586

2011

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Rescue of nonsense mutated p53 by readthrough leads to apoptosis in cancers cells

M.S. Médecine sciences, 2011-06, Vol.27 (6-7), p.585

2011

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Thérapie spécifique d’allèle: Suppression de mutations non-sens par des inducteurs de « translecture

M.S. Médecine sciences, 2012-02, Vol.28 (2), p.193-199

2012

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Allele-specific therapy: suppression of nonsense mutations by readthrough inducers

M.S. Médecine sciences, 2012-02, Vol.28 (2), p.193

2012

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Nectinepsin: A New Extracellular Matrix Protein of the Pexin Family

The Journal of biological chemistry, 1996-10, Vol.271 (42), p.26220-26226

1996

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Characterization of new-generation aminoglycoside promoting premature termination codon readthrough in cancer cells

Statistical analysis of readthrough levels for nonsense mutations in mammalian cells reveals a major determinant of response to gentamicin

Readthrough of premature termination codons in the adenomatous polyposis coli gene restores its biological activity in human cancer cells

In vitro prediction of stop-codon suppression by intravenous gentamicin in patients with cystic fibrosis: a pilot study

Factors influencing readthrough therapy for frequent cystic fibrosis premature termination codons

Different Modes of Stop Codon Restriction by the Stylonychia and Paramecium eRF1 Translation Termination Factors

Drug-induced readthrough of premature stop codons leads to the stabilization of laminin α2 chain mRNA in CMD myotubes

Increased expression of tryptophan and tyrosine tRNAs elevates stop codon readthrough of reporter systems in human cell lines

Sense from nonsense: therapies for premature stop codon diseases

Rescue of non-sense mutated p53 tumor suppressor gene by aminoglycosides

2-Guanidino-quinazoline promotes the readthrough of nonsense mutations underlying human genetic diseases

2-Guanidino-quinazoline promotes the readthrough of nonsense mutations underlying human genetic diseases

In vitro rescue of nonsense variations by drug-induced readthrough of premature termination codons in progressive familial intrahepatic cholestasis type 2

Towards a computational model for −1 eukaryotic frameshifting sites

TLN468 changes the pattern of tRNA used to read through premature termination codons in CFTR

Pharmacological Premature Termination Codon Readthrough of ABCB11 in Bile Salt Export Pump Deficiency: An In Vitro Study

The U UGA C sequence provides a favorable context to ELX-02 induced CFTR readthrough

Translational errors: from yeast to new therapeutic targets: Translational fidelity

Translational errors: from yeast to new therapeutic targets

Drug-induced readthrough of premature stop codons leads to the stabilization of laminin alpha 2 chain mRNA in CMD myotubes

La réactivation par translecture du gène p53 possédant une mutation non-sens induit l’apoptose de cellules cancéreuses

Rescue of nonsense mutated p53 by readthrough leads to apoptosis in cancers cells

Thérapie spécifique d’allèle: Suppression de mutations non-sens par des inducteurs de « translecture

Allele-specific therapy: suppression of nonsense mutations by readthrough inducers

Nectinepsin: A New Extracellular Matrix Protein of the Pexin Family

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