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OCT4 and SOX2 Work as Transcriptional Activators in Reprogramming Human Fibroblasts

Cell reports (Cambridge), 2017-08, Vol.20 (7), p.1585-1596

2017

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Direct engagement of the PI3K pathway by mutant KIT dominates oncogenic signaling in gastrointestinal stromal tumor

Proceedings of the National Academy of Sciences - PNAS, 2017-10, Vol.114 (40), p.E8448-E8457

2017

Volltextzugriff (PDF)

Polycomb Responds to Low Levels of Transcription

Cell reports (Cambridge), 2017-07, Vol.20 (4), p.785-793

2017

Volltextzugriff (PDF)

Regulation of a mammalian gene bearing a CpG island promoter and a distal enhancer

Cell reports (Cambridge), 2013-08, Vol.4 (3), p.445-453

2013

Volltextzugriff (PDF)

A Distant Upstream Locus Control Region Is Critical for Expression of the Kit Receptor Gene in Mast Cells

Molecular and Cellular Biology, 2006-08, Vol.26 (15), p.5850-5860

2006

Volltextzugriff (PDF)

Comparative analysis of mutations in the p53 and K‐ras genes in pancreatic cancer

International journal of cancer, 1994-07, Vol.58 (2), p.185-191

1994

Volltextzugriff (PDF)

A RSC/Nucleosome Complex Determines Chromatin Architecture and Facilitates Activator Binding

Cell, 2010-04, Vol.141 (3), p.407-418

2010

Volltextzugriff (PDF)

The W(sh), W(57), and Ph Kit expression mutations define tissue-specific control elements located between -23 and -154 kb upstream of Kit

Blood, 1999-10, Vol.94 (8), p.2658

1999

Volltextzugriff (PDF)

Isolation of tissue-type plasminogen activator, cathepsin H, and non-specific cross-reacting antigen from SK-PC-1 pancreas cancer cells using subtractive hybridization

FEBS letters, 1996-04, Vol.385 (1), p.72-76

1996

Volltextzugriff (PDF)

Expressed sequence tag (EST) phenotyping of HT-29 cells: cloning of ser/thr protein kinase EMK1, kinesin KIF3B, and of transcripts that include Alu repeated elements

Biochimica et biophysica acta, 1999-07, Vol.1450 (3), p.254-264

1999

Volltextzugriff (PDF)

Erratum to: ‘Expressed sequence tag (EST) phenotyping of HT-29 cells: cloning of ser/thr protein kinase EMK1, kinesin KIF3B, and of transcripts that include Alu repeated elements’: [ Biochimica et Biophysica Acta, 1450 (1999) 254–264]

Biochimica et biophysica acta. Molecular cell research, 1999-12, Vol.1452 (3), p.322-322

1999

Volltextzugriff (PDF)

The Wsh, W57, and Ph Kit expression mutations define tissue-specific control elements located between -23 and -154 kb upstream of Kit

Blood, 1999-10, Vol.94 (8), p.2658-2666

1999

Volltextzugriff (PDF)

The Wsh and Ph mutations affect the c-kit expression profile: c-kit misexpression in embryogenesis impairs melanogenesis in Wsh and Ph mutant mice

Proceedings of the National Academy of Sciences - PNAS, 1995-04, Vol.92 (9), p.3754-3758

1995

Volltextzugriff (PDF)

The Wshand Ph Mutations Affect the c-kit Expression Profile: c-kit Misexpression in Embryogenesis Impairs Melanogenesis in Wshand Ph Mutant Mice

Proceedings of the National Academy of Sciences - PNAS, 1995-04, Vol.92 (9), p.3754-3758

1995

Volltextzugriff (PDF)

Chromosome abnormalities in peripheral blood lymphocytes from untreated Hodgkin's patients: a possible evidence for chromosome instability

Human genetics, 1988-04, Vol.78 (4), p.320-324

1988

Volltextzugriff (PDF)

Centromere splitting in bladder cancer

Human genetics, 1990-07, Vol.85 (2), p.184-186

1990

Volltextzugriff (PDF)

Trisomy 7 may be a primary change in non-invasive transitional cell carcinoma of the bladder

Cancer genetics and cytogenetics, 1990, Vol.50 (1), p.9-14

1990

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Cell Biology
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Biological And Medical Sciences
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Base Sequence
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Biochemistry & Molecular Biology
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Male
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Medical Sciences
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Molecular Sequence Data
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Mutation
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Proto-Oncogene Proteins C-Kit - Genetics
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Tumor Cells, Cultured
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Carcinoma
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Chromatin - Metabolism
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Chromosome Mapping
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Cloning, Molecular
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OCT4 and SOX2 Work as Transcriptional Activators in Reprogramming Human Fibroblasts

Direct engagement of the PI3K pathway by mutant KIT dominates oncogenic signaling in gastrointestinal stromal tumor

Polycomb Responds to Low Levels of Transcription

Regulation of a mammalian gene bearing a CpG island promoter and a distal enhancer

A Distant Upstream Locus Control Region Is Critical for Expression of the Kit Receptor Gene in Mast Cells

Comparative analysis of mutations in the p53 and K‐ras genes in pancreatic cancer

A RSC/Nucleosome Complex Determines Chromatin Architecture and Facilitates Activator Binding

The W(sh), W(57), and Ph Kit expression mutations define tissue-specific control elements located between -23 and -154 kb upstream of Kit

Isolation of tissue-type plasminogen activator, cathepsin H, and non-specific cross-reacting antigen from SK-PC-1 pancreas cancer cells using subtractive hybridization

Expressed sequence tag (EST) phenotyping of HT-29 cells: cloning of ser/thr protein kinase EMK1, kinesin KIF3B, and of transcripts that include Alu repeated elements

Erratum to: ‘Expressed sequence tag (EST) phenotyping of HT-29 cells: cloning of ser/thr protein kinase EMK1, kinesin KIF3B, and of transcripts that include Alu repeated elements’: [ Biochimica et Biophysica Acta, 1450 (1999) 254–264]

The Wsh, W57, and Ph Kit expression mutations define tissue-specific control elements located between -23 and -154 kb upstream of Kit

The Wsh and Ph mutations affect the c-kit expression profile: c-kit misexpression in embryogenesis impairs melanogenesis in Wsh and Ph mutant mice

The Wshand Ph Mutations Affect the c-kit Expression Profile: c-kit Misexpression in Embryogenesis Impairs Melanogenesis in Wshand Ph Mutant Mice

Chromosome abnormalities in peripheral blood lymphocytes from untreated Hodgkin's patients: a possible evidence for chromosome instability

Centromere splitting in bladder cancer

Trisomy 7 may be a primary change in non-invasive transitional cell carcinoma of the bladder

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