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ROS1 Rearrangements Define a Unique Molecular Class of Lung Cancers

Journal of clinical oncology, 2012-03, Vol.30 (8), p.863-870

2012

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Genotypic and histological evolution of lung cancers acquiring resistance to EGFR inhibitors

Science translational medicine, 2011-03, Vol.3 (75), p.75ra26

2011

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MET Amplification Identifies a Small and Aggressive Subgroup of Esophagogastric Adenocarcinoma With Evidence of Responsiveness to Crizotinib

Journal of clinical oncology, 2011-12, Vol.29 (36), p.4803-4810

2011

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Increasing the Adoption and Diffusion of a Novel Pharmacological Therapy That Is Both Mortality Reducing and Cost-Effective

Journal of the American Heart Association, 2019-02, Vol.8 (3), p.e011783

2019

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Histologic and cytomorphologic features of ALK-rearranged lung adenocarcinomas

Modern pathology, 2012-11, Vol.25 (11), p.1462-1472

2012

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Introduction, Cancellation, and Future Promise of Medicare Cardiac Episode Payment Models

Circulation Cardiovascular quality and outcomes, 2018-01, Vol.11 (1)

2018

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Detection of Novel Actionable Genetic Changes in Salivary Duct Carcinoma Helps Direct Patient Treatment

Clinical cancer research, 2013-01, Vol.19 (2), p.480-490

2013

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Activation of PI3K Signaling in Merkel Cell Carcinoma

Clinical cancer research, 2012-03, Vol.18 (5), p.1227-1236

2012

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The Interplay between Financial Incentives, Institutional Culture, and Physician Behavior: An Incompletely Understood Relationship Worth Elucidating

Journal of hospital medicine, 2019-01, Vol.14 (1), p.60-62

2019

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The dichotomy in carcinogenesis of the distal esophagus and esophagogastric junction: intestinal-type vs cardiac-type mucosa-associated adenocarcinoma

Modern pathology, 2011-09, Vol.24 (9), p.1177-1190

2011

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Lack of Improvement in 30-day Readmission Rates for Patients Hospitalized with Heart Failure: Data from Get With The Guidelines-Heart Failure Registry

Journal of cardiac failure, 2015-08, Vol.21 (8), p.S10-S10

2015

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BRAF gene amplification can promote acquired resistance to MEK inhibitors in cancer cells harboring the BRAF V600E mutation

Science signaling, 2010-11, Vol.3 (149), p.ra84

2010

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Clinical activity of crizotinib in advanced non-small cell lung cancer (NSCLC) harboring ROS1 gene rearrangement

Journal of clinical oncology, 2012-05, Vol.30 (15_suppl), p.7508-7508

2012

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Activity of Crizotinib (PF02341066), a Dual Mesenchymal-Epithelial Transition (MET) and Anaplastic Lymphoma Kinase (ALK) Inhibitor, in a Non-small Cell Lung Cancer Patient with De Novo MET Amplification

Journal of thoracic oncology, 2011-05, Vol.6 (5), p.942-946

2011

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A potential role for targeted therapy in a subset of metastasizing adnexal carcinomas

Modern pathology, 2011-07, Vol.24 (7), p.974-982

2011

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Trends in 30-Day Readmission Rates for Patients Hospitalized With Heart Failure: Findings From the GWTG-HF Registry

Circulation. Heart failure, 2016-06, Vol.9 (6)

2016

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Efficacy and safety of crizotinib in patients with advanced ROS1 -rearranged non-small cell lung cancer (NSCLC)

Journal of clinical oncology, 2013-05, Vol.31 (15_suppl), p.8032-8032

2013

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Inactivation of the tumor suppressor WTX in a subset of pediatric tumors

Genes chromosomes & cancer, 2014-01, Vol.53 (1), p.67-77

2014

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Abstract 2210: Activation of PI3K in Merkel cell carcinoma

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.2210-2210

2011

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ROS1 Rearrangements Define a Unique Molecular Class of Lung Cancers

Genotypic and histological evolution of lung cancers acquiring resistance to EGFR inhibitors

MET Amplification Identifies a Small and Aggressive Subgroup of Esophagogastric Adenocarcinoma With Evidence of Responsiveness to Crizotinib

Increasing the Adoption and Diffusion of a Novel Pharmacological Therapy That Is Both Mortality Reducing and Cost-Effective

Histologic and cytomorphologic features of ALK-rearranged lung adenocarcinomas

Introduction, Cancellation, and Future Promise of Medicare Cardiac Episode Payment Models

Detection of Novel Actionable Genetic Changes in Salivary Duct Carcinoma Helps Direct Patient Treatment

Activation of PI3K Signaling in Merkel Cell Carcinoma

The Interplay between Financial Incentives, Institutional Culture, and Physician Behavior: An Incompletely Understood Relationship Worth Elucidating

The dichotomy in carcinogenesis of the distal esophagus and esophagogastric junction: intestinal-type vs cardiac-type mucosa-associated adenocarcinoma

Lack of Improvement in 30-day Readmission Rates for Patients Hospitalized with Heart Failure: Data from Get With The Guidelines-Heart Failure Registry

BRAF gene amplification can promote acquired resistance to MEK inhibitors in cancer cells harboring the BRAF V600E mutation

Clinical activity of crizotinib in advanced non-small cell lung cancer (NSCLC) harboring ROS1 gene rearrangement

Activity of Crizotinib (PF02341066), a Dual Mesenchymal-Epithelial Transition (MET) and Anaplastic Lymphoma Kinase (ALK) Inhibitor, in a Non-small Cell Lung Cancer Patient with De Novo MET Amplification

A potential role for targeted therapy in a subset of metastasizing adnexal carcinomas

Trends in 30-Day Readmission Rates for Patients Hospitalized With Heart Failure: Findings From the GWTG-HF Registry

Efficacy and safety of crizotinib in patients with advanced ROS1 -rearranged non-small cell lung cancer (NSCLC)

Inactivation of the tumor suppressor WTX in a subset of pediatric tumors

Abstract 2210: Activation of PI3K in Merkel cell carcinoma

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