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ROS-Mediated p53 Induction of Lpin1 Regulates Fatty Acid Oxidation in Response to Nutritional Stress

Molecular cell, 2011-11, Vol.44 (3), p.491-501

2011

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Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span

Current biology, 1998-02, Vol.8 (5), p.279-282

1998

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Heterogeneity in leukemia cells that escape drug-induced senescence-like state

Cell death & disease, 2023-08, Vol.14 (8), p.503-503, Article 503

2023

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BTG2 antagonizes Pin1 in response to mitogens and telomere disruption during replicative senescence

Aging cell, 2010-10, Vol.9 (5), p.747-760

2010

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Pidd, a new death-domain-containing protein, is induced by p53 and promotes apoptosis

Nature genetics, 2000-09, Vol.26 (1), p.122-127

2000

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Differential utilization of two ATP-generating pathways is regulated by p53

Cancer cell, 2006-07, Vol.10 (1), p.4-6

2006

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Proteasome-independent p53 degradation

Cell research, 2013-05, Vol.23 (5), p.597-598

2013

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Role of Pirh2 in mediating the regulation of p53 and c-Myc

PLoS genetics, 2011-11, Vol.7 (11), p.e1002360

2011

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TRIMming p53 for ubiquitination

Proceedings of the National Academy of Sciences - PNAS, 2009-07, Vol.106 (28), p.11431-11432

2009

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Apoptosis Caused by p53-Induced Protein with Death Domain (PIDD) Depends on the Death Adapter Protein RAIDD

Proceedings of the National Academy of Sciences - PNAS, 2005-10, Vol.102 (40), p.14314-14319

2005

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DNA damage- and stress-induced apoptosis occurs independently of PIDD

Apoptosis (London), 2009-09, Vol.14 (9), p.1039-1049

2009

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Poly(ADP-ribose) Polymerase-1 Is a Positive Regulator of the p53-mediated G1 Arrest Response following Ionizing Radiation

The Journal of biological chemistry, 2003-05, Vol.278 (21), p.18914-18921

2003

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ATM-dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post-translational activation of p53 protein involving poly(ADP-ribose) polymerase

The EMBO journal, 1997-10, Vol.16 (19), p.6018-6033

1997

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Progerin-Induced Replication Stress Facilitates Premature Senescence in Hutchinson-Gilford Progeria Syndrome

Molecular and cellular biology, 2017-07, Vol.37 (14)

2017

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HDAC5aA Critical Player in the p53 Acetylation Network

Molecular cell, 2013-11, Vol.52 (3), p.289-290

2013

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Ckap2 regulates aneuploidy, cell cycling, and cell death in a p53-dependent manner

Cancer research (Chicago, Ill.), 2005-08, Vol.65 (15), p.6685-6691

2005

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The Involvement of MAPK Signaling Pathways in Determining the Cellular Response to p53 Activation

The Journal of biological chemistry, 2006-02, Vol.281 (7), p.3832-3840

2006

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The p53 gene as a modifier of intrinsic radiosensitivity: implications for radiotherapy

Radiotherapy and Oncology, 1996-09, Vol.40 (3), p.197-223

1996

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Characterization of Cells and Gene-Targeted Mice Deficient for the p53-Binding Kinase Homeodomain-Interacting Protein Kinase 1 (HIPK1)

Proceedings of the National Academy of Sciences - PNAS, 2003-04, Vol.100 (9), p.5431-5436

2003

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The involvement of MAPK signaling pathways in determining the cellular response to p53 activation: cell cycle arrest or apoptosis

The Journal of biological chemistry, 2006-02, Vol.281 (7), p.3832-3840

2006

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p53-Dependent Transcriptional Repression of c-myc Is Required for G 1 Cell Cycle Arrest

Molecular and cellular biology, 2005-09, Vol.25 (17), p.7423-7431

2005

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Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation

Cell, 2003-03, Vol.112 (6), p.779-791

2003

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HDAC5--a critical player in the p53 acetylation network

Molecular cell, 2013-11, Vol.52 (3), p.289-290

2013

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Erythropoietin inhibits chemotherapy-induced cell death and promotes a senescence-like state in leukemia cells

Cell death & disease, 2019-01, Vol.10 (1), p.22-22, Article 22

2019

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p53-Dependent Transcriptional Repression of c-myc Is Required for G1 Cell Cycle Arrest

Molecular and Cellular Biology, 2005-09, Vol.25 (17), p.7423-7431

2005

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ROS-Mediated p53 Induction of Lpin1 Regulates Fatty Acid Oxidation in Response to Nutritional Stress

Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span

Heterogeneity in leukemia cells that escape drug-induced senescence-like state

BTG2 antagonizes Pin1 in response to mitogens and telomere disruption during replicative senescence

Pidd, a new death-domain-containing protein, is induced by p53 and promotes apoptosis

Differential utilization of two ATP-generating pathways is regulated by p53

Proteasome-independent p53 degradation

Role of Pirh2 in mediating the regulation of p53 and c-Myc

TRIMming p53 for ubiquitination

Apoptosis Caused by p53-Induced Protein with Death Domain (PIDD) Depends on the Death Adapter Protein RAIDD

DNA damage- and stress-induced apoptosis occurs independently of PIDD

Poly(ADP-ribose) Polymerase-1 Is a Positive Regulator of the p53-mediated G1 Arrest Response following Ionizing Radiation

ATM-dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post-translational activation of p53 protein involving poly(ADP-ribose) polymerase

Progerin-Induced Replication Stress Facilitates Premature Senescence in Hutchinson-Gilford Progeria Syndrome

HDAC5aA Critical Player in the p53 Acetylation Network

Ckap2 regulates aneuploidy, cell cycling, and cell death in a p53-dependent manner

The Involvement of MAPK Signaling Pathways in Determining the Cellular Response to p53 Activation

The p53 gene as a modifier of intrinsic radiosensitivity: implications for radiotherapy

Characterization of Cells and Gene-Targeted Mice Deficient for the p53-Binding Kinase Homeodomain-Interacting Protein Kinase 1 (HIPK1)

The involvement of MAPK signaling pathways in determining the cellular response to p53 activation: cell cycle arrest or apoptosis

p53-Dependent Transcriptional Repression of c-myc Is Required for G 1 Cell Cycle Arrest

Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation

HDAC5--a critical player in the p53 acetylation network

Erythropoietin inhibits chemotherapy-induced cell death and promotes a senescence-like state in leukemia cells

p53-Dependent Transcriptional Repression of c-myc Is Required for G1 Cell Cycle Arrest

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