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Activation of the omega-3 fatty acid receptor GPR120 mediates anti-inflammatory actions in immortalized hypothalamic neurons

Journal of neuroinflammation, 2014-03, Vol.11 (1), p.60-60

2014

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Phoenixin Activates Immortalized GnRH and Kisspeptin Neurons Through the Novel Receptor GPR173

Molecular endocrinology (Baltimore, Md.), 2016-08, Vol.30 (8), p.872-888

2016

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Glucose Alters Per2 Rhythmicity Independent of AMPK, Whereas AMPK Inhibitor Compound C Causes Profound Repression of Clock Genes and AgRP in mHypoE-37 Hypothalamic Neurons

PloS one, 2016-01, Vol.11 (1), p.e0146969

2016

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Impact of nutrients on circadian rhythmicity

American journal of physiology. Regulatory, integrative and comparative physiology, 2015-03, Vol.308 (5), p.R337-R350

2015

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Palmitate Attenuates Insulin Signaling and Induces Endoplasmic Reticulum Stress and Apoptosis in Hypothalamic Neurons: Rescue of Resistance and Apoptosis through Adenosine 5′ Monophosphate-Activated Protein Kinase Activation

Endocrinology (Philadelphia), 2010-02, Vol.151 (2), p.576-585

2010

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Bisphenol A Induces Agrp Gene Expression in Hypothalamic Neurons through a Mechanism Involving ATF3

Neuroendocrinology, 2021-06, Vol.111 (7), p.678-695

2021

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Promise and Perils of MicroRNA Discovery Research: Working Toward Quality Over Quantity

Endocrinology (Philadelphia), 2023-08, Vol.164 (9)

2023

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Immortalization and Characterization of GFAP-expressing Glial Cells from the Adult Mouse Hypothalamus, Cortex, and Brain Stem

Neuroscience, 2024-07, Vol.551, p.43-54

2024

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Bisphenol A induces Pomc gene expression through neuroinflammatory and PPARγ nuclear receptor-mediated mechanisms in POMC-expressing hypothalamic neuronal models

Molecular and cellular endocrinology, 2019-01, Vol.479, p.12-19

2019

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Palmitate alters miRNA content of small extracellular vesicles secreted from NPY/AgRP-expressing hypothalamic neurons

Brain research, 2023-07, Vol.1810, p.148367-148367, Article 148367

2023

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Antipsychotics differentially regulate insulin, energy sensing, and inflammation pathways in hypothalamic rat neurons

Psychoneuroendocrinology, 2019-06, Vol.104, p.42-48

2019

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Npy transcription is regulated by noncanonical STAT3 signaling in hypothalamic neurons: Implication with lipotoxicity and obesity

Molecular and cellular endocrinology, 2024-05, Vol.586, p.112179-112179, Article 112179

2024

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Palmitate induces neuroinflammation, ER stress, and Pomc mRNA expression in hypothalamic mHypoA-POMC/GFP neurons through novel mechanisms that are prevented by oleate

Molecular and cellular endocrinology, 2018-09, Vol.472, p.40-49

2018

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Bisphenol A Alters Bmal1, Per2, and Rev-Erba mRNA and Requires Bmal1 to Increase Neuropeptide Y Expression in Hypothalamic Neurons

Endocrinology (Philadelphia), 2019-01, Vol.160 (1), p.181-192

2019

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Hypothalamic reproductive neurons communicate through signal transduction to control reproduction

Molecular and cellular endocrinology, 2020-12, Vol.518, p.110971-110971, Article 110971

2020

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Phenylbutyric acid robustly increases Npy mRNA expression in hypothalamic neurons by increasing H3K9/14 acetylation at the Npy promoter

Biochemical and biophysical research communications, 2023-05, Vol.658, p.18-26

2023

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Palmitate and Nitric Oxide Regulate the Expression of Spexin and Galanin Receptors 2 and 3 in Hypothalamic Neurons

Neuroscience, 2020-11, Vol.447, p.41-52

2020

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Beneficial Effects of Metformin and/or Salicylate on Palmitate- or TNFα-Induced Neuroinflammatory Marker and Neuropeptide Gene Regulation in Immortalized NPY/AgRP Neurons

PloS one, 2016-11, Vol.11 (11), p.e0166973-e0166973

2016

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Insulin signalling in hypothalamic neurones

Journal of neuroendocrinology, 2021-04, Vol.33 (4), p.e12919-n/a

2021

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Palmitate alters miR‐2137 and miR‐503‐5p to induce orexigenic Npy in hypothalamic neuronal cell models: Rescue by oleate and docosahexaenoic acid

Journal of neuroendocrinology, 2023-05, Vol.35 (5), p.e13271-n/a

2023

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Bisphenol S induces Agrp expression through GPER1 activation and alters transcription factor expression in immortalized hypothalamic neurons: A mechanism distinct from BPA-induced upregulation

Molecular and cellular endocrinology, 2022-07, Vol.552, p.111630-111630, Article 111630

2022

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Bisphenol A induces miR-708-5p through an ER stress-mediated mechanism altering neuronatin and neuropeptide Y expression in hypothalamic neuronal models

Molecular and cellular endocrinology, 2022-01, Vol.539, p.111480-111480, Article 111480

2022

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Molecular Basis for the Activation of Gonadotropin-Inhibitory Hormone Gene Transcription by Corticosterone

Endocrinology (Philadelphia), 2014-05, Vol.155 (5), p.1817-1826

2014

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Regulation of Gpr173 expression, a putative phoenixin receptor, by saturated fatty acid palmitate and endocrine-disrupting chemical bisphenol A through a p38-mediated mechanism in immortalized hypothalamic neurons

Molecular and cellular endocrinology, 2019-04, Vol.485, p.54-60

2019

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The Involvement of the microRNAs miR-466c and miR-340 in the Palmitate-Mediated Dysregulation of Gonadotropin-Releasing Hormone Gene Expression

Genes, 2024-04, Vol.15 (4), p.397

2024

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Activation of the omega-3 fatty acid receptor GPR120 mediates anti-inflammatory actions in immortalized hypothalamic neurons

Phoenixin Activates Immortalized GnRH and Kisspeptin Neurons Through the Novel Receptor GPR173

Glucose Alters Per2 Rhythmicity Independent of AMPK, Whereas AMPK Inhibitor Compound C Causes Profound Repression of Clock Genes and AgRP in mHypoE-37 Hypothalamic Neurons

Impact of nutrients on circadian rhythmicity

Palmitate Attenuates Insulin Signaling and Induces Endoplasmic Reticulum Stress and Apoptosis in Hypothalamic Neurons: Rescue of Resistance and Apoptosis through Adenosine 5′ Monophosphate-Activated Protein Kinase Activation

Bisphenol A Induces Agrp Gene Expression in Hypothalamic Neurons through a Mechanism Involving ATF3

Promise and Perils of MicroRNA Discovery Research: Working Toward Quality Over Quantity

Immortalization and Characterization of GFAP-expressing Glial Cells from the Adult Mouse Hypothalamus, Cortex, and Brain Stem

Bisphenol A induces Pomc gene expression through neuroinflammatory and PPARγ nuclear receptor-mediated mechanisms in POMC-expressing hypothalamic neuronal models

Palmitate alters miRNA content of small extracellular vesicles secreted from NPY/AgRP-expressing hypothalamic neurons

Antipsychotics differentially regulate insulin, energy sensing, and inflammation pathways in hypothalamic rat neurons

Npy transcription is regulated by noncanonical STAT3 signaling in hypothalamic neurons: Implication with lipotoxicity and obesity

Palmitate induces neuroinflammation, ER stress, and Pomc mRNA expression in hypothalamic mHypoA-POMC/GFP neurons through novel mechanisms that are prevented by oleate

Bisphenol A Alters Bmal1, Per2, and Rev-Erba mRNA and Requires Bmal1 to Increase Neuropeptide Y Expression in Hypothalamic Neurons

Hypothalamic reproductive neurons communicate through signal transduction to control reproduction

Phenylbutyric acid robustly increases Npy mRNA expression in hypothalamic neurons by increasing H3K9/14 acetylation at the Npy promoter

Palmitate and Nitric Oxide Regulate the Expression of Spexin and Galanin Receptors 2 and 3 in Hypothalamic Neurons

Beneficial Effects of Metformin and/or Salicylate on Palmitate- or TNFα-Induced Neuroinflammatory Marker and Neuropeptide Gene Regulation in Immortalized NPY/AgRP Neurons

Insulin signalling in hypothalamic neurones

Palmitate alters miR‐2137 and miR‐503‐5p to induce orexigenic Npy in hypothalamic neuronal cell models: Rescue by oleate and docosahexaenoic acid

Bisphenol S induces Agrp expression through GPER1 activation and alters transcription factor expression in immortalized hypothalamic neurons: A mechanism distinct from BPA-induced upregulation

Bisphenol A induces miR-708-5p through an ER stress-mediated mechanism altering neuronatin and neuropeptide Y expression in hypothalamic neuronal models

Molecular Basis for the Activation of Gonadotropin-Inhibitory Hormone Gene Transcription by Corticosterone

Regulation of Gpr173 expression, a putative phoenixin receptor, by saturated fatty acid palmitate and endocrine-disrupting chemical bisphenol A through a p38-mediated mechanism in immortalized hypothalamic neurons

The Involvement of the microRNAs miR-466c and miR-340 in the Palmitate-Mediated Dysregulation of Gonadotropin-Releasing Hormone Gene Expression

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