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The SirT3 Divining Rod Points to Oxidative Stress

Molecular cell, 2011-06, Vol.42 (5), p.561-568

2011

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Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging

Cell, 2013-12, Vol.155 (7), p.1624-1638

2013

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SIRT1 Suppresses the Epithelial-to-Mesenchymal Transition in Cancer Metastasis and Organ Fibrosis

Cell reports (Cambridge), 2013-04, Vol.3 (4), p.1175-1186

2013

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SIRT1 Activates MAO-A in the Brain to Mediate Anxiety and Exploratory Drive

Cell, 2011-12, Vol.147 (7), p.1459-1472

2011

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Depletion of a Putatively Druggable Class of Phosphatidylinositol Kinases Inhibits Growth of p53-Null Tumors

Cell, 2013-11, Vol.155 (4), p.844-857

2013

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Qo site of the mitochondrial complex III is required for the transduction of hypoxic signaling via reactive oxygen species production

The Journal of cell biology, 2007-06, Vol.177 (6), p.1029-1036

2007

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SirT1 is required in the male germ cell for differentiation and fecundity in mice

Development (Cambridge), 2014-09, Vol.141 (18), p.3495-3504

2014

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Metformin Decreases Glucose Oxidation and Increases the Dependency of Prostate Cancer Cells on Reductive Glutamine Metabolism

Cancer research (Chicago, Ill.), 2013-07, Vol.73 (14), p.4429-4438

2013

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Sirtuin 1 Promotes Deacetylation of Oct4 and Maintenance of Naive Pluripotency

Cell reports (Cambridge), 2016-10, Vol.17 (3), p.809-820

2016

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Mitochondrial Reactive Oxygen Species Trigger Hypoxia-Inducible Factor-Dependent Extension of the Replicative Life Span during Hypoxia

Molecular and Cellular Biology, 2007-08, Vol.27 (16), p.5737-5745

2007

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Global microRNA depletion suppresses tumor angiogenesis

Genes & development, 2014-05, Vol.28 (10), p.1054-1067

2014

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Identification of CDCP1 as a hypoxia-inducible factor 2α (HIF-2α) target gene that is associated with survival in clear cell renal cell carcinoma patients

Proceedings of the National Academy of Sciences - PNAS, 2013-02, Vol.110 (9), p.3483-3488

2013

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Mitochondrial Complex III-generated Oxidants Activate ASK1 and JNK to Induce Alveolar Epithelial Cell Death following Exposure to Particulate Matter Air Pollution

The Journal of biological chemistry, 2009-01, Vol.284 (4), p.2176-2186

2009

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ABC transporters: human disease and pharmacotherapeutic potential

Trends in molecular medicine, 2023-02, Vol.29 (2), p.152-172

2023

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Reductive glutamine metabolism by IDH1 mediates lipogenesis under hypoxia

Nature (London), 2012-01, Vol.481 (7381), p.380-384

2012

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Mitochondrial oxygen sensing: regulation of hypoxia-inducible factor by mitochondrial generated reactive oxygen species

Essays in biochemistry, 2007-01, Vol.43, p.17-28

2007

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Reductive glutamine metabolism is a function of the α-ketoglutarate to citrate ratio in cells

Nature communications, 2013, Vol.4 (1), p.2236-2236, Article 2236

2013

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Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation

Cell metabolism, 2005-06, Vol.1 (6), p.409-414

2005

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PPARδ modulation rescues mitochondrial fatty acid oxidation defects in the mdx model of muscular dystrophy

Mitochondrion, 2019-05, Vol.46, p.51-58

2019

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Selective PPARδ Modulators Improve Mitochondrial Function: Potential Treatment for Duchenne Muscular Dystrophy (DMD)

ACS medicinal chemistry letters, 2018-09, Vol.9 (9), p.935-940

2018

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Natriuretic peptide receptor-B in adult rat ventricle is predominantly confined to the nonmyocyte population

American journal of physiology. Heart and circulatory physiology, 2002-06, Vol.282 (6), p.H2117-H2123

2002

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Targeting the mitochondria for cancer therapy: regulation of hypoxia-inducible factor by mitochondria

Antioxidants & redox signaling, 2008-03, Vol.10 (3), p.635-640

2008

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Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROS

The FASEB journal, 2009-03, Vol.23 (3), p.783-794

2009

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Mitochondrial regulation of oxygen sensing

Mitochondrion, 2005-10, Vol.5 (5), p.322-332

2005

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Correction to “Selective PPARδ Modulators Improve Mitochondrial Function: Potential Treatment for Duchenne Muscular Dystrophy (DMD)”

ACS medicinal chemistry letters, 2019-06, Vol.10 (6), p.1013-1013

2019

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The SirT3 Divining Rod Points to Oxidative Stress

Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging

SIRT1 Suppresses the Epithelial-to-Mesenchymal Transition in Cancer Metastasis and Organ Fibrosis

SIRT1 Activates MAO-A in the Brain to Mediate Anxiety and Exploratory Drive

Depletion of a Putatively Druggable Class of Phosphatidylinositol Kinases Inhibits Growth of p53-Null Tumors

Qo site of the mitochondrial complex III is required for the transduction of hypoxic signaling via reactive oxygen species production

SirT1 is required in the male germ cell for differentiation and fecundity in mice

Metformin Decreases Glucose Oxidation and Increases the Dependency of Prostate Cancer Cells on Reductive Glutamine Metabolism

Sirtuin 1 Promotes Deacetylation of Oct4 and Maintenance of Naive Pluripotency

Mitochondrial Reactive Oxygen Species Trigger Hypoxia-Inducible Factor-Dependent Extension of the Replicative Life Span during Hypoxia

Global microRNA depletion suppresses tumor angiogenesis

Identification of CDCP1 as a hypoxia-inducible factor 2α (HIF-2α) target gene that is associated with survival in clear cell renal cell carcinoma patients

Mitochondrial Complex III-generated Oxidants Activate ASK1 and JNK to Induce Alveolar Epithelial Cell Death following Exposure to Particulate Matter Air Pollution

ABC transporters: human disease and pharmacotherapeutic potential

Reductive glutamine metabolism by IDH1 mediates lipogenesis under hypoxia

Mitochondrial oxygen sensing: regulation of hypoxia-inducible factor by mitochondrial generated reactive oxygen species

Reductive glutamine metabolism is a function of the α-ketoglutarate to citrate ratio in cells

Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation

PPARδ modulation rescues mitochondrial fatty acid oxidation defects in the mdx model of muscular dystrophy

Selective PPARδ Modulators Improve Mitochondrial Function: Potential Treatment for Duchenne Muscular Dystrophy (DMD)

Natriuretic peptide receptor-B in adult rat ventricle is predominantly confined to the nonmyocyte population

Targeting the mitochondria for cancer therapy: regulation of hypoxia-inducible factor by mitochondria

Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROS

Mitochondrial regulation of oxygen sensing

Correction to “Selective PPARδ Modulators Improve Mitochondrial Function: Potential Treatment for Duchenne Muscular Dystrophy (DMD)”

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