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Loss of Dishevelleds Disrupts Planar Polarity in Ependymal Motile Cilia and Results in Hydrocephalus

Neuron (Cambridge, Mass.), 2014-08, Vol.83 (3), p.558-571

2014

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Age-dependent brain gene expression and copy number anomalies in autism suggest distinct pathological processes at young versus mature ages

PLoS genetics, 2012-03, Vol.8 (3), p.e1002592

2012

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Activation of the Amyloid Cascade in Apolipoprotein E4 Transgenic Mice Induces Lysosomal Activation and Neurodegeneration Resulting in Marked Cognitive Deficits

The Journal of neuroscience, 2008-04, Vol.28 (18), p.4690-4701

2008

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Altered proliferation and networks in neural cells derived from idiopathic autistic individuals

Molecular psychiatry, 2017-06, Vol.22 (6), p.820-835

2017

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Possible Role of Tau in Mediating Pathological Effects of apoE4 in vivo prior to and following Activation of the Amyloid Cascade

Neuro-degenerative diseases, 2010-01, Vol.7 (1-3), p.16-23

2010

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ApoE4-dependent Aβ-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

Journal of Neural Transmission, 2009-11, Vol.116 (11), p.1427-1434

2009

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ApoE4-Driven Accumulation of Intraneuronal Oligomerized Aβ42 following Activation of the Amyloid Cascade In Vivo Is Mediated by a Gain of Function

International journal of alzheimer's disease, 2011-02, Vol.2011, p.792070-792070

2011

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Laquinimod Treatment Improves Myelination Deficits at the Transcriptional and Ultrastructural Levels in the YAC128 Mouse Model of Huntington Disease

Molecular neurobiology, 2019-06, Vol.56 (6), p.4464-4478

2019

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ApoE4-dependent Ab-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

Journal of Neural Transmission, 2009-11, Vol.116 (11), p.1427-1434

2009

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Loss ofDishevelleds Disrupts Planar Polarity in Ependymal Motile Cilia and Results in Hydrocephalus

Neuron (Cambridge, Mass.), 2014-08, Vol.83 (3), p.558

2014

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Pathological synergism between amyloid-beta and apolipoprotein E4--the most prevalent yet understudied genetic risk factor for Alzheimer's disease

Journal of Alzheimer's disease, 2009, Vol.17 (3), p.469

2009

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Following activation of the amyloid cascade, apolipoprotein E4 drives the in vivo oligomerization of amyloid-β resulting in neurodegeneration

Journal of Alzheimer's disease, 2010-01, Vol.22 (3), p.959

2010

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Intraneuronal amyloid-β plays a role in mediating the synergistic pathological effects of apoE4 and environmental stimulation

Journal of neurochemistry, 2007-11, Vol.103 (3), p.1031-1040

2007

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P3-209: ApoE4 induces Aβ mediated apoptosis in vivo

Alzheimer's & dementia, 2006-07, Vol.2 (3), p.S437-S437

2006

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Intraneuronal amyloid-[beta] plays a role in mediating the synergistic pathological effects of apoE4 and environmental stimulation

Journal of neurochemistry, 2007-11, Vol.103 (3), p.1031

2007

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Neuron-specific susceptibility to apolipoprotein E4

Neurobiology of aging, 2007-05, Vol.28 (5), p.689-692

2007

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ApoE4-dependent Abeta-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

Journal of neural transmission (Vienna, Austria : 1996), 2009-11, Vol.116 (11), p.1427-1434

2009

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P2‐285: The role of LRP in mediating the synergistic pathological effects of apoE4 and amyloid‐β in vivo

Alzheimer's & dementia, 2010-07, Vol.6 (4S_Part_13), p.S397-S397

2010

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The role of LRP in mediating the synergistic pathological effects of apoE4 and amyloid-β in vivo

Alzheimer's & dementia, 2010-07, Vol.6 (4), p.S397-S397

2010

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Dual epithelial and immune cell function of Dvl1 regulates gut microbiota composition and intestinal homeostasis

JCI insight, 2016-07, Vol.1 (10)

2016

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ApoE4‐Driven Accumulation of Intraneuronal Oligomerized A β 42 following Activation of the Amyloid Cascade In Vivo Is Mediated by a Gain of Function

International journal of alzheimer's disease, 2011-01, Vol.2011 (1)

2011

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ApoE4-driven accumulation of intraneuronal oligomerized A[beta]42 following activation of the amyloid cascade in vivo is mediated by a gain of function

International journal of alzheimer's disease, 2011-01

2011

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Activation of the Amyloid Cascade by Intracerebroventricular Injection of the Protease Inhibitor Phosphoramidon

Neuro-degenerative diseases, 2008-01, Vol.5 (3-4), p.166-169

2008

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P3-291: Activation of the amyloid cascade in apoE4 transgenic mice by inhibition of neprilysin induces neurodegeneration and learning and memory deficits

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T607-T608

2008

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O4-01-03: A common unifying mechanism underlies the amyloid-β and environmental stimulation-related pathological effects of apoE4 in vivo

Alzheimer's & dementia, 2006-07, Vol.2 (3), p.S75-S75

2006

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Loss of Dishevelleds Disrupts Planar Polarity in Ependymal Motile Cilia and Results in Hydrocephalus

Age-dependent brain gene expression and copy number anomalies in autism suggest distinct pathological processes at young versus mature ages

Activation of the Amyloid Cascade in Apolipoprotein E4 Transgenic Mice Induces Lysosomal Activation and Neurodegeneration Resulting in Marked Cognitive Deficits

Altered proliferation and networks in neural cells derived from idiopathic autistic individuals

Possible Role of Tau in Mediating Pathological Effects of apoE4 in vivo prior to and following Activation of the Amyloid Cascade

ApoE4-dependent Aβ-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

ApoE4-Driven Accumulation of Intraneuronal Oligomerized Aβ42 following Activation of the Amyloid Cascade In Vivo Is Mediated by a Gain of Function

Laquinimod Treatment Improves Myelination Deficits at the Transcriptional and Ultrastructural Levels in the YAC128 Mouse Model of Huntington Disease

ApoE4-dependent Ab-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

Loss ofDishevelleds Disrupts Planar Polarity in Ependymal Motile Cilia and Results in Hydrocephalus

Pathological synergism between amyloid-beta and apolipoprotein E4--the most prevalent yet understudied genetic risk factor for Alzheimer's disease

Following activation of the amyloid cascade, apolipoprotein E4 drives the in vivo oligomerization of amyloid-β resulting in neurodegeneration

Intraneuronal amyloid-β plays a role in mediating the synergistic pathological effects of apoE4 and environmental stimulation

P3-209: ApoE4 induces Aβ mediated apoptosis in vivo

Intraneuronal amyloid-[beta] plays a role in mediating the synergistic pathological effects of apoE4 and environmental stimulation

Neuron-specific susceptibility to apolipoprotein E4

ApoE4-dependent Abeta-mediated neurodegeneration is associated with inflammatory activation in the hippocampus but not the septum

P2‐285: The role of LRP in mediating the synergistic pathological effects of apoE4 and amyloid‐β in vivo

The role of LRP in mediating the synergistic pathological effects of apoE4 and amyloid-β in vivo

Dual epithelial and immune cell function of Dvl1 regulates gut microbiota composition and intestinal homeostasis

ApoE4‐Driven Accumulation of Intraneuronal Oligomerized A β 42 following Activation of the Amyloid Cascade In Vivo Is Mediated by a Gain of Function

ApoE4-driven accumulation of intraneuronal oligomerized A[beta]42 following activation of the amyloid cascade in vivo is mediated by a gain of function

Activation of the Amyloid Cascade by Intracerebroventricular Injection of the Protease Inhibitor Phosphoramidon

P3-291: Activation of the amyloid cascade in apoE4 transgenic mice by inhibition of neprilysin induces neurodegeneration and learning and memory deficits

O4-01-03: A common unifying mechanism underlies the amyloid-β and environmental stimulation-related pathological effects of apoE4 in vivo

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