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Widespread changes in dendritic and axonal morphology in Mecp2-mutant mouse models of rett syndrome: Evidence for disruption of neuronal networks

Journal of comparative neurology (1911), 2009-05, Vol.514 (3), p.240-258

2009

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The "Down Syndrome Critical Region" Is Sufficient in the Mouse Model to Confer Behavioral, Neurophysiological, and Synaptic Phenotypes Characteristic of Down Syndrome

The Journal of neuroscience, 2009-05, Vol.29 (18), p.5938-5948

2009

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Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues

PloS one, 2015-07, Vol.10 (7), p.e0134861-e0134861

2015

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Genetic dissection of the Down syndrome critical region

Human molecular genetics, 2015-11, Vol.24 (22), p.6540-6551

2015

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Synaptic and cognitive abnormalities in mouse models of down syndrome: Exploring genotype-phenotype relationships

Journal of comparative neurology (1911), 2007-10, Vol.504 (4), p.329-345

2007

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Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome

Neurobiology of disease, 2012-02, Vol.45 (2), p.683-691

2012

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Hippocampal Long-Term Potentiation Suppressed by Increased Inhibition in the Ts65Dn Mouse, a Genetic Model of Down Syndrome

The Journal of neuroscience, 2004-09, Vol.24 (37), p.8153-8160

2004

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Increased App Expression in a Mouse Model of Down's Syndrome Disrupts NGF Transport and Causes Cholinergic Neuron Degeneration

Neuron (Cambridge, Mass.), 2006-07, Vol.51 (1), p.29-42

2006

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Evidence for both neuronal cell autonomous and nonautonomous effects of methyl-CpG-binding protein 2 in the cerebral cortex of female mice with Mecp2 mutation

Neurobiology of disease, 2009-04, Vol.34 (1), p.71-77

2009

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Comparative study of brain morphology in Mecp2 mutant mouse models of Rett syndrome

Journal of comparative neurology (1911), 2008-05, Vol.508 (1), p.184-195

2008

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Neuroprotection of Host Cells by Human Central Nervous System Stem Cells in a Mouse Model of Infantile Neuronal Ceroid Lipofuscinosis

Cell stem cell, 2009-09, Vol.5 (3), p.310-319

2009

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Neuronal Plasticity and Dendritic Spines: Effect of Environmental Enrichment on Intact and Postischemic Rat Brain

Journal of cerebral blood flow and metabolism, 2002-01, Vol.22 (1), p.89-96

2002

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Three-dimensional synaptic ultrastructure in the dentate gyrus and hippocampal area CA3 in the Ts65Dn mouse model of down syndrome

Journal of comparative neurology (1911), 2011-05, Vol.519 (7), p.1338-1354

2011

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Mouse Models for Down Syndrome-Associated Developmental Cognitive Disabilities

Developmental neuroscience, 2011-01, Vol.33 (5), p.404-413

2011

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Excitatory-inhibitory relationship in the fascia dentata in the Ts65Dn mouse model of down syndrome

Journal of comparative neurology (1911), 2009-02, Vol.512 (4), p.453-466

2009

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MODULATING GAMMA SECRETASE ACTIVITY TO ADDRESS APP GENE DOSE EFFECTS IN DOWN SYNDROME

Alzheimer's & dementia, 2014-07, Vol.10 (4), p.P624-P625

Belichenko, Pavel V

2014

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An Anti-β-Amyloid Vaccine for Treating Cognitive Deficits in a Mouse Model of Down Syndrome

PloS one, 2016-03, Vol.11 (3), p.e0152471-e0152471

2016

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Synaptic structural abnormalities in the Ts65Dn mouse model of down syndrome

Journal of comparative neurology (1911), 2004-12, Vol.480 (3), p.281-298

2004

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Guidelines for reporting clinical features in cases with MECP2 mutations

Brain & development (Tokyo. 1979), 2001-07, Vol.23 (4), p.208-211

2001

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Human chromosome 21 orthologous region on mouse chromosome 17 is a major determinant of Down syndrome-related developmental cognitive deficits

Human molecular genetics, 2014-02, Vol.23 (3), p.578-589

2014

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Genetic analysis of Down syndrome facilitated by mouse chromosome engineering

Bioengineered, 2012-01, Vol.3 (1), p.8-12

2012

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Failed Retrograde Transport of NGF in a Mouse Model of Down's Syndrome: Reversal of Cholinergic Neurodegenerative Phenotypes Following NGF Infusion

Proceedings of the National Academy of Sciences - PNAS, 2001-08, Vol.98 (18), p.10439-10444

2001

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Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists

The Journal of neuroscience, 2012-07, Vol.32 (27), p.9217-9227

2012

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Identification and characterization of a new Down syndrome model, Ts[Rb(12.1716)]2Cje, resulting from a spontaneous Robertsonian fusion between T(171)65Dn and mouse chromosome 12

Mammalian genome, 2005-02, Vol.16 (2), p.79-90

2005

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Widespread changes in dendritic and axonal morphology in Mecp2-mutant mouse models of rett syndrome: Evidence for disruption of neuronal networks

The "Down Syndrome Critical Region" Is Sufficient in the Mouse Model to Confer Behavioral, Neurophysiological, and Synaptic Phenotypes Characteristic of Down Syndrome

Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues

Genetic dissection of the Down syndrome critical region

Synaptic and cognitive abnormalities in mouse models of down syndrome: Exploring genotype-phenotype relationships

Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome

Hippocampal Long-Term Potentiation Suppressed by Increased Inhibition in the Ts65Dn Mouse, a Genetic Model of Down Syndrome

Increased App Expression in a Mouse Model of Down's Syndrome Disrupts NGF Transport and Causes Cholinergic Neuron Degeneration

Evidence for both neuronal cell autonomous and nonautonomous effects of methyl-CpG-binding protein 2 in the cerebral cortex of female mice with Mecp2 mutation

Comparative study of brain morphology in Mecp2 mutant mouse models of Rett syndrome

Neuroprotection of Host Cells by Human Central Nervous System Stem Cells in a Mouse Model of Infantile Neuronal Ceroid Lipofuscinosis

Neuronal Plasticity and Dendritic Spines: Effect of Environmental Enrichment on Intact and Postischemic Rat Brain

Three-dimensional synaptic ultrastructure in the dentate gyrus and hippocampal area CA3 in the Ts65Dn mouse model of down syndrome

Mouse Models for Down Syndrome-Associated Developmental Cognitive Disabilities

Excitatory-inhibitory relationship in the fascia dentata in the Ts65Dn mouse model of down syndrome

MODULATING GAMMA SECRETASE ACTIVITY TO ADDRESS APP GENE DOSE EFFECTS IN DOWN SYNDROME

An Anti-β-Amyloid Vaccine for Treating Cognitive Deficits in a Mouse Model of Down Syndrome

Synaptic structural abnormalities in the Ts65Dn mouse model of down syndrome

Guidelines for reporting clinical features in cases with MECP2 mutations

Human chromosome 21 orthologous region on mouse chromosome 17 is a major determinant of Down syndrome-related developmental cognitive deficits

A Syntenic Cross Species Aneuploidy Genetic Screen Links RCAN1 Expression to β-Cell Mitochondrial Dysfunction in Type 2 Diabetes

Genetic analysis of Down syndrome facilitated by mouse chromosome engineering

Failed Retrograde Transport of NGF in a Mouse Model of Down's Syndrome: Reversal of Cholinergic Neurodegenerative Phenotypes Following NGF Infusion

Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists

Identification and characterization of a new Down syndrome model, Ts[Rb(12.1716)]2Cje, resulting from a spontaneous Robertsonian fusion between T(171)65Dn and mouse chromosome 12

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